Neuro: General Comprehensive Flashcards

1
Q

The patient is awake and attentive to normal levels of stimulation. Interactions with the therapist are normal and appropriate.

A

Alert

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

The patient appears drowsy and may fall asleep if not stimulated in some way. Interactions with the therapist may get diverted. Patient may have difficulty in focusing or maintaining attention on a question or task.

A

Lethargic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

The patient is difficult to arouse from a somnolent state and is frequently confused when awake. Repeated stimulation is required to maintain consciousness. Interactions with the therapist may be largely unproductive.

A

Obtunded

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

The patient responds only to strong, generally noxious stimuli and returns to the unconscious state when stimulation is stopped. When aroused, the patient is unable to interact with the therapist.

A

Stupor (semi-coma)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

The patient cannot be aroused by any type of stimulation. Reflex motor responses may or may not be seen. There are no sleep wake cycles.

A

Coma (deep coma)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Return of irregular sleep–wake cycles and normalization of the so-called vegetative functions (respiration, digestion, and blood pressure control). The patient may be aroused, but remains unaware of his or her environment. There is no purposeful attention or cognitive responsiveness.

A

Minimally conscious (vegetative state) - Persistent if present after 1 yr following TBI - Persistent if present after 3 months following anoxic brain injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Glasgow Coma Scale: 8 or less

A

Severe Brain Injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Glasgow Coma Scale: 9 - 12

A

Moderate Brain Injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Glasgow Coma Scale: 13 - 15

A

Mild Brain Injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Sustained contraction and posturing of the upper limbs in flexion and the lower limbs in extension. - The elbows, wrists, and fingers are held in flexion with shoulders adducted tightly to the sides while the legs are held in extension, internal rotation, and plantarflexion.

A

Decorticate Rigidity - Indicative of damage to the corticospinal tract lesion at the level of diencephalon (above the superior colliculus).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Sustained contraction and posturing of the trunk and limbs in a position of full extension. - The elbows are extended with shoulders adducted, forearms pronated, and wrist and fingers flexed. - The legs are held in stiff extension with plantarflexion.

A

Decerebrate Rigidity - Corticospinal lesion in the brainstem between the superior colliculus and vestibular nucleus.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Noted Impairments: - Inability to plan motor tasks (apraxia) - Difficulty in initiating, sequencing and processing a task - Difficulty in producing or comprehending speech - memory impairment - perseveration of speech or motor behaviors

A

Left hemisphere damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Noted Impairments: - Poor judgement and safety awareness - Unrealistic expectations - Denial of disability or deficits - disturbances in body image - irritability -lethargy

A

Right Hemisphere damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q
  • Allows for processing of information sequentially, logically for analysis. - Language production - Recognize words and comprehend what has been read - Mathematical calculations - Sequence and perform movements and gestures - Expression of positive emotions
A

Left Hemisphere

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Major motor pathway - Origonates in the frontal lobe in the primary and premotor cortices->internal capsule->synapse on anterior horn cells in the spinal cord

A

Corticospinal tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Rancho LOCF: Patient appears to be in a deep sleep and is completely unresponsive to any stimuli.

A

Level I: no response

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Rancho LOCF: Patient reacts inconsistently and nonpurposefully to stimuli in a nonspecific manner. Responses are limited and often the same regardless of stimulus presented. Responses may be physiological changes, gross body movements, and/or vocalization.

A

Level II: generalized response

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Rancho LOCF: Patient reacts specifically but inconsistently to stimuli. Responses are directly related to the type of stimulus presented. May follow simple commands such as closing eyes or squeezing hand in an inconsistent, delayed manner.

A

Level III: Localized Response

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Rancho LOCF: Patient is in a heightened state of activity. Behavior is bizarre and nonpurposeful relative to immediate environment. Does not discriminate among persons or objects; is unable to cooperate directly with treatment efforts. Verbalizations frequently are incoherent and/or inappropriate to the environment; confabulation may be present. Gross attention to environment is very brief; selective attention is often nonexistent. Patient lacks short- and long-term recall.

