Obesity in Horses Flashcards

1
Q

Define obesity

A

Medical condiiton where excess fat accumulation -> negative affect on health

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2
Q

How is obesity measured in horses?

A
NOT BMI (accurate weight difficult) 
- BCS (geenralised adiposity) or CNS (regional adiposity)
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3
Q

What are the 2 BCS scales? What are optimum scores for each?

A
  • 0-5 with 2.5-3 optimum (use 1/2 scores)

- 1-9 with 5 optimum (use whole scores)

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4
Q

Which areas are emphasised in condition scoring?

A

Tailhead, back, withers, neck, rib, behind the shoulder

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5
Q

What is the optimum CNS?

A

2

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6
Q

Which ponies are usually seen to be obese?

A

Show ponies

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7
Q

Which animals are most commonly seen to develop obesity?

A
  • draught/cobs/native/welsh ponies
  • good doers
  • pleasure/nonridden
  • summer more than winter
    > underrecognised by owners
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8
Q

Why does adiposity increase in the summer?

A
  • ^POMC from pituitary pars intermedia -> apetite and adipogeneiss
  • evolutionary adaptation to get through winter
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9
Q

What adverse effects does chronic adiposity have?

A
  • Insulin resistance (fat releases hormones that inhibit insulin)
  • Mild pro-inflammatory state
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10
Q

Give 3 conditions associated with physical presence of excess fat (1*)

A
  • excercise intolerance
  • abnormal repro
  • mesenteric lipoma (±strangulation)
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11
Q

Give 4 condiitons asscoaited with obesity and IR (2*)

A
  • Laminitis
  • Hyperlipaemia (stress induced)
  • DOD (developmental orthopaedic problems)
  • EMS
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12
Q

Which aspect of obesity causes the greatest risk of laminitis?

A

> IR

- suggeted changes in insulin signalling, inflammaotry cytokines, endothelial dysfucntion

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13
Q

What are the epigenetics implications of obesity?

A
Pre natal (genes switched off or on during late foetal and early post-natal stages) dependency on maternal diet
> obesity or emaciation  
- obese mothers have ^ glucose conc and NEFA -> epigentic changes, possibly permenant (appetite control, neuroendocrine, fuel metabolism and energy partintioning) 
- sub-optimum nutrition -> v pancreatic weight and B cells, structual changes -> impaired glucose homeostasis
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14
Q

What must be considered when controlling calorie intake in horses?

A
  • DMI must be maintained

- welfare concerns associated with restricting DMI (sterotypies, colic, ulcers, dental)

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15
Q

How can weight loss be acheived?

A
> resitrict calorie intake 
- grass muzzle
- double net forage
- restirct pasture and feed hay/straw 
- soak hay to wash out nutritents
- hang from middle of stable - difficult for horse to get food 
- subsitute hay with chaff or unmollassed beet pulp (IF OWNER WANTS TO FEED MEALS - NOT necessary) 
> increase energy expenditure 
- water and food at opposite ends of pasture
- strip graze (circumferential) 
- clip
- do not rug
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16
Q

How does ^ energy expenditure -> weight loss?

A
  • promotes glucose uptake and use by skeletal mm. by insulin indepentant route for ~24hrs
  • ^ insulin sensitivity
  • v inflammation
  • v feed intake
17
Q

What % body weight should a horse eat in forage? How much are they capable of eating?

A

1.5%

can consume 3% BW in 3 hours at grass!!