MED/SURG EXAM #4 Flashcards

1
Q

Thyrotoxicosis manifestations

A

Bulging eyes
Skinny

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2
Q

Myxedema coma manifestations

A

– Puffy
– Slow metabolism
– Fatigue

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3
Q

Foods to take when diabetic glucose is low

A

– 8oz SKIM milk
– 3 graham crackers
– 4oz orange juice
– 1 TBS honey
– 4 tsp sugar

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4
Q

Healthy snacks for person with Addison’s disease

A

– Turkey and cheese sandwich –

NOTE: want diet with low K and high Na, carbs, and protein

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5
Q

Foods HIGH in Potassium (K+)

A

– Bananas
– Baked potatoes
– Plain yogurt w/ peaches

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6
Q

Grave’s disease

A

– Most common of hyperthyroid
– T3, T4 = high; TSH = low

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7
Q

Hashimoto’s disease

A

– Excessive hypothyroidism

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8
Q

Metabolic Syndrome

A

– RF for diabetes
– hypercholesteralemia
– enlarged waist size

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9
Q

Which of the following clients are at risk for developing Cushing’s Syndrome?
a. A client w/ a tumor on the pituitary gland, which is causing too much ACTH to be secreted
b. A client taking glucocorticoids for several weeks
c. A client with a tuberculosis infection
d. A client who is post-op from an adrenalectomy

A

B. A client taking glucocorticoids for several weeks
– DO NOT stop abruptly b/c may cause

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10
Q

Addison’s Disease

A

A decreased secretion of aldosterone and cortisol
– HYPONa+, HYPERK+, decreased weight, HYPOglycemia, HYPERCa2+

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11
Q

Primary vs Secondary etiology of Adrenocortical Insufficiency

A

1~: Addison’s disease; reduction of glucocorticoids, mineralocorticoids, and androgens

2~: lack or pituitary ACTH; lack of glucocorticoids and androgens

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12
Q

Clinical Manifestations: Graves Disease

A

Watch out for cardiac concerns

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13
Q

Addison’s FIVE S’s

A
  1. Sudden pain in back, legs, stomach
  2. Syncope
  3. Shock
  4. Super low BP
  5. Severe diarrhea, H/A, vomiting

NOTE: Think “tan, skinny, betch!*

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14
Q

Causes of Addison’s

A

– autoimmune (adrenalitis)
– idiopathic
– surgery
– infection (TB)

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15
Q

Acute Thyrotoxicosis

A

Thyroid storm
– Severe tachy, heart failure
– Shock
– Hyperthermia (up to 106.8F)
– Agitation
– Seizures
– Abdominal pain
– Delirium, coma

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16
Q

Diagnostic tests for acute thyrotoxicosis

A

Decreased TSH (less than 0.4 mU/L)
 Increased free thyroxine (free T4)
 Total T3 and T4 (not definitive)
 Radioactive iodine uptake (RAIU)
 Distinguishes Graves’ disease from other forms of thyroiditis

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17
Q

Medication treatment for Addison’s Crisis

A

IV Solu-Cortef – cortison needed NOW
– IV + glucocorticoid to make patient stable
1. NS w/ D5 = increase Na+ and K+
2. Hydrocortisone sodium (100-300mg) OR Dexamethasone (4-12mg IV bolus)
3. 100mg Solu-Cortef by continuous IV infusion over next 8hrs
4. Hydrocortisone (80mg) IM w/ H2O q12hr
5. IV H2 histamine blocker to reduce ulcer irritation (e.g. Ranitidine)

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18
Q

Client has elevated temp +tachy w/ palpitations + HYPERthyroidism. What to do?

A

Thyroid storm
– Necessitates aggressive treatment
 Give medications that block thyroid hormone production
and SNS
 Monitor for dysrhythmias
 Ensure adequate oxygenation
 Fluid and electrolyte replacement

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19
Q

RN Implementation for Acute Thyrotoxicosis

A

– Ensure adequate rest
– Cool, quiet room
– Light bed coverings (switch frequently b/c of they’re hot)
– Encourage and assist w/ exercise

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20
Q

Postoperative Care for HYPERthyroidism

A

Hypothyroidism
 Hypocalcemia
 Hemorrhage
 Laryngeal nerve damage
 Thyrotoxicosis
 Infection

Have IV Calcium ready

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21
Q

Treatment for HYPOthyroidism

A

Synthroid Levothyroxine
Complication that would cause a concern: Tachycardia, high temp, insomnia –> S/Sx of HYPERthyroidism

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22
Q

HIGH Ca2+ manifestations vs. LOW

A

HIGH: Tingling of mouth and extremities; at risk for tetany (oral paresthesia)
LOW:

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23
Q

Levothyroxine to Tx HYPOthyroidism, TSH levels?

