MCP Flashcards

1
Q

name structure

A

amylopectin

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2
Q

name structure

A

amylose

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3
Q

name structure

A

cellulose

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4
Q

name structure

A

galactose

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5
Q

name structure

A

fructose

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6
Q

name structure

A

glucose

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7
Q

name structure

A

glycogen

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8
Q

name structure

A

isomaltose

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9
Q

name structure

A

lactose

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10
Q

name structure

A

maltose

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11
Q

name structure

A

sucrose

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12
Q

name linkage

A

alpha linkage

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13
Q

name linkage

A

beta linkage (draw beta going up)

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14
Q

(L/D) sugars are more abundant biological structures

A

D

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15
Q

four major dietary carbs

A

amylose (starch, amylopectin (starch), lactose, sucrose (table sugar)

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16
Q

why can we not digest cellulose

A

we dont’ have an enzyme that cleaves a beta 1-4 linkage

17
Q

where is table sugar digested

A

in the small intestine by brush border enzymes

18
Q

where does starch digestion begin

A

in the mouth, with amylase

19
Q

increased levels of serum amylase probably indicates

A

pancreatitis

20
Q

the specificity of glycosidases is based on these three things

A

type of linkage, type of sugars, position of the linkage (terminal or internal)

21
Q

(starch/disaccharides) are cleaved in the mouth by salivary amylase

A

starch

22
Q

difference between lactase non persistence and lactose intolerance

A

lactase non persistence–enzyme is no longer expressed at high levels

lactose intolerance–symptoms

23
Q

NADPH is critical for (synthesis/degradation) pathways

A

synthesis

24
Q

gluconeogenesis makes glycogen from (monosaccharides/amino acids)

A

amino acids, lactate

25
Q

where is glycogen mainly stored

A

liver and skeletal muscle (skeletal muscle stores it for its own use)

26
Q

oxidation of glucose to generate ATP and pyruvate

A

glycolysis

27
Q

oxidation of glucose to 5 carbon sugar to produce NADPH needed for biosynthetic pathways

A

pentose phosphate pathway

28
Q

during fasting, a decrease in blood glucose causes a release of (insulin/glucagon) into the bloodstream to mobilize fuels and increase blood glucose

A

glucagon

29
Q

when glucagon is released into the bloodstream, lipolysis (increases/decreases)

A

increases, to release fatty acids to be used as alternative fuel

30
Q

during the fed state, an increase in blood glucose causes release of (insulin/glucagon) into the bloodstream to promote fuel storage

A

insulin

31
Q

when insulin is released, fatty acid synthesis (decreases/increases)

A

increases

32
Q

when insulin is released, triglyceride synthesis is (increased/decreased)

A

increased, to store fatty acids for later use