Module 2 Flashcards

(135 cards)

1
Q

what is CMT

A

unilateral shortening of the SCM and fertile rotation away from involved side

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2
Q

what side is CMT named for

A

shortened side

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3
Q

what nerve is involved with CMT

A

spinal accessory nerve

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4
Q

insertion and origin of CMT

A

mastoid process and nuchal line
sternal head and clavicular head

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5
Q

what is CD

A

distortion of the skull shape
can be pre or post natally

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6
Q

how many infants with CMT have CD

A

90 percent

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7
Q

what does CD increase risk of

A

facial, ear, or mandibular asymmetry

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8
Q

what can cause CMT prenatally

A

ischemic injury or head position

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9
Q

what can cause CMT perinatally

A

birth trauma from Breech or assisted delivery

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10
Q

what can cause CMT postnatally

A

hip dysplasia, CD, or positional preferences

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11
Q

what happens with CMT with sternomastoid tumor? what can be present?

A

excessive fibrosis, hyperplasia, atrophy
nodules may be present

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12
Q

what to do if treating an infant with SCM nodules

A

refer to surgery

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13
Q

factors that influence CMT prognosis

A

age of referral, severity of ROM limitations, thickness of nodules, interventions

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14
Q

what can worsen CD postnatally

A

positioning- supine
CMT

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15
Q

factors associated with CD

A

male, first born, forceps/vacuum delivery, supine position

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16
Q

bradycephaly CMT

A

wide medial-lateral

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17
Q

plagiocephaly CMT

A

long anterior- posterior

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18
Q

grading for CMT

A

1-8 (higher is worse)
1-3 between 0-6 months
4 and 6 between 7-9 months
5 between 10-12 months
7 between 7-12 months
8 is older than a year
higher stages have more nodules

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19
Q

grading for CD plagiocephaly

A

1-5

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20
Q

grading for CD brachycephaly

A

1-3

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21
Q

how can families help with therapy at home for CMT?

A

put toys on sides that the child is tilted away from
get them to look other direction
facilitate wanted movement

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22
Q

spine involvement with CMT?

A

changes rotation - curved on opposite side

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23
Q

what is craniosynostosis?

A

sutures fuse and if they fuse early the skull cannot expand
important to differentiate from CD

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24
Q

who can get acquired CMT?

A

ocular lesion, benign paroxysmal torticolis, dystonic syndrom, infections, arnold-chiari malformation

