NEB Flashcards

1
Q

How does NEB occur

A

lactation drains body reserves. glucose vital from brain and milk yield, causes gluconeogenesis and lipolysis = lots of problems

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2
Q

what is primary ketosis

A

AKA starvation ketosis, sometimes d/t specific nutritional deficiencies = excessive breakdown of fat and muscl

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3
Q

what is secondary ketosis

A

any condition that makes a cow loose appetite - LDA/RDA, lameness, endometritis, mastitis

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4
Q

what are the 2 forms of ketosis

A
wasting = reduced appetite, reduced weight, firm dry faeces, bouts of staggering
neuro = depraved appetite, licking, chewing, walks in circles, head pushing, hyperaesthesia
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5
Q

dx of ketosis

A

serum NEFAs elevated, lower blood glucose, elevated BHBA, milk and urine ketone levels

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6
Q

tx for ketosis

A

iv dextrose 200g slow drip, propylene glycol 250mlx2/d - can also try corticosteroids, appetite stimulants

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7
Q

signs of fat mobilisation syndrome

A

BCS>4, rapid decline in BCS, recumbent, neuro signs? concurrent illness, inappetance d/t fat mobilisation as NEFAs deposited in liver

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8
Q

signs of pregnancy toxaemia

A

fat animal, last few weeks of gestation, d/t reduced feed intake, mainly in cattle and sheep = separated animal, recumbent, depressed, constipated, can be related to parasitism

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9
Q

tx of pregnancy toxaemia

A

supplementary feeding and early tx w propylene glycol, drench w rehydration solution, induce parturition? provide high quality feed, IVFT?, consider insulin, steroids, choline…

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10
Q

prevention of pregnancy toxaemia

A

prevent fat or skinny animals, good quality food, transitional diets, careful husbandry!

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