Module 2: Stroke Flashcards
Carotid endarterectomy /stents
Start in urinary tract
Place filter to keep things out of your blood stream
use a ballot blow up artery
leave in metal piece to hold open
Blood to the brain:
Posterior cerebral artery: vision
Central cerebral artery: language
Interior cerebral artery: executive functioning
What is Aphasia?
disorder that results from stroke Acquired Neurogenic Language based Not a Problem of sensation, motor function, or intellect
How many people have Aphasia?
~ 1 million Americans
What causes Aphasia?
Damage to the language-processing areas of the cerebral cortex Stroke Traumatic Brain Injury Brain tumor Infections Toxic exposure
Who typically acquires Aphasia?
It’s most common among the elderly…
… but can effect anybody
Brief Overview of the History of Aphasia
Major debate in the field of aphasia- Localism vs. holism:
do neurons and brain areas have specific functions (localism) or is it fairly undifferentiated, working more as an aggregate field (holism)? Localists-mental abilities are localized to specific areas of the brain Franz Gall (1751-1828) (localist)
Paul Broca 1861-observation of 2 patients (Localist)
1861 Conclusions:
We speak with our left hemisphere
Broca’s area: Special faculty of articulated language
Term of aphemia proposed: Aphasia
But, in 1864 the term aphasia was coined.
AND the “auditory center” localized to the temporal lobe
Carl Wernicke
1874
Observed that patients with damage to left temporal lobe had difficulties with speech and understanding
Hypothesis: An area in left temporal lobe responsible for speech processing
Damage=sensory aphasia
Different areas of the brain (centers) support different language functions
These centers are connected by neural pathways
Linguistic information is transmitted across these pathways
Brodmann’s areas
there are 52 distinct areas in the brain. How the cell are organizes in the brain
Speech output 44
Auditory comprehension:
began loclalistic approach to how the brain functions
Current thinking
extreme localation has been replaced by “connectionism”
How to Distinguish Aphasia Types
Naming Problems : The hallmark of aphasia
Fluency of expression
Are they able to easily produce words and sentence, how is their grammar
Language Comprehension: to that degree is understood
Repetition: word, and phrase
Additional Considerations: in distinguishing
Motor output: Are the motor systems involved with speech affected? This indicates a concomitant motor-speech disorder
Reading and writing: To what extent is reading and writing affected? This usually reflects the overall impact of aphasia on language more generally
often mirrors fluency of expression
Naming
Ability to retrieve and produce a targeted word
Anomia: disturbance in the ability to name
Paraphasias (patterns of speech errors):
Phonemic: substitution or transposition of the targeted phoneme
e.g. “paker” for “paper”; “fable” for “table”
Semantic: error is related or in the same category but is incorrect
e.g. “cat” for “dog”
Neologic paraphasia: nonsense words substituted for target word
e.g. “sparndle” for “fork”
seen more in fluent aphasia
Fluency
Expression of thoughts using a smooth, uninterrupted flow and rate of speech
how many words can you put together in one breath
7 or more words in phrase lenth
Fluent aphasia:
spontaneous speech flow with adequate phrase length
posterior brain damage (temporal/parietal regions)
Non-fluent aphasia:
diminished phrase length, slowed or labored speech production, grammatical errors anterior brain damage (frontal lobe) Language Comprehension Ability to understand spoken messages Influenced by: Amount of information Frequency of word usage Personal relevance of information Part of speech
Repetition
Ability to accurately reproduce verbal stimuli
Receive and process incoming stimulus
Convey the information to regions of brain that formulate and plan motor sequence for speech
Articulate to reproduce the initial stimulus
Reading and Writing
Written language disturbances usually parallel spoken language impairments
Physical impairments
Hemiparesis Hemiplegia Hemisensory impairment Hemianopsia Dysphagia Seizure disorders or epilepsy
Psychosocial changes
Behavior
Mood swings
Depression
Non-Fluent Types:
anterior part affected
Broca’s Aphasia
Transcortical Motor Aphasia
Global Aphasia
Marked to severe deficits in all aspects of language (Non fluent)
Speech nonfluent, may be limited to stereotypy
Overlearned, automatized sequences may be preserved
——counting, days of the week, singing
Result of lesion covering a large portion of perisylvian area, often caused by total occlusion of left Middle cerebral artery
May be two lesions, one anterior, one posterior
May also result in (non fluent)
severe motor and sensory deficits involving right side
Visual field defects
Oral, ideomotor, ideational apraxia