Minerals Flashcards

1
Q

Macro vs micro minerals

A

required in macro concentrations (gram amounts)
Required in micro concentration (mg amounts, mg/kg)

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2
Q

What are the 7 macro minerals

A

Ca, P, Na
Mg, K, Cl, S

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3
Q

What factors influence optimum trace mineral status

A

Bioavailability of TM- supplemental TM, antagonist minerals, synergistic minerals/vitamins

Demand for TM- Physiological status, disease challenges, environmental stressors

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4
Q

What is a mineral antagonism?

A

compete for binding sites and absorption

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5
Q

What is bioavailability?

A

the fraction of the ingested nutrient that is utilized for normal physiological functions or storage

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6
Q

Physiological factors that influence bioavailability

A
  1. Species and genetics
  2. Age and sex
  3. Metabolic function (growth, lactation, maintenance)
  4. Nutritional status
  5. Intestinal or Rumen Microflora
  6. Physiological stress
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7
Q

Extrinsic factors that influence bioavailability

A
  1. Solubility of element
  2. Binding to other dietary components
  3. State of oxidation
  4. Competitive antagonisms
  5. Chelate effects
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8
Q

Organic vs inorganic mineral

A

Inorganic: bound to sulfate, chloride, carbonate, or oxide
Organic: bound to carbohydrate or protein

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9
Q

What is a mineral chelate?

A

Chemically attached to an organic molecule

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10
Q

Why is it important to know intake when making mineral purchases?

A

Decrease costs, avoid toxicity

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11
Q

Electrolyte macrominerals

A

important in acid-base balance, water balance, muscle contractions, nerve impulses

Na, Cl, K

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12
Q

Na/K Pump

A

maintains osmotic pressure
3 Na to extracellular
2 K to intracellular

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13
Q

Cl function

A
  1. balances Na in extracellular fluid with bicarbonates
  2. form HCl for gastric secretion
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14
Q

Ca deficiency

A

Milk fever, also called parturient paresis. Result is paralysis after calving

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15
Q

What might cause a cow to become Ca deficient?

A

Increased Ca demand for milk production

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16
Q

Where can Ca come from to meet requirements?

A
  1. Absorption from GI tract
  2. mobilization from bone
  3. resorption from kidney
17
Q

How can milk fever be prevented? How is it treated?

A

Prevent by feeding a greater amount of anions prior to calving
Treat with intravenous administration of Ca gluconate

18
Q

What is urinary calculi?

A

imbalance of Ca and P (too much P) in sheep. P form kidney stones, impacts urination, bladder fills up with urine

19
Q

What is grass tetany and when is it likely to occur?

A

Magnesium deficiency causes muscle weakness (grass staggers)
Occurs during early lactation

20
Q

What happens during sulfur toxicity? How does it progress to PEM?

A

High sulfur in diet converted to H2S gas, burped and inhaled.
From there it crosses blood brain barrier, interferes with TPP, and causes PEM

21
Q

PEM

A

acute signs of polioencephalomalacia is blindness, seizures, comatose

22
Q

What part of the US is selenium deficient? Toxic?

A

Indiana, Ohio
Midwest

23
Q

Se deficiency

A

White muscle disease

24
Q

Se toxicity

A

Hair loss
Hoof deformities
Weight loss
Diarrhea

25
Q

What is the legal level of Se supplementation

A

0.3 ppm

26
Q

I function

A

constituent of T3 and T4
associated with basal metabolic rate

27
Q

I deficiency

A

Reduced BMR
Dry skin, brittle hair
Goiter (enlarged thyroid)

28
Q

I toxicity

A

overactive metabolism, increased BMR

29
Q

How does nitrate toxicity progress?

A
  1. NO3 reduced to No2
  2. alters iron in hemoglobin (Fe2+ to Fe3+)
  3. cannot transport oxygen
  4. death
30
Q

When is nitrate toxicity likely to occur?

A

accumulate nitrate (NO3) in forages or water