Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Biochem Exam 5 > Oral Pain and Its Management > Flashcards

Oral Pain and Its Management Flashcards

1
Q

_____ is the most prevalent type of pain in the orofacial region

A

Toothache (odontalgia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

T/F: Not all pains are the same, not all toothaches are the same

A

True

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Pulpal and Periodontal pain are categorized as ______ orofacial pain

A

Odontogenic pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Myofascial, Sinus/nasal, Neurovascular, Neuropathic, Idiopathic, Cardiogenic (rare), and Systemic (rare) pain are categorized as ______ orofacial pain

A

Non-Odontogenic pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Temporomandibular Disorders are more common in males or females?

A

Females

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Temporomandibular Disorders need treatment what percent of the time?

A

3.6-7%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Unpleasant sensory and emotional experience associated with actual or potential tissue damage or described in terms of such damage

A

pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

_____ Instills protective behavior but if unabated, pain can be harmful

A

pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

________: It is always subjective; It may or may not be tied to a stimulus; It is always a consequence of an emotional experience and psychological state

A

pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Environmental stimulus (thermal, mechanical, chemical, polymodal), Receptor activation•Generation of action potential, Transmission through primary afferent to dorsal horn, and Projection from dorsal horn to brain for perception and interpretation are ways we experience _____

A

pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

____ is not pain until it reaches and is processed by higher centers (supraspinal structures)

A

nociception

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

_____ is a symphony - a complex response that includes not just a distinct sensation but also motor activity, a change in emotion, a focusing of attention, a brand new memory

A

pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

____ and ____ are descending pathways of pain modulation

A

supraspinal and spinal pathways

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

The _____ pathway (descending) are influenced by psychological factors; neurons from the cortex and amygdala; periaqueductal gray and rostroventral medulla

A

supraspinal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

The ____ pathway (descending) are associated with endogenous opioid signaling and non-opiod inhibitory neurotransmitters

A

spinal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Serotonin, noradrenaline, GABA, and glycine are _________ neurotransmitters

A

non-opioid inhibitory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

_____ is a dynamic process that can occur at multiple levels of the ascending and descending pathways

A

pain modulation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

_____ is characterized by an increased responsiveness of nociceptive neurons under normal input and by a recruitment of a response (APs) under subthreshold input

A

sensitization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Nociceptive neurons at periphery of receptive field; Increased responsiveness of nociceptive neurons; Reduced threshold to stimulation.(Primary hyperalgesia)

A

Peripheral sensitization

20
Q

Increased pain response to a painful stimulus

A

Hyperalgesia

21
Q

Nociceptive neurons in the central nervous system; Increased responsiveness to normal or subthreshold afferent input; may also result from dysfunctional endogenous pain control (Primary and secondary hyperalgesia) (Allodynia)

A

Central sensitization

22
Q

Pain resulting from a stimulus that does not normally provoke pain

A

Allodynia

23
Q

Myelinated (fast) non-nociceptive afferent fiber can activate inhibitory interneurons modulating nociceptive transmission•Reason you instinctively wave, hold, clench your fingers when they burn•Reason why TENS helps relieve pain

A

Revised gate control theory

24
Q

The threshold for nociception can be raised when another noxious stimulus is provoked in another area.“Pain inhibits pain”

A

Diffuse noxious inhibitor control

25
Q

Psychological; Leads to release of endogenous analgesic substances

A

Placebo effect

26
Q

What are the 3 different ways to categorize pain?

A

neurophysiology, structures involved, and timing

27
Q

Pain resulting from damage or threatened damage to non-neural tissue; Activation of nociceptors; Sharp

A

Nociceptive pain

28
Q

Pain resulting from the presence of a lesion or disease of the somatosensory nervous system

A

Neuropathic pain

29
Q

Pain that arises from altered nociception •Does not satisfy the definitions of nociceptive or neuropathic pain•It is possible for a patient to present with nociceptive and this pain at the same time

A

nociplastic pain

30
Q

Craniofacial pain that is characterized by throbbing, wakes patient, and autonomic and systemic signs

A

neurovascular

31
Q

Craniofacial pain that is characterized by pressure, deep pain, muscle tenderness, and dysfunction

A

musculoskeleton

32
Q

Craniofacial pain that is characterized by sensory dysfunction, dermatomal pain, and burning or electric pain

A

neuropathic

33
Q

Pain with close temporal relationship to an stimulus, injury, disease; Tends to respond to treatment in a linear dose-dependent fashion

A

Acute Pain

34
Q

Pain that has lasted >3 months•Does not typically respond to treatment in in a linear dose-dependent fashion•Presence of other/multiple ongoing pains is a predictor for transition from acute to chronic•More influence of psychosocial factors•More difficult to treat

A

Chronic Pain

35
Q

What are the 3 type of heterotopic pain?

A

Central, projected, and referred pain

36
Q

Anxiety•Major depression•Personality disorders•Pain distress are the most common _______ disorders

A

psychosocial disorders

37
Q

Perceived control•Self-efficacy•Catastrophic thinking•Hypervigilance•Fear avoidance are ____ for psychosocial disorders

A

coping mechanisms

38
Q

Pain intensity, Pain distress, Pain-related interference (Functional limitation, disability), and Oral Habits are ____ of pain

A

assessments

39
Q

Can you treat site of pain in homotopic pain?

A

Yes

40
Q

Can you treat site of pain in heterotopic pain?

A

No

41
Q

____ pain: site = source

A

homotopic pain

42
Q

___ pain: site does not equal source

A

heterotopic pain

43
Q

Heterotopic pain where Source is central but perceived peripherally

A

Central Pain

44
Q

Heterotopic pain where Pain follows same nerve distribution as primary source•Dermatome or motor distribution•Hyperalgesia may be present

A

Projected pain

45
Q

Heterotopic pain where Pain occurs in different nerve than primary source and is spontaneous (non-provoked)•Sensitization of interneurons –central sensitization•Not aggravated by palpation•Does not respond to anesthesia at site of pain –must block primary site•Typically does not cross midline (only if generated at midline)•Refers upward: cervical to trigeminal, mandibular to maxillary

A

referred pain

Biochem Exam 5 (9 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions