Low Back Pathologies 3 Flashcards

1
Q

key characteristics of the skeleton

A

metabolically active

continuous remodeling throughout life with annual bone turn over

remodeling is necessary to maintain structure integrity of skeleton and to serve a metabolic function as a storehouse of calcium and phosphorus

bone mass peaks between 25-35

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2
Q

osteoporosis is a persistent progressive metabolic disease characterized by what

A

low bone mass

impaired bone health

decreased bone strength

enhanced risk of fx

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3
Q

types of osteoporosis

A

primary is most common; associated with ARJC, hormone, and calcium levels and PA

secondary is because of disease or meds

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4
Q

describe the incidence of osteoporosis

A

not often presenting

70% undiagnosed

increasing in number and expected to keep going up

MOST common metabolic disease

highest in post-menopausal women with estrogen deficit and Scandinavian ancestry

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5
Q

precursor to osteoporosis

A

osteopenia (low bone mass)

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6
Q

% of men/women who have osteoporosis

A

33% women and 20% men over 50 will suffer from osteoporosis

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7
Q

risk factors for osteoporosis

A

lower hormone levels; specifically estrogen and testosterone

genetics

social habits

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8
Q

how do estrogen and testosterone levels affect osteoporosis

A

estrogen limits bone resorption and aids in calcium absorption

associated with menopause and abnormal menses

testosterone also limits resorption

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9
Q

how can genetics affect osteoporosis

A

family hx

also plays a role with parathyroid hormone for calcium balance and smaller bone stature

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10
Q

what social habits can affect osteoporosis

A

> 2 beers or > 1 glass of wine or > 1 liquor shot or > 3 cups caffeine per day

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11
Q

risk factors for osteoporosis

A

physical inactivity

depression alters hormones

meds (>3 months corticosteroids)

tobacco

dietary dysfunction with vit D and calcium levels associated with eating disorders, low protein, SAD, or conditions that alter absorption of nutrients

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12
Q

when should pts get a DEXA scan

A

women at 65
men at 70

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13
Q

pathogenesis of osteoporosis

A

metabolic disorder

osteoclastic > osteoblastic activity

endocrine disorder that limit calcium regulating and sex hormones

loss of inner cancellous bone

wedging, compression, and fx of vertebral body most often in lower thoracic and upper lumbar regions

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14
Q

where are fxs most common

A

femurs, ribs, radius = common areas

nontraumatic > traumatic

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15
Q

objective changes that may indicate asymptomatic osteoporosis

A

FHP

loss of height

increased thoracic and lumbar kyphosis

fulcrums

rounded/slouched posture

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16
Q

when is it common for fxs to occur with osteoporosis

A

often with seemingly benign flexion activity

i.e. bending or sneezing

17
Q

S&S of osteoporotic fx

A

fx S&S plus

severe back pain mid thoracic/upper lumbar (especially with flx, compression, and valsalva stresses)

pain may refer to flanks/abdominal

18
Q

ROM and resisted testing for osteoporosis

A

ROM = pain and limits with flx but possibly all direction

resisted = pain and weakness, primarily with flx but possibly with all directions

19
Q

stress tests and neuro finding for osteoporosis

A

pain with compression and likely relief with distraction

pain with PA pressure

most often neuro is neg

20
Q

special tests for osteoporosis

A

percussion

supine sign (not able to lie flat without pain)

21
Q

common hx for those with osteoporosis

A

> 52

no LE pain

BMI <22

no regular exercise

female

22
Q

referral for osteoporosis

A

if a fx is suspected, possibly urgent

emergency if neuro symptoms or inability to walk

23
Q

PT rx for osteoporosis

A

even if there is a fx, pt is usually stable and able to tolerate R xdue to ligamentous structure

proceed based on symptoms

minimize ertebral compression

position/directional preferences with edu, treatments, and activite=ies

bracing/assistive devices i.e. cane/reacher

24
Q

MET focus for osteoporosis

A

bone integrity (maintenance or improving density)

balance

walking/resistance

25
Q

MT for osteoporosis pt

A

caution with JM (especially higher grades may be contraindicated) if advanced level of disease or more than 3 months of corticosteroid use

can use JM to normalize motion and stresses through spine

26
Q

prognosis for osteoporosis fx

A

majority will heal after 8-12 weeks of conservative treatment with subsequent decline in pain

27
Q

MT for osteoporosis pt

A

caution with JM (especially higher grades may be contraindicated) if advanced level of disease or more than 3 months of corticosteroid use

can use JM to normalize motion and stresses through spine

28
Q

MD Rx for osteoporosis

A

percutaneous vertebroplasty

good treatment for some pts with acute/subacute pain

addition of exercises vs none provided better subjective outcomes startign at 6 months and lasting out to 2 years following sx

29
Q

what is osteomalacia

A

bone softening without loss of bone mass or brittleness as with osteoporosis

30
Q

etiology factors for osteomalacia

A

insufficient intestinal calcium absorption due to lack of calcium or more likely low vitamin D

increased kidney phosphate loss (i.e. due to kidney conditions, long term antacid use, or hyperparathyroidism disorders that alter clacium levels)

31
Q

risk factors for osteomalacia

A

lack of dietary or sunlit vit D

malabsorption conditions including age that affect digestive and metabolic fxns

meds that alter vit D, calcium, or phosphate

32
Q

pathogenesis of osteomalacia

A

lack of calcium salts

structure unchanged as mentioned but still weakens the bone with possible fx

primarily affects vertebra and femurs

33
Q

clinical manifestations and S&S of osteomalacia

A

difficult and delayed S&S due to diffuse achiness and fatigue with wt loss

LBP and pelvic and LE pain aggrevated when WB

myalgia/arthalgia

proximal muscle weakness and polyneuropathy

altered gait

increased falls

progresses to deformities like increased kyphosis and genu varum (bow legged)