Neuro & motor speech Flashcards

1
Q

What is thrombosis?

A

A collection of blood material that blocks the flow of blood

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2
Q

What is embolus?

A

Traveling mass of arterial debris or a clump of tissue from tumor that gets lodged in a smaller artery and blocks the flow of blood

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3
Q

What are hemorrhagic strokes?

A

Caused by bleeding in the brain due to ruptured blood vessels.

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4
Q

What’s the definition of aphasia?

A

Neurologically based language disorder

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5
Q

What aphasia a are no fluent?

A

Global

Broca’s

Transcortical motor aphasia

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6
Q

What are the fluent aphasias?

A

Wernicke’s

Transcortical sensory aphasia

Conductive

Anomia

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7
Q

Characteristics of broca’s aphasia?

A

No fluent, effortfull, slow, halting, and uneven speech

Limited word output, short phrases and sentences

Misarticulated or distorted speech sounds

A grammatical error telegraphic speech

Poor repetition

Inspired naming

Poor oral reading and comprehension

Monotonous speech

Aware of deficits

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8
Q

Characteristics of transcortical motor aphasia?

A

Absent or reduced spontaneous speech

Nonfluent, paraphasic, agrammatic, and telegraphic speech

Echolalia

Limited word fluency

Good comprehension

Slow and difficult reading aloud

Inspired writing

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9
Q

What is akinesia?

A

Decreased sense of movement

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10
Q

What is bradykinesia?

A

Slowness of movement

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11
Q

Characteristics of Wernicke’s?

A

Press of speech (logarrehea)

Rate of speech with normal and good articulation

Severe word finding problems

Neologisms

Circumlocutions

Empty speech (this, that, stuff & thing)

Impaired conversational turn taking

Impaired repetition skills

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12
Q

What is ischemic?

A

Blocked or interrupted blood supply to the brain.

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13
Q

Characteristics of global

A

Profound anomia

Virtually no speech output

Very poor auditory comp

Stereotypical utterances

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14
Q

Lesion site for Broca’s

A

Anterior MCA

Frontal lobe (posterior)

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15
Q

Lesion site for Wernicke’s

A

Posterior temporal gyrus

Posterior section of middle and inferior temporal gyrus

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16
Q

Lesion site for global

A

Impacts frontal, parietal and temporal lobes

Often caused by occlusion of MCA prior to branching in LH

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17
Q

Lesion site for transcortical motor aphasia

A

Sub cortical anterior to frontal horn of lateral ventricle (left frontal lobe)

