Oral Mucosal (white/red patches) Flashcards

1
Q

The oral mucosal lining

A

stratified squamous epithelium

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2
Q

What is underneath the epithelium

A

lamina propira (loose connective tissue)

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3
Q

3 types of oral mucosa

A

lining
masticatory
gustatory

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4
Q

What happens in the stratum basale

A

where the cells divide

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5
Q

What happens in the stratum spinosum

A

cells mature here and have small spines

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6
Q

Acanthosis

A

thicking of epithelium due to trauma or immunological damage
(hyperplasia of stratum spinosum)

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7
Q

Elongated rete ridges

A

hyperplasia of basal cells

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8
Q

Atrophy

A

reduction in viable layers

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9
Q

Erosion

A

partial thickness loss

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10
Q

atypia

A

describes changes in cells

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11
Q

dysplasia

A

disordered maturation in tissue

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12
Q

Geographic tongue

A

alteration to the maturation and the replacment of normal epithelial surface
Thinning of epithelial
aymptomatic but sometimes sens to spicy foods
basal cell replication will stop and restart in patchy areas

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13
Q

Black hairy tongue

A

elongation of surface papillae which then becomate stained with pigments from food stuff
use tongue scraper or nectroine stone

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14
Q

Fissure tongue

A
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15
Q

Glossitis

A

inflammation of the tongue

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16
Q

Why are some lesions white?

A

Due to thickening of the mucosa on the surface which gives less viability of blood cells in connective tissue beneath
as thickening increases the opacity of the epithelium will increase
Microscopically - hyperkeratosis, parakeratosis

17
Q

Types of keratosis

A

frictional keratosis
smokers keratosis
fordyce spots
chemical burn
heridiatry - white spongy nauvus
candial leukoplakia

18
Q

leukoplakia

A

a white patch which cannot be scraped off and has an unknown cause

19
Q

Fordyce spots

A

small clusters of spots <5mm on buccal mucosa or lip from ectopic sebacous gland

20
Q

Frictional keratosis

A

trauamtic source obvious
thickening of mucosa

21
Q

Smokers keratosis

A

seen as hyperkeratosis with raised levels of dysplasia but minimal infiltrate
likely to develop to leukoplakia

22
Q

White spongy naevus

A

family linked
occurs in areas subject to trauma
starts posterior and moves anterior

23
Q

Chemical burn

A

substance held in contact with mucosa which has casued cogulation of proteins and damges epithelial surface

24
Q

Candial lukoplakia

A

hyperplasitc candidosis (smoking noramally the cause)
present in commisures and tissues appears dyplastic
candida removed tissue returns to normalbiopsy for check for dysplasia

25
Q

Why are lesions sometimes red?

A

Atrophy of the epithelium allowing for blood vessels to be seen below or increased blood vessels in area

26
Q

Erythroplakia

A

red patch - area of unexplained red change in mucosa
more concern for malgninancy than leukoplakia

27
Q

Management of white/red patched

A

if unexplained - biopsy which identifies malginancies, dysplasia or lichen planus

28
Q

Local Causes of brown/black lesions

A

amaglam tatoo
malgninacy melonoma
poigmentatry incontence
Kaposi’s sarcoma
Macule/naevus
peutz-jehger’s syndrome

29
Q

Generalised causes of brown/black lesions

A

Smoking
racial
addisions
drugs

30
Q

what is an hamartoma

A

tumour like overgrowth - regular tissue but abnormal number or distribution

31
Q

Haemangioma

A

vascualr hamartoma

32
Q

lymphagioma

A

hamartoma of the lymph
common on base of tongue

33
Q

cystic hygoma

A

commonly found on left posterior triangle of neck