A

Level IV: Confused Agitated

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Rancho LOCF: Patient is able to respond to simple commands fairly consistently. However, with increased complexity of commands or lack of any external structure, responses are nonpurposeful, random, or fragmented. Demonstrates gross attention to the environment but is highly distractible and lacks ability to focus attention on a specific task. With structure, may be able to converse on a social automatic level for short periods of time. Verbalization is often inappropriate and confabulatory. Mem- ory is severely impaired; often shows inappropriate use of objects; may perform previously learned tasks with structure but is unable to learn new information.

A

Level V: Confused Inappropriate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Rancho LOCF: Patient shows goal-directed behavior but is dependent on external input or direction. Follows simple directions consis- tently and shows carryover for relearned tasks such as self-care. Responses may be incorrect due to memory problems, but they are appropriate to the situation. Past memories show more depth and detail than recent memory.

A

Level VI: Confused-Appropriate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Rancho LOCF: Patient appears appropriate and oriented within the hospital and home settings; goes through daily routine automatically, but frequently robot-like. Patient shows minimal to no confusion and has shallow recall of activities. Shows carryover for new learning but at a decreased rate. With structure is able to initiate social or recreational activities; judgment remains impaired.

A

Level VII: Automatic-Appropriate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Rancho LOCF: Patient is able to recall and integrate past and recent events and is aware of and responsive to environment. Shows carryover for new learning and needs no supervision once activities are learned. May continue to show a decreased ability relative to premorbid abilities, abstract reasoning, tolerance for stress, and judgment in emergencies or unusual circumstances.

A

Level VIII: Purposeful-Appropriate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Most common CVA type

A

Middle cerebral artery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

CVA: - Contralteral hemiplegia - Upper extremity greater than lower - loss of sensation primarily in arm and face - Infarction in dominant left hemisphere may produce aphasia and apraxia

A

Middle Cerebral Artery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

CVA: - Rarely involved artery - LE with greater involvement than UE – contralateral hemiplegia and sensory loss - mental confusion - aphasia - contralateral neglect with extensive dominant side involvement

A

Anterior Cerebral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

CVA: - Persistent pain syndrome or contralateral loss of pain and sensory - Homonomous hemianopsia - aphasia - thalamic pain syndrome

A

Posterior Cerebral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

CVA: - Most often results in death from infarct associated edema - “locked in” state with only eye blinking available - vertigo - coma - diplopia - nausea - dysphagia - ataxia

A

Vertebral Basilar - With pons affected: quadriparesis and bulbar palsy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

CVA: - unilateral deafness - contralateral pain and temperature loss - paresis of lateral gaze - unilateral Horner’s Syndrome (ptosis, constructed pupil, loss of sweating) - ataxia - vertigo - nystagmus

A

Anterior Inferior Cerebellar (AICA)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

CVA: - severe ataxia - dysarthria - dysmetria - contralateral loss of pain and temperature

A

Superior Cerebellar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

CVA: - Wallenberg Syndrome: — (vertigo, nausea, hoarseness, dysphagia, and decreased impairment of sensation in the ipsilateral face, contralateral torso and limbs. - Horner’s Syndrome also possible

A

Posterior Inferior Cerebellar (PICA)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

An inability of the patient to produce movement either on command or automatically and represents a complete breakdown in the conceptualization of the task. The patient has no idea how to do the movement and thus cannot formulate the required motor programs.

A

Ideational apraxia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

The patient is unable to produce a movement on command but is able to move automatically. Thus, the patient can perform habitual tasks when not commanded to do so and often perseverates, repeating the activity over and over

A

Ideomotor apraxia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

Synergy: Scapular Retraction and elevation (or hyperextension)

A

UE flexor synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

Synergy: Shoulder abduction, external rotation

A

UE flexor synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

Synergy: Elbow flexion*

A

UE flexor synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

Synergy: Forearm supination

A

UE flexor synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

Synergy: Wrist and finger flexion

A

UE flexor AND extensor synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

Synergy: Scapular protraction

A

UE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

Synergy: - Shoulder adduction,* internal rotation

A

UE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

Synergy: - Elbow extension

A

UE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

Synergy: - Forearm pronation*

A

UE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

Synergy: - Hip flexion,* abduction, external rotation

A

LE Flexor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
44
Q

Synergy: - Knee flexion

A

LE Flexor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
45
Q

Synergy: - Ankle dorsiflexion, inversion

A

LE Flexor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
46
Q

Synergy: - Toe dorsiflexion

A

LE Flexor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
47
Q

Synergy: - Hip extension, adduction,* internal rotation

A

LE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
48
Q

Synergy: - Knee extension*

A

LE Extensor Synergy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
49
Q

Synergy: - Ankle plantarflexion,* inversion

A

LE Extensor Synergy

50
Q

Synergy: - Toe plantarflexion

A

LE Extensor Synergy

51
Q

Stage of Motor Recovery: - Flaccidity — often with no voluntary movement

A

Stage 1

52
Q

Stage of Motor Recovery: - Development of spasticity, hyperreflexia, and mass patterns of movement (obligatory synergies) - Minimal voluntary movement

A

Stage 2 (Early Synergy)

53
Q

Stage of Motor Recovery: - Spasticity is marked with full strong obligatory synergies - Voluntary control of movement synergies appears

A

Stage 3

54
Q

Stage of Motor Recovery: - Synergy influence begins to decline as some isolated out-of-synergy joint movements emerge.

A

Stage 4

55
Q

Stage of Motor Recovery: - Relative independence of synergy - Spasticity continues to wane and isolated joint movements become more apparent. - Synergies no longer dominant and movement becomes more complex

A

Stage 5

56
Q

Stage of Motor Recovery: - Patterns of movement are near normal. - Spasticity is gone - Coordination and movement approach normal

A

Stage 6

57
Q

Which hemisphere affected? - Problems with spatial awareness and E-H coordination

A

Right hemisphere

58
Q

Which hemisphere affected? - Irritability and short attention span

A

Right hemisphere

59
Q

Which hemisphere affected? - Cannot retain information; difficulty learning individual steps - Difficulty grasping the whole idea of a task or activity.

A

Right hemisphere

60
Q

Which hemisphere affected? - Poor judgement affecting personal safety

A

Right hemisphere

61
Q

Which hemisphere affected? - Diminished body image with left sided neglect

A

Right hemisphere

62
Q

Which hemisphere affected? - Quick and impulsive

A

Right hemisphere

63
Q

Which hemisphere affected? - Apraxia

A

Left hemisphere

64
Q

Which hemisphere affected? - Difficulty starting and sequencing tasks

A

Left hemisphere

65
Q

Which hemisphere affected? - Perseveration

A

Left hemisphere

66
Q

Which hemisphere affected? - Easily frustrated with high levels of anxiety

A

Left hemisphere

67
Q

Which hemisphere affected? - Inability to communicate verbally

A

Left hemisphere

68
Q

Which hemisphere affected? - Cautious and slow

A

Left hemisphere

69
Q

Which Aphasia? - The patient demonstrates difficulty in comprehending spoken language and in following commands. - Speech flows smoothly with a variety of grammatical constructions and preserved melody of speech.

A

Wernicke’s Aphasia (fluent aphasia) - Receptive [impairment] aphasia - Sensory [impairment] aphasia - Damage to: LEFT LATERAL TEMPORAL LOBE - Use word repetition and manual cues to to assist in communicating desired action during rehabilitation

70
Q

Which Aphasia? - The flow of speech is slow and hesitant, vocabulary is limited, and syntax is impaired. - Speech production is labored or lost completely whereas comprehension is good.

A

Broca’s Aphasia (non-fluent aphasia) - Expressive [imparment] aphasia - Damage to: PREMOTOR AREA OF LEFT FRONTAL LOBE - Use simple “yes” or “no” questions to avoid confusion and frustration

71
Q

Which Aphasia? - Characterized by marked impairments of both production and comprehension of language.

A

Global Aphasia: - It is often an indication of extensive brain damage. - Use symbolic gestures to reduce the chance of overestimating the patient’s ability to understand speech.

