Lecture 3 Flashcards

1
Q

Previously believed non-causes of stuttering (4)

A
  1. Punishment from God
  2. Oral fixation
  3. Structural deficits
  4. Psychoneruosis
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2
Q

What makes the phenomenon of stuttering difficult to explain?

A

Variability, Onset v. development, persistence v. recovery

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3
Q

Aspects of a good theory (4)

A
  1. Validity (able to predict something)
  2. Elegant and parsimonious (simple)
  3. Testable and flexible
  4. Has explanatory power
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4
Q

Overarching types of stuttering theories (2)

A
  1. Moment–refers to overt stuttering

2. Etiology–refers to the why; can cover covert stuttering

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5
Q

Sub-types of etiological stuttering theories (3)

A
  1. Breakdown (tries to find the why in process)
  2. Repressed need (tries to find why in unconscious psych)
  3. Anticipatory struggle (tries to find why in conscious psych)
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6
Q

Cerebral Dominance Theory –Orton and Travis

A

Most people have cerebral dominance, but PWS have bilateral dominance and signals are sent at the same time.

This is not generally believed now, but modern studies do show unusual hemispheric activity in PWS.

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7
Q

Failure of Automaticity –West; Mysak

A

PWS attempt to exert conscious control over the automatic process of speech

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8
Q

Diagnosogeic Theory –Johnson

A

Stuttering is created by negative reactions to normal disfluency

The Tudor study tried to create PWS through negative feedback to normal disfluency, but failed.

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9
Q

Anticipatory Avoidance –Johnson

A

Stuttering is interruptions caused by the speaker’s attempts not to stutter.

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10
Q

Continuity Hypothesis –Bloodstein

A

Stuttering deviates from normal fluency breaks because of communicative pressure

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11
Q

Approach-Avoidance Conflict –Sheehan

A

The desire to avoid speaking outweighs the desire to speak (silent blocks)

The desire to speak outweighs the desire to avoid speaking (stuttering)

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12
Q

Early-onset Psycholinguistic –Bernstein-Ratner

A

Stuttering occurs because of difficulty encoding syntactic units

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13
Q

Phonological Psycholinguistic –Wingate

A

Stuttering occurs because of a difficulty transitioning between the onset and the rime

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14
Q

Neuropsycholinguistic model –Perkins, Kent, Curlee

A

Asynchronous arrival of segmental and suprasegmental information causes stuttering–typical v. atypical is determined by the speaker’s perceptionof time pressure

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15
Q

Covert Repair Hypothesis

A

Internal, pre-articulatory hypermonitoring and repairing of utterances

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16
Q

Vicious Cycle Hypothesis

A

Hypermonitoring of otherwise normal articulatory plans

17
Q

Ex-Plan Model

A

Dyssynchrony between the planning and execution. Linguistic plans are sent out incomplete.

18
Q

Computational Models

A

Based on automaticity models

19
Q

Adaptive Motor Theory Model

A

Stuttering is caused by an intermittent flaw in the integration of perception and production

20
Q

DIVA Model –Max et al

A

Stuttering is caused by weakness in the feed-forward system and an over reliance on feedback

21
Q

Multifactorial Model –Zimmerman

A

Reduced tolerance/instability of oral movements is caused by various stressors

22
Q

Demands and Capacities Model

A

The environmental demands for fluency are higher than the child’s capacity

23
Q

Strengths and flaws of multifactorial models

A

Multifactorial models can help clinicians explain the nature of stuttering to patients, but are too broad for use in research