M25 - Abscesses Flashcards

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1
Q

Give dental abscesses synonyms.

A
  • Dentoalveolar abscess
  • Periapical abscess
  • Apical abscess
  • Chronic periapical dental infection
  • Dental pyogenic infection
  • Periapical periodontitis
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2
Q

Describe the development of dentoalveolar abscesses.

A
•  Carious lesion
•  Bacteria spread to pulp
–  via dentinal tubules
•  Acute inflammation
–  pulpitis
–  necrosis of the pulp
•  Chronic localised 
–  abscess
–  pulp remains viable
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3
Q

what causes a dentoalveolar abscess?

A
•  Traumatic fracture or tooth wear
•  Traumatic exposure during treatment
•  Via Periodontal membrane & root canals
•  Anachoresis
–  seeding via pulpal blood supply 
–  rare
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4
Q

what do dentoalveolar abscesses remain?

A
  • Acute or chronic

* tender to pressure

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5
Q

how does dentoalveolar access soft tissue?

A

– Direct spread

– Indirect spread

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6
Q

what are the symptoms of abscesses?

A
  • Pain
  • Swelling
  • Erythema
  • Suppuration
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7
Q

What are key factors of abscesses?

A
  • Number of virulent bacteria • Local and systemic immunity

* Anatomical damage

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8
Q

Name facultative anaerobes from dentoalveolar abscesses.

A
–  S. anginosus-group
•  (especially S. anginosus) 
–  S. oralis-group
–  Enterococcus faecalis
–  Actinomyces spp.
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9
Q

Name strict anaerobes from dentoalveolar abscesses.

A
–  Peptostreptococcus spp.
–  Porphyromonas gingivalis
–  Tanerella forsythia
–  Prevotella spp (10-87%).
–  Fusobacterium nucleatum
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10
Q

Describe the treatment of abscesses.

A
•  Specimen collection 
–  needle aspiration
(anearobes)
•  Local Management
–  Drain the pus (incision through root canal) - remove residual pus through incision
–  e.g. buccal sulcus
 •  Treatment
–  Amoxicillin or clarithromycin
–  Metronidazole
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11
Q

what is a periodontal abscess?

A

Infection of periodontium acute or chronic

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12
Q

what is the cause of periodontal abscesses?

A

– Occlusion of opening
prevents drainage
– Impaction of foreign objects

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13
Q

what are the symptoms of a periodontal abscess?

A

– Sudden onset
– Swelling
– Redness/tenderness
– May spread & destroy bone/soft tissue

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14
Q

What organisms are involved in abscesses?

A
•  GNABs
–  Porphymonas,
Prevotella
•  Streptococci –  variety
•  Others
–  Treponema,
Actinomyces,
–  F. nucleatum
–  Propionobacterium
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15
Q

Describe the extraction of of abscesses.

A

– severe disease
– poor prognosis
– recurrent infection

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16
Q

Describe the drainage of abscesses.

A

– gentle scaling
– irrigate with 0.9% saline
– antibiotics:
Penicillin, Erythromycin or metronidazole

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17
Q

What gram negative bacteria are associated with infection after root canal treatment?

A

– F. nucleatum
– Prevotella
– Campylobacter rectus

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18
Q

What gram positive bacteria are associated with infection after root canal treatment?

A
–  S. oralis, S. mitis, S.
anginosus, S. gordonii
–  Enterococcus faecalis
–  Candida albicans
–  Lactobacilli
19
Q

Describe features of enterococcus faecalis.

A
  • Facultative anaerobe
  • Common in intestine
  • Can be eradicated in small numbers
  • Difficulty comes with high levels
20
Q

What are key features of enterococcus faecalis?

A

– Adhere to collagen
– Persistence in nutrient poor environments
– Biofilm formation
– Resistant to calcium hydroxide & sodium hypochlorite
– Low-high pH range
– Salinity & temp resistance

21
Q

Describe ‘dry’ socket.

A
  • Localised infection
  • Following extraction the socket fails to heal
  • Sparse anaerobic infection
22
Q

what is the prophylaxis of ‘dry socket’?

