MOD E Tech 32 Head & Maxillofacial injuries Flashcards
Head Anatomy
Intracranial Contents:
Intracranial Contents:
•
- Brain – 80%
- Blood – 12%
CSF – 8%
Head Trauma - Types
Open
•Skull compromised
and brain exposed
Closed
•Skull not compromised
and brain not exposed
•Contra Coup injury
Head Injuries – Scalp Wounds
Scalp wounds
- Highly vascular, bleeds briskly
- May be difficult to pinpoint the source
- Patient may suffer concussion
Base of Skull Fracture
signs
Brain Injury
Primary brain injury
•Immediate damage
due to force
- Coup and contracoup
- Fixed at time of injury
- Localised
- Diffuse
Management
•Directed at prevention
Brain Injury
Secondary brain injury
Secondary brain injury
•Results from hypoxia, hypercarbia
or decreased perfusion
- Response to primary injury
- Develops over hours
Management
•Good prehospital care can help prevent
Brain Injury
Response to injury
Response to injury
- Swelling of brain
- Vasodilatation with increased blood volume
- Increased ICP
- Decreased blood flow to brain
- Perfusion decreases
- Cerebral ischemia (hypoxia)
Early efforts
to maintain brain perfusion
can be life-saving.
Brain Injury - Contusion
Cerebral contusion
- Bruising of brain tissue
- Swelling may be rapid and severe
- Level of consciousness
•Prolonged unconsciousness,
profound confusion or amnesia
- Associated symptoms
- Focal neurological signs
- May have personality changes
Brain Injuries – Vascular Damage
Intracranial haemorrhage
- Epidural / Extradural
- Between skull and dura
- Subdural
- Between dura and arachnoid
- Intracerebral
- Directly into brain tissue
Intracranial Haemorrhage
Intracranial Haemorrhage
Acute epidural haematoma
Intracranial Haemorrhage
Acute subdural haematoma
Consequences & Complications of Head Injuries
Consequences & Complications of Head Injuries
Compression
- Extradural and sudural haematomas can cause a sudden or gradual increase in ICP
- Large pressure changes can result in shifting of the brain and “coning” of the brain stem
- A deterioration in level of consciousness is an important indicator of an increasing ICP
- Deterioration in LOC
- Noisy, slow breathing (Cheyne-Stokes in terminal stages)
- Slow, full pulse
- Raised temperature
- Hot, flushed face
- Unequal pupils – dilated and fixed on side of injury
- Unilateral facial or body weakness / paralysis
- Intense headache
- Convulsions