OCD Flashcards

1
Q

What is the prominent model?

A

Compulsive checking

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2
Q

Who made the Compulsive checking model

A

Rachman 2002

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3
Q

What is the psychological intervention?

A

CBT

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4
Q

what is the biological intervention?

A

SSRI’s

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5
Q

symptoms of OCD

A

recurrent obsessions and/or compulsions with attempts to suppress. repetitive behaviours

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6
Q

Compulsive checking model structure

A

P responsibility, P probability of harm, P seriousness of harm, anxiety, preventative checking, self perpetuating mechanism, out of control, reduces self-esteem, need to be careful

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7
Q

First part of CCM

A

perceived responsibility

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8
Q

what comes after perceived responsibility

A

perceived probability of harm

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9
Q

what comes after perceived probability of harm

A

perceived seriousness of harm

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10
Q

what comes after perceived seriousness of harm

A

anxiety and preventative checking

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11
Q

what comes after preventative checking

A

self-perpetuating mechanism (reinforcement and impaired meta memory)

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12
Q

what comes after self-perpetuating mechanism

A

out of control behaviour and reduced self esteem

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13
Q

what comes after reduced self esteem

A

the need to be careful - preventative checking cycle

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14
Q

name a study investigating the Compulsive checking model (1)

A

radomskey et al 2006 stove study

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15
Q

what is the radomskey et al 2006 study

A

PP’s turned on stove rated confidence of turning off stove, took part in either relevant or irrelevant checking and rated confidence of first turning off stove

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16
Q

who did this study?
PP’s turned on stove rated confidence of turning off stove, took part in either relevant or irrelevant checking and rated confidence of first turning off stove

A

radomskey et al 2006

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17
Q

what did the radomskey et al 2006 study find

A

Relevant checking group had a decrease in confidence irrelevant checking stayed the same

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18
Q

name a study investigating the Compulsive checking model (2)

A

Boschen & Vuksanovik 2007 replication

19
Q

positive of radomskey et al 2006 study

A

tight control to seperate cause and effect

20
Q

negatives of radomskey et al 2006 study

A

lack of external validity and not using OCD pp’s

21
Q

what is the Boschen & Vuksanovik 2007 study

A

a replication of radomskey et al 2006 study using control vs OCD patients

22
Q

who did this study
a replication of radomskey et al 2006 study using control vs OCD patients

A

Boschen & Vuksanovik 2007

23
Q

what did the Boschen & Vuksanovik 2007 study find

A

same findings as radomskey et al 2006 for OCD patients- Relevant checking group had a decrease in confidence irrelevant checking stayed the same

24
Q

what is CBT for OCD

A

exposure and response prevention

25
Q

what did the Olatunji et al. (2013): study look at

A

waitlist vs active controls for CBT on OCD in RCT

26
Q

who did this study?
waitlist vs active controls for CBT on OCD in RCT

A

Olatunji et al. (2013):

27
Q

how does CBT for OCD work

A

gradual exposure to anxiety- prevent response and exercises to reduce feelings of responsibility

28
Q

a study for CBT in OCD

A

Olatunji et al. (2013): meta-analysis of 16 trials of CBT for OCD

29
Q

what did the Olatunji et al. (2013): study look at

A

waitlist vs active controls for CBT on OCD in RCT

30
Q

what did the Olatunji et al. (2013): study find

A

CBT was more effective than other treatments (large effect)

31
Q

what did the Olatunji et al. (2013) follow up studies find

A

3 followed up and found CBT was more effective than other treatments over the long term

32
Q

what are the cons of the Olatunji et al. (2013): study

A

bias from RCT but investigated and found no difference

33
Q

what do SSRI’S do

A

prevent the re uptake of serotonin in the synapse to elevate the availability of serotonin

34
Q

a study for SSRIs

A

Bloch 2010

35
Q

what did Bloch 2010 study do

A

meta analysis of 9 trails of the use of SSRI’s in OCD

36
Q

what did Bloch 2010 find

A

SSRI’s showed greater improvements to the placebo and higher doses worked more

37
Q

problem with SSRI’s

A

bad side effects which caused drop outs

38
Q

pros of Bloch 2010

A

RCT’s were done properly: All randomized, double-blind, and placebo-controlled.

39
Q

what are obsessions

A

as recurrent, persistent thoughts and urges that usually cause distress and the patient must attempt to supress them with another action or thought with little to no success

40
Q

what are compulsions

A

repetitive behaviours that the individual feels compelled to perform in response to be obsession, these must be aimed to preventing anxiety but the behaviours are not related in a realistic way

41
Q

symptoms according to the DSM V are…

A

time consuming, not due to substance abuse or another mental health problem

42
Q

how many people have OCD

A

between 1-4%

43
Q
A