LP #108 Physiological Basis, Signs & Symptoms Of Hypertension, Orthostatic Hypotension & Circulatory Shock Flashcards
- chronic elevation of BP
- usually d/t increase PR
Hypertension
Hypertension categories
1st (Essential) -90-95% of all cases -chronic elevation of BP -idiopathic
2nd
- 5-10% of all cases
- d/t another condition, ex. Kidney dysfunction
Hypertension risk factors:
- Family history
- Age
- Race
- Insulin resistance/ metabolic abnormalities
- Obesity
- High salt intake
- Excessive alcohol consumption
- Dietary intake of potassium
Causes of hypertension
- kidney disease
- kidney deals w/fluid & electrolyte regulation
- vascular disorders
- increase PR or decrease bv diameter
1st Hypertension s/s
- fatigue
- nosebleeds
- headaches
- mild edema
2nd hypertension s/s
- nosebleeds
- severe headaches
- vomiting
- nausea
- edema
- visual disturbances
In severe causes: convulsions& coma
- low P system
- BP normally at 28/8 mmHg
Pulmonary circulation
- increase P pulmonary aa
- may result in cor pulmonale: hypertrophy or R ventricle
- usually 2nd to other conditions
- common w/ advanced respiratory disorders
Pulmonary hypertension
Pulmonary hypertension causes:
- increase L atrial P- transmitted back to pulmonary circulation
- increase pulmonary BF
- increase PR in pulmonary bv’s
- hypoxia
1st pulmonary hypertension:
- rare & lethal
- characterized by fibrosis of pulmonary aa & arterioles
- heart failure d/t a 1st lung disease & pulmonary hypertension
- hypertrophy of R ventricle d/t increase workload
- pumps blood against increase P in pulmonary aa
Cor pulmonale
- ABN decrease in BP when moving from lying down to standing:
- surge of blood to lower extremities abuses decrease in overall BP
- or ally, Baroreceptors & Vasomotor centre trigger vasoconstriction to increase BP
Orthostatic hypotension
Orthostatic hypotension causes:
- crease in BVol
- meds
- age
- prolonged bed rest
- ANS dysfxn
- idiopathic or assoc w/ neuro deficits
Orthostatic hypotension s/s
- vertigo
- syncope
- Shock: condition in which bv’s are inadequately filled & excessive decrease BF
- Impaired BF: CV system responds to maintain BVol & BP
- effectiveness depends on extent of BF impairment
Circulatory shock
- heart fails to pump blood sufficiently to meet body’s demands
- may occur as an end stage condition of coronary artery disease or cardiomyopathy
Cardiogenic shock
Cardiogenic shock may occur suddenly from a number of causes:
- myocardial infarction
- myocardial contusion
- sustained arrhythmias
- cardiac surgery
- large scale loss of BVol (most common)
- compensatory mechanisms:
- increase HR & CO
- increase respiratory rate
- activation of kidney BP mechanisms
- reflex vasoconstriction compensation
Hypovolemic shock
Hypovolemic shock causes:
- hemorrhage
- diarrhea
- excessive burns
- excessive vomiting
Hypovolemic shock s/s
- thirst
- decrease skin & body temp
- weak pulse
- tachycardia
- decrease urine output
- restless & apprehensive (early)
- apathy & stupor (late)
- d/t obstruction in great vv, lungs o heart
- excessive decrease BF to structures distal to obstruction
- results in ischemia, hypoxia & cell death
Obstructive shock
- aka vascular shock
- inadequate circulation & tissue perfusion d/t excessive vasodilation
- leads to excessive decrease in PR & decrease BP
Distributive shock
Type of distributive shock
- neurogenic shock
- anaphylactic shock (anaphylaxis)
- septic shock (septicemia)
- d/t loss of Vasomotor tone
- causes include:
- brain injury
- hypoxia
- decrease blood glucose
- SCI
- s/s:
- decrease HR
- skin is warm & dry
Neurogenic shock
- d/t presence of excessive amounts of vasodilators in blood
- hypersensitivity immune rxn to allergens
- s/s include:
- abdominal cramps
- apprehension
- burning sensation
- uticaria
- pruritis
- dyspnea
Anaphylactic shock (anaphylaxis)
- d/t presence of bacterial toxins that cause vasodilation
- caused by severe bacterial infection
- s/s include:
- fever
- warm, flushed skin
Septic shock (septicemia)