72
Q

Paralysis of: - intrinsic hand muscles (interossei, thenar, hypothenar) - flexors of the wrist and fingers (flexor carpi ulnaris, ulnar half of flexor digitorum profundis) - forearm pronators

A

Klumpke Paralysis: (C8-T1) - Presents as intrinsic minus or claw hand - Horner’s syndrome may also be present

73
Q

Order of testing for fall history/balance impairment

A

Cognition -> sensory testing -> static balance tests (i.e. CTSIB) -> dynamic balance tests.

74
Q

Gaze stability (X1 viewing) and postural stability are treatments for:

A

Unilateral and Bilateral Vestibular Hypofunction.

75
Q

What training is used for UVH patients with continual complaints of dizziness?

A

Habituation training (motion sensitivity training) - can also help patients with central vestibular lesions.

76
Q

Patients with lesions that occur above the conus medullaris and sacral segments develop a _______ bladder.

A

Spastic of hyperreflexive. - manage with suprapubic stroking or tapping

77
Q

To allow for function in long sitting, hamstrings should be ranged to what degree?

A

110 degrees.

78
Q

Sensory: C6 nerve root

A

Palmar surface of the thumb and radial forearm

79
Q

Sensory: C7 nerve root

A

Middle of the hand (second, third and fourth fingers Palmar surface)

80
Q

Sensory: C8 nerve root

A

Ulnar border of the hand (fifth finger)

81
Q

Sensory: T1 nerve root

A

Medial surface of the forearm

82
Q

Modified Ashworth: - No increase in muscle tone.

A

MAS: 0

83
Q

Modified Ashworth: - Slight increase in muscle tone, manifested by a catch and release or by minimal resistance at the end of the ROM when the affected part(s) is moved in flexion or extension.

A

MAS: 1

84
Q

Modified Ashworth: - Slight increase in muscle tone, manifested by a catch, followed by minimal resistance through- out the remainder (LESS THAN HALF) of the ROM.

A

MAS: 1+

85
Q

Modified Ashworth: - More marked increase in muscle tone through most of the ROM, but affected part(s) easily moved.

A

MAS: 2

86
Q

Modified Ashworth: - Considerable increase in muscle tone, passive movement difficult.

A

MAS: 3

87
Q

Modified Ashworth: - Affected part(s) rigid in flexion or extension.

A

MAS: 4

88
Q

Deep Tendon Reflex Grading: - Absent, no response

A

DTR: 0

89
Q

Deep Tendon Reflex Grading: - Slight reflex, present but depressed, low normal

A

DTR: 1+

90
Q

Deep Tendon Reflex Grading: - Normal, typical reflex

A

DTR: 2+

91
Q

Deep Tendon Reflex Grading: - Brisk reflex, possibly but not necessarily abnormal

A

DTR: 3+

92
Q

Deep Tendon Reflex Grading: - Very brisk reflex, abnormal, clonus

A

DTR: 4+

93
Q

I present with: Hyperalgesia (increased sensitivity to pain), allodynia (all stimuli perceived as painful), and hyperpathia (increased intensity) What am I?

A

Complex Regional Pain Syndrome (Stage 1)

94
Q

I present with: Increased pain with edema and atrophic skin and nail changes. What am I?

A

Complex Regional Pain Syndrome (Stage 2)

95
Q

I present with: Spreading pain, hardening of edema, cool dry and cyanotic skin, developing osteoporosis, and ankylosis What am I?

A

Complex Regional Pain Syndrome (Stage 3)

96
Q

The MOST useful test to determine whether demyelination has taken place in polyneuropathy.

A

Nerve Conduction Velocity

97
Q

Recommendations for pressure ulcers in patients with spinal cord injury who are wheelchair users include:

A

10 to 15 seconds of pressure relief (push-ups) for every 15-20 minutes of sitting.