A

TSH = will reduce/slow down (b/c thyroid being stimulated b/c not enough in body)
T3, T4 = increased

24
Q

True or False: You aspirate insulin shots into the subcutaneous fat.

A

False, not EBP anymore.

25
Difference between subclinical HYPOthyroidism vs. Nonthyroidal illness syndrome (NTIS)
Subclinical hypothyroidism  TSH is greater than 4.5 mIU/L  T4 levels normal  Affects up to 10% of women over 60  Nonthyroidal illness syndrome (NTIS)  Critically ill patients  Low T3, T4, and TSH levels
26
Primary vs Secondary HYPOthyroidism
Primary hypothyroidism -- Caused by destruction of thyroid tissue or defective hormone synthesis Secondary hypothyroidism -- Caused by pituitary disease (decreased TSH) or hypothalamic dysfunction or (decreased TRH)
27
Diagnostic studies for HYPOtyroidsim
-- History and physical examination TSH and free T4: -- TSH increases with primary hypothyroidism -- TSH decreases with secondary hypothyroidism Thyroid antibodies: autoimmune origin LABS -- Elevated: cholesterol, triglycerides, creatine kinase -- Low: RBCs (anemia)
28
Cushing's Manifestations
**S**kin fragile **T**runcal obesity **R**ound face ("moon face") **E**cchymosis w/ increased BP **S**triae on extremity (purple stretchmarks) **S**ugars increased **E**xcessive body hair **D**orsal cervical fat pad **D**epression LABS: HYPERNa+, HYPERglycemia, increased carbohydrates, HYPOK+
29
Difference b/w Addison's vs. Cushing's
_Cushing's:_ increased secretions of cortisol (stress hormone) _Addison's:_ decreased secretion of cortisol
30
Positive sign for Trosseau and Chvostek are indicated in which laboratory values?
Decreased Mg2+ Decreased Vitamin D Decreased Ca2+ NOTE: Trosseau is for *head* & Chvostek is for *face*
31
What type of insulin do we use to treat DKA?
32
List examples of 15 g of carbohydrates to treat hypoglycemia.
33
What is the onset of action for regular insulin?
34
List ways to prevent complications of diabetes.
35
Criteria to diagnose diabetes.
36
Locations for insulin injections.
37
Criteria to diagnose Diabetes Insipidus (what about specific gravity?)
38
Considerations for metformin.
39
Risk factors for diabetes.
40
When a client is sick and has diabetes, how often should they check their blood glucose?
41
Manifestations of Cushing’s.
42
Diet considerations for Cushing’s.
HIGH Na+ and LOW K+
43
Nursing considerations to manage Addison’s Disease & Addison’s Crisis.
44
Manifestations of hypoparathyroidism.
45
What happens to magnesium level in a client with primary HYPERparathyroidism?
Mg2+ level is increased Ca2+ level is increased P3+ level is decreased
46
Nursing considerations for client with hyperparathyroidism.
47
Manifestations of hypothyroidism.
Mg2+ level is decreased Ca2+ level is decreased P3+ level is increased
48
Post-op care & considerations following thyroidectomy.
Watch out for: hypothyroidism, airway compromising (ending in tracheostomy), check Ca2+ (parathyroid which controls might be nicked), assess for Chvosteks and Trousseau
49
What is the test to determine if the thyroid is overactive, appropriately active or underactive?
50
Why would a client need to add whole grains to their diet if they have hypothyroidism?
51
Risk factors for SIADH?
52
Manifestations of hypercortisolism.
53
Lab Values
Phosphate: 3-4.5 Calcium: 8.5-10.5 Ca & Phosphate = inversely related
54
What happens to phosphorous in a client who has hypoparathyroidism?
Increases in the blood (Phospohorous), but will decrease calcium levels
55
We encourage clients to taper steroids and not suddenly stop because of what possible syndrome?
56
Drugs to know: metformin, glipizide, prednisone, levothyroxine, vasopressin, regular insulin, NPH, glargine, hydrocortisone, hydromorphone