Modalities Ch. 6 Flashcards
orthopedic injury model
normal tissue
- cells
- two blood vessels
- nerve
immediate ultrastructural change
primary traumatic damage
damage to the nerve
breakdown of the injured cells
hemorrhage
few minutes only
clot forms, stopping hemorrhage
what happens of a clot forms
hematoma forms
how does a clot form
fibrins form a network
-platelets are caught and it created a plug
pain from damaged nerve
-results
muscle spasm and more pain
neural inhibition
-leads to decreases in strength and ROM
the damaged cells release…
chemical mediators as a signal to the body that an injury has taken place
result of broken blood vessels
extravascular swelling
chemical mediators released from dying cells cause…
hemodynamic changes permeability changes -histamine -kinins -serotonin leukocyte migration
what happens at this point?
secondary enzymatic injury
hemodynamic changes
blood flow slows down OR blood flow ceases result -tissue oxygen decreases --hypoxia --metabolic changes -secondary hypoxic injury soon seen
phagocytosis
as cells are broken down free proteins are released
free proteins signal edema
pressure on undamaged nearby pain fibers cause additional…
pain
muscle spasm and inhibition
immediate care
subset of acute care
first 12 hours
RICES
Rest Ice Compression Elevation Stabilization
why RICES?
protection from further damage decrease or minimize the development of -swelling -pain -muscle spasm -neural inhibition -secondary injury -total injury (because of decreased secondary injury)
time course of swelling
immediate swelling -comes from hemorrhaging edema -begins minutes to hours after injury -continues to develop over many hours
secondary injury and edema
secondary injury leads to edema
increased edema increases secondary injury
two mechanisms
-increased distance between blood vessel and tissue cells
-edema can compress the blood vessel
both decrease circulation
-decreased oxygen to tissues