98
Q

Wheelchair for C4 tetraplegia:

A

Power wheelchair with sip and puff controls - Face, neck, diaphragm, and trapezius innervated. - Also needs tilt in space frame for pressure relief

99
Q

Wheelchair for C6-C7 tetraplegia

A

Manual wheelchair with friction surface handrims: - Used when patients do not have functional grip or strength necessary to adequately propel a wheelchair

100
Q

Wheelchair for C5 tetraplegia

A

Manual wheelchair with handrim projections OR power wheelchair with joystick controls - Have biceps, brachialis, brachioradialis, deltoids, rhomboids, supinator. - Energy expenditure required may necessitate a power wheelchair with joystick controls

101
Q

Sensations transmitted by the dorsal column–medial lemniscal pathway include:

A
  • discriminative touch - stereognosis - tactile pressure - barognosis - graphesthesia - recognition of texture - kinesthesia - two-point discrimination - proprioception - vibration.
102
Q

The three major tracts of the spinothalamic system and the sensations they carry:

A

Anterior (ventral) spinothalamic tract: – crudely localized touch and pressure Lateral spinothalamic tract: – Pain and temperature Spinoreticular tract: – Diffuse pain sensations.

103
Q

Seven-point scale which examines 18 items (based on observations of patient performance) regarding self care, sphincter control, transfers, locomotion, communication, and social cognitive abilities.

A

Functional Independence Measure

104
Q

Mini Mental State Exam score: less than 20

A

Associated with dementia, delirium, schizophrenia, or an affective disorder

105
Q

Mini Mental State Exam score: 24

A

Minimum score to avoid being classified as having a cognitive impairment

106
Q

Mini Mental State Exam score: 30

A

Highest score = cognitively within normal limits

107
Q

11 question measure that assesses five areas of cognition: - orientation - registration - attention/calculation - recall - language

A

Mini Mental State Exam - 5 minutes to complete - Scores range from 0-30

108
Q

This tract is commonly assessed through “heel walking”

A

Corticospinal tract: - Damage to this tract results in: – positive Babinski – absent superficial abdominal and cremasteric reflexes - loss of fine motor or skilled voluntary movement

109
Q

Reticulospinal tract responsible for:

A

facilitation or inhibition of voluntary or reflex activity through the influence on gamma and alpha motor neurons

110
Q

Rubrospinal tract is responsible for:

A

Motor input of gross postural tone, facilitating activity of flexor muscles, inhibiting activity of extensor muscles

111
Q

Tectospinal tract is responsible for:

A

Contralateral postural muscle tone associated with auditory/visual stimuli

112
Q

ASIA: - No motor or sensory function is pre- served in the sacral segments S4 to S5.

A

ASIA A: Complete

113
Q

ASIA: - Sensory but not motor function is preserved below the neurological level and includes the sacral segments S4 to S5.

A

ASIA B: Incomplete

114
Q

ASIA: - Motor function is preserved below the neurological level, and more than half of key muscles below the neurological level have a muscle grade less than 3.

A

ASIA C: Incomplete

115
Q

ASIA: - Motor function is preserved below the neurological level, and at least half of key muscles below the neurological level have a muscle grade of 3 or more.

A

ASIA D: Incomplete

116
Q

ASIA: - Motor and sensory function is normal.

A

ASIA E: Normal

117
Q
  • Ipsilateral paralysis and sensory loss and ipsilateral loss of proprioception, light touch, and vibratory sense. - Contralateral loss of sense of pain and temperature
A

Brown-Sequard - The ipsilateral loss of proprioception, light touch, and vibratory sense is due to damage to the dorsal column; - Ipsilateral paralysis results from damage to the lateral corticospinal tract. - Damage to the spinothalamic tracts results in contralateral loss of sense of pain and temperature.

118
Q
  • Loss of motor function and loss of the sense of pain and temperature below the level of the lesion. - Proprioception, light touch, and vibratory sense are generally preserved.
A

Anterior cord syndrome: - Frequently related to flexion injuries of the cervical region with resultant damage to the anterior portion of the cord and/or its vascular supply from the anterior spinal artery.

119
Q
  • Characteristically more severe neurological involvement of the upper extremities (UEs) than of the LEs - Varying degrees of sensory impairment occur but tend to be less severe than motor deficits
A

Central Cord Syndrome - Generally occurs from hyperextension injuries to the cervical region. - It also has been associated with congenital or degenerative narrowing of the spinal canal.

120
Q

Muscles necessary for crutch use:

A
  • Shoulder depressors and extensors, and elbow extensors
    • Lower trapezius
    • Latissimus dorsii
    • (pectoralis major)