A

chlorohexidine irrigation prior to & post extraction

23
Q

what is the treatment for ‘dry socket’?

A

antispeptic dressing & metronidazole

24
Q

Describe how abscesses are caused by dental implants.

A

• Endentulous treatment of dental implants
– surgical trauma (overheating of bone or compression of bone chips)
– persistence of root particles or foreign bodies
– infection of implant surface
(saliva & bacterial plaque)
– implant into infected site
• Immediate or delayed abscesses
– S. aureus (0.7-15%) & S. epidermidis (4-65%)
– Fusobacteria, anaerobic Streptococci
• Remove implant & antibiotic therapy

25
Q

What is Ludwig’s angina?

A

• Acute Cellulitis
• Bilateral infection
– sublingual & submandibular spaces

26
Q

What are the symptoms of Ludwig’s angina?

A

– Base of mouth & tongue
swell
– Brawny oedema & swelling of neck tissues
– Airway obstruction (asphyxiation)

27
Q

How often is there post extraction infection in Ludwig’s angina?

A

90% of cases

28
Q

what are the oral commensals of Ludwig’s angina?

A

– ß-hemolytic oral Streptococci (41%)
– Porphyromonas, & Prevotella, Fusobacteria,
– Staphylococci (27%-50%) & Enterococci

29
Q

what is the management of Ludwig’s angina?

A
•  Ensure airway remains open
–  surgical intervention
–  Drainage
_ parenteral hydration
•  High dose antibiotic treatment –  intravenous penicillin
–  ceftriaxone + metrinodazole
30
Q

What is the osteomyletis of the jaw?

A

Inflammation of medullary cavity of the mandible or the maxilla

31
Q

what are the symptoms of the acute osteomyelitis of the jaw?

A

pain, mild fever, loosening of teeth & exudate of pus through gingiva or sinuses of affected skin

32
Q

what are the symptoms of the chronic osteomyelitis of the jaw?

A

few symptoms, tender & indurated skin

33
Q

what bacteria can cause osteomyletis of the jaw?

A

• Normally endogenous oral flora;
– Tanerella, Prevotella & Porphymonas spp
– M. tuberculosis, & T. pallidium rarely

34
Q

what can occur in post radiation therapy of osteomyletis?

A

– necrosis of blood supply, reduced saliva flow
– Exogenous bacteria
• e.g. E. coli, Proteus, & Klebsiella

35
Q

Name 4 bacterial infections of the salivary glands.

A
  • Acute bacterial parotitis
  • Chronic bacterial parotitis
  • Recurrent parotitis of childhood
  • Submandibular sialadentitis
36
Q

what are the predisposing factors of acute bacterial parotitis?

A

– drugs (prescription)
– abnormalities
– generalised sialectasis

37
Q

what is the presentation of acute bacterial parotitis?

A

– swelling of parotid gland(s) – pain
– purulent secretions
– rarely fever & chills

38
Q

What is the microbiology of acute bacterial parotitis?

A

– S. aureus, oral Streps, Haemophilus & anaerobes

39
Q

what is the treatment of acute bacterial parotitis?

A

_ co-amoxyclav
– flucloxacillin, erythromycin
– salavation
• (increased fluid intake)

40
Q

Describe chronic bacterial parotitis.

A
•  Recurrent infections 
•  Damaged glands or
Sjorgen’s syndrome
•  Chronic nature can lead
to replacement fibrosis 
•  Destruction of gland
41
Q

Describe parotitis of childhood.

A
–  Observed prior to puberty 
–  Repeated acute episodes
–  Cause :
•  duct abnormalities
•  preceding mumps 
•  foreign body
•  trauma
42
Q

Describe submandibular sialadenitis.

A
–  Rare
–  similar to acute parotitis
–  calculi or strictures
–  treatment & micro 
•  as acute parotitis
43
Q

Give summary slide.

A
•  CMS information
–  Pulpitis microbiology
–  Abscesses in the oral environment
–  Salivary Gland infections
•  Microbiology
–  Assign likely
endogenous species
–  Infection evidence of underlying clinical problem that requires addressing once treated.