learning and memory Flashcards

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1
Q

learning

A

how experiences change the brain

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2
Q

memory

A

how changes are stored and subsequently reactivated

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3
Q

amnesia

A

retrograde- unable to remember the past

anterograde- unable to form new memories

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4
Q

what is the simplest form of learning

A

habitation

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5
Q

scientific contributions of HMs case

A

medal temporal lobes are involved in memory. STM and LTM are distinctly separate. he was unable to move memories for STM to LTM. memory may exist but not be recalled

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6
Q

explicit memories

A

conscious memories

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7
Q

implicit memories

A

unconscious memories, as when HM shows the benefits of prior experiences. sub cortical

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8
Q

repetition priming tests

A

used to assess implicit memory

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9
Q

medial temporal lobe amnesia

A

not all with this form of amnesia are unable form new explicit long term memories

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10
Q

semantic memory

A

general information may function normally while episodic memory does not- they are able to learn facts, but do not remember doing so

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11
Q

Korsakoff’s syndrome seen is

A

chronic alcoholics

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12
Q

korsakoffs symptoms

A

amnesia, confusion, personality changes, and physical problems. they don’t think they have a memory problem

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13
Q

korsakoffs damage

A

typically in the ,medial diencephalon
medial thalamus +medial hypothalamus
causes are multifactorial

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14
Q

Alzheimers disease (AD)

A

begins with slight loss of memory and processes to dementia.

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15
Q

what causes the amnesia seen in AD

A

decreased acetylcholine, due to basal forebrain degeneration. medial temporal lobe and prefrontal cortex also involved. damage is diffuse

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16
Q

post traumatic amnesia

A

concussions may cause retrograde amnesia for the period before the blow and some anterograde amnesia after. Mild TBI is less than 24 hours. period of anterograde amnesia suggest a temporary failure of memory consolidation

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17
Q

reconsolidation

A

when a memory is retrieved from LTM it sis temporarily held in STM. memory in STM is susceptible to post traumatic amnesia until it is reconsolidated. the memory trace becomes temporarily vulnerable during recall

18
Q

robot gradient

A

first one in last one out

19
Q

hippocampus

A

perhaps the hippocampus stores memories temporarily

20
Q

removal of rhinal cortex

A

bilateral removal of the rhinal cortex consistently results in object-recognition deficits

21
Q

hippocampus removal effects of object recognition

A

bilateral removal of the amygdala has no effect on object-recognition

22
Q

hippocampus role

A

plays a key role in memory for spatial location. many hippocamal cells are place cells. a GPS with a 24 hour cycle

23
Q

cognitive map theory

A

constructs and stores allocentric maps of the world

24
Q

configure association theory

A

involved in retaining the behavioural significance of combinations of stimuli

25
Q

where are memories stored

A

each memory is stored diffusely throughout the brain structures that were involved in its formation. damage to a variety of structured results in memory deficits

26
Q

hippocampus storage

A

spatial location

27
Q

rhinal cortex storage

A

object recognition

28
Q

basal forebrain

A

Alzheimers disease

29
Q

inferotemporal cortex

A

visual perception of objects

30
Q

lesions in amygdala lead to

A

lack of learned fear

31
Q

prefrontal cortex

A

temporal order of events and working memory, strategy use

32
Q

prefrontal cortex

A

damage leads to problems with tasks involving a series of responses

33
Q

cerebellum and striatum

A

sensorimotor tasks

34
Q

cerebellum

A

stores memories of sensorimotor skills: conditioned eye blink, possibly working memory

35
Q

striatum

A

habit formation, associatiions between stimuli and responses.

36
Q

Long term potentiation (LTP)

A

synapses are effectively made stronger by repeated stimulation. LTP only occurs if presynaptic firing is followed by postsynaptic firing

37
Q

Hebbs postulate for learning

A

co-occurrence is necessary for learning and memory

38
Q

where is LTP greatest

A

in brain areas involved in learning and memory

39
Q

LTP can be viewed as a three part process

A

induction or encoding/acquistion
maintenance or consolidation
expression or retrieval

40
Q

what is the neurological marker of memory?

A

protein, it means new material is created