Lecture 3- Vision Sensation Flashcards
optic chiasm
where half the ganglion cells in the optic nerve cross over to the other side of the brain
(light from the RIGHT visual field enters the pupil from such an angle that it actually hits the LEFT side of the retina & vice versa
thalamus
relay centre(s) of the brain. Most sense-related info passes through it. Cross-modal processing begins
lateral geniculate nucleus (LGN)
Receives info from both eyes in a “retinotopic” fashion- cells in the LGN have a map-like correspondence to cells in the retina
- filter for irrelevant info
Topographic mapping
right LGN processes info from the left visual field and vice versa
hemianopia
blindness to parts of the visual field (wedge-shaped, hourglass)- caused by LGN damage
superior colliculus
has connections between info coming from each eye. allows for richer and more complex processing of info
primary visual cortex/ striate cortex
striated/ striped, damage = blind
cortical magnification
distortion of the visual field
extrastriate cortex
region of the cortex bordering the primary visual cortex and containing multiple areas involved in visual processing
heteronomous
different sides
homonymous
same sides
Area V2
receives input from V1 & outputs to V3, V4, V5
- most neurons in this area tuned to simple visual characteristics: orientation, spatial frequency, size, colour, shape
Area V3
implicated in the process of global motion
“what” pathway
names & functions of objects, regardless of location
- areas V1, V2, V3 provide info to V4, which passes to V5 and other areas
Cerebral achromatopsia
cannot perceive colour, caused by damage to “what” pathway