Lecture 7 Flashcards

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1
Q

How do our senses respond biologically to relevant stimuli?

A

They are specialized and have evolved to give us useful information

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2
Q

What are sound waves

A

Periodic compressions, causing vibrations

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3
Q

What is sound frequency

A

The number of compressions per time. Relates to perceived PITCH

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4
Q

What is sound amplitude

A

Intensity of the sound wave, which relates to perceived loudness

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5
Q

What is sound

A

A complex set of acoustic information is transferred

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6
Q

How are sound waves detected

A

Pinna (outer ear) then travels to the Tympanic membrane (eardrum). The system converts sound waves into waves of greater pressure onto oval window of cochlea to move the viscos fluid within towards the hair cells which are displaced by vibrations, then goes to the auditory nerve (nerve bundle)

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7
Q

What is the pinna

A

The outer ear

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8
Q

What is a tympanic membrane

A

Eardrum - a thin membrane where information is carried via vibrations and then via the instruments of the middle ear

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9
Q

What is the cochlea

A

3 fluid tunnels (snail shaped) and allows us to distinguish between frequencies. Has hair cells in it that are displaced when there are vibrations

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10
Q

What are the auditory nerves

A

Nerve bundles

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11
Q

Where is the sound info processed

A

After reaching the ear, it goes to the cochlear nucleus then over to the primary auditory cortex. There is a CROSSOVER between left ear and right hemisphere

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12
Q

What makes us able to distinguish between different frequencies

A

Basilar membrane of cochlea and primary auditory cortex. High frequency sounds excite near the base, low frequency near the apex. 4% of people are tone deaf = amusia. Neighbouring cells respond to neighbouring frequencies

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13
Q

If the primary auditory cortex is damaged will it cause deafness

A

Sound processing would be affected but not cause deafness

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14
Q

If the middle ear is damaged, could it cause deafness?

A

It can cause conductive deafness if the bones in the middle of the ear are damaged. Or nerve deafness when cochlea, hair cells or auditory nerve is damaged

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15
Q

How can we locate sounds

A

Both ears receive different information. There’s three differences: time of arrival, intensity, phase in sound waves

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16
Q

Can we hear all loud sounds in our close srrounding

A

No. We have highly specialized hearing systems.

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17
Q

What does a vestibular organ do

A

Detects position and movement of the head - crucial for balance

18
Q

How many canals does the vestibular organ have

A

3

19
Q

Describe the vestibular organ canals

A

Next to inner ear, filled with jelly-like substance, lined with hair cells

20
Q

How does vestibular sensation work

A

Head moves, then within the 3 semicircular canals the jelly-like substance moves. Calcium carbonate particles move against hair cells towards brainstem then cerebellum.

21
Q

Why are there 3 canals

A

They represent three dimensions and different movement directions

22
Q

Where are taste buds found

A

Grouped in papillae on the tongue

23
Q

What do fungiform papillae affect

A

Sensitivity to all tastes - many = high sensitivity

24
Q

How many receptor cells are there in one taste bud

A

50

25
Q

How do taste buds behave

A

Like neurons - release neurotransmitters to excite nearby neurons. But they are not neurons - they are modified skin cells and replaced after 10-14 days.

26
Q

What are the four primary tastes

A

Sweet, salty, sour and bitter

27
Q

Can the 4 primary tastes describe all tastes

A

No. - glutamate taste receptors, the monosodium glutamate in fast food

28
Q

How does the brain encode taste

A
  1. Taste nerves. 2. Medulla (at nucleus of the tractus solitaris). Then either Insula (for taste) or somato-sensory cortex (for touch)
29
Q

Why are there other sensory organs

A

By tasting, humans obtain information on the chemical environment with physical contact and with a focus on food, but there is additional important information in the chemical environment.

30
Q

How do we detect smell

A

Olfactory receptor cells - in nasal cavity, hundreds of types for many different chemicals. Olfactory bulb - processes the information (in the limbic system)

31
Q

Discuss the vomeronasal sensation

A

Auxiliary olfactory - in nonhuman mammals. It detects odourless chemicals that affect behaviour sing receptors at the olfactory bulb

32
Q

Name two neurodegenerative diseases

A

Huntington’s disease and Parkinson’s disease

33
Q

Discuss Huntington’s disease

A

Motor disorders - arm jerks, facial twitches. Psychological disorders - depression, anxiety, hallucinations. Gradual brain damage, firstly in the basal ganglia then cerebellum, cerebral cortex and more. Strong genetic influence

34
Q

Discuss Parkinson’s diseas

A

Motor disorders - spontaneous, rigidity and slowness. Cognitive disorders - imagining movement and events. Gradual death of neurons in substantia nigra (mid-brain), decrease in dopamine activity, reduced stimulation of the motor cortex. Genetic & exposure to toxins

35
Q

How do we move

A

Skeletal muscles - control movement of body in relation to the environment. Smooth muscles control internal organs. Cardiac muscles controls heart muscles and fuse to make a pump. Movement results from muscle fiber contractions

36
Q

Name the 3 muscle types

A

Skeletal muscles. Smooth muscles. Cardiac muscles.

37
Q

What is neuromuscular junction

A

Synapse of muscle fiber & motor neuron axon. Each muscle fiber is innervated by one motor neuron axon

38
Q

How do skeletal muscles move

A

Acetylcholine released by axons = muscle contraction. Acetylocholine absence = muscle relaxation

39
Q

What is required in order for movement to other directions

A

Antagonistic muscles

40
Q

How does the brain control complex movements

A

Primary motor cortex - axons - brainstem&spinal cord - impulses that control muscles

41
Q

Discuss the primary motor cortex

A

It has a map of areas of the body movements - stimulation at each spot causes specific movement, active even when we think about movement

42
Q

What other brain areas are involved in movement

A

Cerebral cortex (movement preparation) - prefrontal cortex, supplementary motor cortex, posterior parietal cortex. Basal ganglia (initating actions). Cerebellum (timing/attention/balance)