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25
what to assess during PT exam for CMT?
cervical PROM and AROM prone tolerancee gross motor function pain cervical strength integumentary eval craniofacial for asymmetries
26
what to assess during PT exam for CD?
cranial shape cervical AROM
27
prognosis for CMT treated at <3 months? 3-6months? 6-18 months?
100% 75% 30%
28
two factors that help with resolution of CD?
parent education on repositioning helmet
29
is CMT causal with scoliosis?
no but it is associated
30
helmet protocols for CD
start at 4-6 months wear for 20-23 hours a day for 2-7 months
31
how does botox help with CMT?
relax SCM by inhibiting EACh release or causing atrophy
32
discharge criteria for CMT
full PROM of neck, trunk, extremities within 5deg of unaffected side symmetrical movement appropriate GM development no visible head tilt proper family understanding to maintain gains
33
highest rates of injury per 1000 hours of game in women
basketball, cross country
34
highest rate of injury based on athletic exposure
cross country for males, soccer and cross country for females
35
who has highest rate of injuries
young males as they age
36
why do males have higher rate of injury
higher body mass can develop greater force
37
what sport has highest number of catastrophic injuries
football
38
who has higher risk of ACL injury
girls 2-10x
39
purpose of PPE for sports participation
determine general health identify medical contraindications to participation identify sports that can be safely played general health screen fulfill elgal and insurance requirements evaluate physical maturation
40
preseason training program should include
identification of strengths and limitations individualized training body function assessment QoL assessment
41
risk factors for sports injuries
training error, muscle tendon imbalance, anatomical malaligment, improper footwear, surfaces, associated diseases, growth factors
42
two types of fractures
stress growth plate
43
osgood-schlatter
repeat injury at tibial tubercle
44
most common joint injuries in kids
ligamentous sprains
45
who should kids see if they have a concussion
vestibular sports med
46
diagnosis of concussions includes at least one of the following
period of loss of or decreased consciousness loss of memory altered mental state headache, dizziness, nausea, vomiting, sensitivity to light or sound
47
concussion assessment options
BESS, sensory testing, neck ROM/ strength, posture assessment, ocular testing, cardiovascular testing
48
how often do cervical injuries happen and what is likelihood of death
1-5% of sports injuries but 50-100% death
49
ICF framework for kids?
Family Friends Fun Function Fitness
50
ITW inclusion critera
kids older than 2 walking on toes at least 25% of time no known medical cause
51
typical walking pattern for ITW
forward trunk, flat foot, step by falling
52
3 phases of stance
1st rocker: PF and heel strike 2nd rocker: forward translation of tibia 3rd rocker: push off
53
things that can contribute to ITW
genetics neurological conditions at birth sensory processing neuropsychiatric
54
posture and ITW
will have poor postural stability
55
can you keep seeing child for ITW if you find yellow or red flag?
yes but refer to neuro or ortho too
56
toe walking assessment tool is called?
williams
57
is williams a diagnostic tool
no but it can help you rule out other causes of toe walking
58
what are some potential MSK problems if ITW is not treated?
ankle injuries and foot pain decreased gross motor activities
59
signs to refer kid out with ITW
heel cord tightness GM delays pain impaired QoL
60
important to remember when treating pediatrics and developing POC
discuss with families! work together
61
systems review to include in exam for ITW
pain integumentary sensory processing neuro parent concerns
62
postural assessment things to look at in NWB for ITW?
leg length thigh foot angle hindfoot alignment
63
what is the gold standard for gait assessment
gait assessment lab
64
what to use for ankle foot assessment (questionnaire)
Oxford foot and ankle questionnaire
65
what to include in exam for ITW
body structure/function, activity limitations, and participation restrictions and QOL
66
common interventions for ITW
PT orthotic management serial casting botox surgery
67
PT interventions for ITW
stretching, strengthening, joint mobs, locomotor/treadmill training, Motor control, taping, sensory based, balance, augmented auditory feedback, vibration, visual training, serial casting, orthotics
68
goals of treatment for ITW
10 deg ankle PROM DF and KE, core and LE strengthening, posture and balance training, locomotor training, orthotic management
69
how to strengthen for ITW
anterior tibial- heel walk, DF, incline walk hip and knee extensors, core and pelvic stabilizers
70
posture control for ITW
weight shifting
71
goals of PT intervention for ITW
address impairments and max function for pain free participation in home school and community activities
72
goals of orthotics for ITW
foot and ankle alignment
73
when to consider orthotics for ITW
toe walking more than 25% of time
74
orthotics options for ITW
AFOs SMOs foot orthoses carbon foot footplates (CFOs)
75
what does serial casting help with and what can be used with it
ankle DF range of motion botox
76
how long is serial casting for? what do you need to do after