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18
Q

Characteristics of transcortical motor

A

Non fluent

Good repetition

Good auditory comp

Impaired imitation

Phrase length ~ 5

Agrammatic

Telegraphic

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19
Q

Characteristics of conduction aphasia

A

Fluent

Good auditory comp

Poor repetitions

Self corrections

Phrase length WNL

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20
Q

Site of lesion for conduction aphasia

A

Arcuate fasciculus

21
Q

Characteristics of anomic aphasia

A

Good comprehension

Good repetition

Fluent

Circumlocutions

Semantic paraphasias

Phrase length- WNL

22
Q

Lesion sit for anomic aphasia

A

Inferotemporal gyrus

23
Q

Characteristics for transcortical sensory aphasia

A

Poor comprehension

Good repetition

Severe anomia

Fluent

Artic, prosody, grammar= good

Empty content

Perseveration

24
Q

Lesion site for transcortical sensory aphasia

A

Angular gyrus

Post middle temporal gyrus

Lateral part of occipital

25
Treatment for AOS
Articulatory accuracy Slower rate Systematic practice Gradual increase in the rate Normal prosody Self monitoring and self correction
26
Treatment for aphasia
Auditory comprehension single words spoken sentences yes/no & following directions ``` Verbal expression- naming use functional words use modeling to evoke incomplete sentences phonetic cues syllabic cues ``` Verbal expression- expanded utterances Reading skills survival reading skills reading newspapers reading and comprehending words, phrases, sentences and paragraphs Writing skills egocentric functional words
27
Types of dysarthria
flaccid spastic dysarthria hypokinetic dysarthria hyperkinetic dysarthria ataxia UUMN
28
Describe flaccid dysarthria
hyper nasality deficits in automatic (reading, swallowing), reflexive (coughing), and voluntary (speech) movements imprecise consonant productions breathiness of voice decreased loudness monotone and slow rate of speech common etiology: myasthenia graves, guillain-barre, ALS
29
describe spastic dysarthria
Harsh Strain-strangled Low pitch Slow rate Pitch breaks Slow and regular AMRs
30
describe ataxia dysarthria
Irregular articulatory breakdowns (inconsistent) Irregular AMRs Excess and equal stress Slow and inaccurate Distorted vowels “Drunken speech” Poorly coordinate movement pattern Scanning or staccato pattern of speech Ataxic Gait Friedrich’s Ataxia
31
describe hypo kinetic dysarthria
Decreased ROM Affects aspects of motor control (preparation, maintenance, switching of motor programs) Increased rate of speech with reduced intelligibility Monopitch and monoloudness Palilalia Associated with basal ganglia pathology (Parkinson’s)
32
What is AOS?
Apraxia of speech. A motor planning disorder, that primarily affects prosody and articulation
33
Where is the site of lesions for AOS?
Left hemisphere, motor strip and Broca's area. Frequently co-exists with Broca's aphasia
34
Characteristics of apraxia of speech
Slow rate Excess and equal stress Groping Inconsistent errors
35
Assessment for AOS
Imitative speech tasks, phonemes, syllables, and progressively longer words DDK (they won't be able to do these) Automatic speech tasks Picture descriptions Limb movement to rule out limb apraxia WAB or apraxia battery for adults
36
Tx for AOS
Repetition Articulating accuracy slower rate Modification of prosody Encourage client to communicate in whichever mode is best for them (writing, a communication book, gestures, or speech)
37
What is Dysarthria
It is a neurological speech disorder
38
What is the hallmark for dysarthria?
Impaired muscle control In addition, it may impact all systems of language
39
Characteristics of ataxic dysarthria and the systems of speech
Phonation: mono pitch, monoloud, harsh quality Articulation:imprecise consonants, distorted vowels. Drunken speech Prosody: prolonged phonemes and word junction, slow rate of speech, excess and even stress Other: gait disturbances and uncoordinated, slow, halting movements
40
Characteristics of spastic dysarthria and the systems of speech
Results from bilateral damage to the UMN Respiration: short phrases, reduced AMEs/DDK Phonation: mono pitch, monoloud, harsh, strained-strangled Articulation: imprecise consonants, distorted vowels Prosody: reduced stress, slow rate Other: muscle spasticity and weakness, hyperactive gag reflex, reduced range and slowness of movement
41
Characteristics of flaccid dysarthria and the systems of speech
Damage to CN that I control LMN Respiration: weakness linked to CN weakness, short phrases Phonation: breathy quality, audible inspiration, monoloudness Articulation: imprecise consonants Resonance: hypernasality Other: muscle twitches and other muscular disorders
42
Characteristics of hyperkinetic dysarthria and the systems of speech
Huntington's Basal ganglia Respiration: audible inspiration and forced or sudden inspiration or expedition Phonation: voice tremor, intermittent strained voice, harsh voice, voice stopping, dystonia Articulation: imprecise consonants, distorted vowels Prosody: slower rate, excess loudness, equal stress Resonance: hypernasality Other: trimmers and ticks, movement disorders including facial and in movements, involuntary movements, abrupt and the severe contractions of extremities, and neck spasms.
43
Characteristics of hypokinetic dysarthria and the systems of speech
Basil ganglia Respiration: irregular, rapid rate of breathing, reduced to vital capacity Phonation: mono pitch mono loud, breathy or harsh quality Articulation: imprecise confidence, "stuttering like" repetitions Prosody: reduced stress, short bursts, inappropriate silences Other: "masked" face, resting tremor, rigidity and increased muscle tone, micrographic writing, decreased swelling
44
Assessment for dysarthria
Obtain medical history Obtain a conversational speech sample and a reading sample to evaluate connected speech Assess DDKs OME Determine the speech intelligibility rating Frenchay dysarthria assessment
45
Intervention for dysarthria
Dysarthria is very unique, intervention varies Postural adjustments for adequate respiration Slow rate Over articulation AAC biofeedback fur resonance and phonation SLOP
46
5 parameters of dysarthria
Respiration, phonation, resonance, articulation and prosody
47
Lesion site for AOS
Left hemisphere, frontal lobe More cortical than sub cortical
48
What two types of blockages are there?
Thrombosis and embolism