4-6 weeks rebuild strength after
77
when is serial casting recommended
less than 0 degrees of ankle DF KE
78
how does botox work
prevents release of acetylcholine at NMJ which prevents muscle contraction
79
outcomes of ITW
improve ankle ROM/ DF improve gait
80
does botox alone improve ROM or gait
no
81
what is a second skeletal abnormality of ITW
external tibial torsion
82
who gets DMD
males- X linked recessive
83
who gets SMA
males and females- autosomal recessive chromosome 5
84
what causes DMD
deletion of dystrophin gene
85
onset of DMD
1-5 yo
86
what is increased in DMD
creatine kinase
87
what other organs impacted in DMD
heart brain and smooth muscle
88
what is seen in a child with DMD
muscle weakness pseudo hypertrophy slow 10m WT
89
what does a 10WT of 12+ seconds indicate in a child with DMD
loss of ambulation within 1 year
90
ROM findings in child with DMD
tightness of gastroc-soleus complex and TFL first
91
posture of child with DMD
lordotic posture with scapulae winging and scoliosis
92
5 stages of DMD
presymptomatic early ambulatory late ambulatory early non ambulatory late non ambulatory
93
age and clinical signs of early ambulatory stage of DMD
5 yo clumsy and falling proximal LE weakness calf pseudohypetrophy Gowers maneuver
94
clinical signs of late ambulatory stages of DMD
increased LL + trendeleberg stair issues heel cord, ITB, and hip contractures
95
age and clinical signs of early non ambulatory phase of DMD
10-12yo UE geetting weaker scoliosis risk pulmonary function decreasing d/t low strength in trunk and diaphragm
96
clinical signs of late non ambulatory phase of DMD
UE is weaker and posture is bad UE and LE contractures cardiac and respiratory failure
97
why do joint contractures occur in DMD
loss of AROM sedentary time in flexed posture muscle imbalance fibrotic chances
98
PT treatment for contractures
frequent stretching orthotics serial casting dynamic and solid AFOs
99
incidence of scoliosis in DMD
63-90%
100
why is scoliosis bad with DMD
rapidly progressing decreases VC loss of functional status
101
activity outcome measures for DMD
6mwt, timed testing 9 hole peg
102
how frequent to reassess kids with DMD
4-6 months
103
motor function scales to use for DMD
NSAA PUL
104
goal of PT for early ambulatory phase
keep muscles supple and prevent / minimize tightness
105
goal of PT for late ambulatory phase of DMD
keep muscles flexible and minimize muscle weakness
106
goal of PT for early non ambulatory phase of DMD
UE range of motion and flexibility accommodations and adaptive equipment
107
goal of PT for late non ambulatory phase
UE flexibility and supportive equipment
108
general exercise recommendations for DMD
regular submit aerobic exercise avoid high resistance and eccentric contractions
109
meds for DMD
corticosteroids to improve strength and function
110
what is the 2nd most common NMD
SMA
111
what causes SMA
genetic mutation effecting motor neurons
112
three types of SMA and severity and life expectancy and prevalence
1: most common and severe, limited life expectancy 2: 2nd most common and severe, short life expectancy 3: least common and severe, normal life expectancy
113
age of onset for each SMA type
1: 0-4 months 2: 6-12 months 3: 1-10 yrs
114
activity outcome measures for SMA
6mwt timed testing timed towers timed 10m
115
what to address in non sitters and sitters with SMA
muscle weakness postural control contractures
116
what to address in walkers with SMA
muscle weakness and asymmetries
117
outcome measure for non sitters SMA
CHOP intend
118
outcome measure for sitters SMA
upper limb function hammersmith
119
outcome measures for walkers SMA
6mwt
120
precautions for SMA
scoliosis
121
what is blount's disease?
tibia vara (proximal tibia)
122
what is the etiology of blount's disease?
unknown may be due to altered endochondral ossification
123
what happen's with blount's disease
decelerated growth at posterior-medial proximal tibial physics and causes various deformity of tibia
124
what are the 3 clinical classifications of blount's disease?
infantile: 0-4yo juvenile: 4-10yo adolescent: 10+ yo
125
what to observe with Blount's disease?
radiographic classification metaphyseal- diaphysial angle and alignment (uni or bilateral)
126
activity limitations of blount's disease
decreased activity, ambulation, and mobility pain
127
conservative treatment for blount's disease
orthotics- young and unilateral involvement HKAFOs, KAFOs, elastic bracing
128
surgical treatment for blount's disease
guided growth tibial osteotomy LLD
129
PT goals of bracing with Blount's disease
gait re-ed strengthening and stretching hip and core strengthening balance and coordination weight loss
130
goals of PT post surgery for Blount's disease
return to normal activities ASAP and prevent secondary compensatory problems
131
5 attributes to typical walking
stability in stance sufficient foot clearance in swing appropriate positioning for initial contact adequate step length energy conservation
132
neuro pre reqs for gait
CPGs for muscle firing muscle synergies coordination
133
biomechanics pre reqs for gait
ROM strength bone structure and body composition management of gravity forces proper muscle and joint kinematics typical growth
134
five determinants for mature walking
duration of single limb stance walking velocity cadencee step length ratio of pelvic span to ankle spread
135