Last Week Eating Disorders Flashcards

0
Q

ANOREXIA NERVOSA overview of. Criteria

A

DSM 5 criteria:
 Restriction of energy intake relative to requirements, leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health.
 Intense fear of gaining weight or of becoming fat, or persistent behaviour that interferes with weight gain, even though at a significantly low weight.
 Disturbance in the way in which one’s body weight or shape is experienced, undue influence of body weight or shape on self-evaluation, or persistent lack of recognition of the seriousness of the current low body weight.
Subtypes:
 Restricting type
 Binge-eating/purging type

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1
Q

DSM-V: FEEDING AND EATING DISORDERS

A

Pica
Rumination Disorder
Avoidant/Restrictive Food Intake Disorder
Anorexia Nervosa
 Restricting type
 Binge-eating/purging type
Bulimia Nervosa Binge-Eating Disorder
Other Specified Feeding or Eating Disorder Unspecified Feeding or Eating Disorder

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2
Q

ANOREXIA NERVOSA: CORE FEATURES

A

PERSISTENT ENERGY INTAKE RESTRICTION
Restriction of energy intake relative to requirements, leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health.




Severity in DSM-V depends on BMI
BMI =
weight in kg height in meters 2
Mild: BMI > 17
Moderate: BMI = 16–16.99 Severe: BMI = 15-15.99 Extreme: BMI < 15
BMI is not the only consideration
18.5 BMI “cutoff” used by Centers for Disease Control and Prevention and WHO
 But can depend on clinical history; treatment aim usually BMI = 20
For adolescents, compare to expected developmental trends (e.g., failure to make weight gain)

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3
Q

ANOREXIA NERVOSA: CORE FEATURES
INTENSE FEAR OF GAINING WEIGHT

A

Intense fear of gaining weight or of becoming fat, or persistent behaviour that interferes with weight gain, even though at a significantly low weight.
 Phobia of gaining weight
 Fear of gaining weight may not be alleviated by weight loss (may actually increase)
 Fear of gaining weight at times denied: focus on behaviour preventing weight gain

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4
Q

DISTURBANCE IN SELF-PERCEIVED WEIGHT OR SHAPE

A

Disturbance in the way in which one’s body weight or shape is experienced, undue influence of body weight or shape on self-evaluation, or persistent lack of recognition of the seriousness of the current low body weight.
 Distortions in self-view (not always present): overall or specific body parts
 Excessive focus on body weight and shape (e.g., frequent weighing, using tape measure, mirror)
 Body weight and shape tied to self-esteem and self-concept
 Thinness valued over health concerns
 Can also overvalue the “control” of weight itself (seen as an accomplishment or mark of discipline)

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5
Q

ANOREXIA NERVOSA: SUBTYPES

A

RESTRICTING TYPE
Weight loss primarily accomplished through dieting, fasting, and/or excessive exercise.
 Forbidden foods
 Small portion sizes or specific ways of eating (e.g., small plates, small utensils)

PURGING TYPE
Binge eating or purging behaviour (self-induced vomiting, misuse of diuretics, laxatives or enemas)
 Compared to bulimia, small portions and more consistent purging
Characterized by underweight, small portions. Aside on insulin purge

About 50% of patients correspond to either type
Unclear whether truly distinct
 Severity?
 Continuum? (common to change subtype)
 Personality traits

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6
Q

ANOREXIA NERVOSA: CLINICAL CORRELATES

A

Rigid, inflexible thinking
 Difficulty thinking about alternatives; don’t like change; cling to preconceptions  Impediment to treatment

Depressive symptoms
 E.g., depressed mood, social withdrawal, feelings of ineffectiveness
Self critical intensify symptoms

Perfectionism
 Especially self-critical perfectionism
 Intensifies certain aspects of the ED (e.g., extreme standards are applied to eating, weight, and shape)

Obsessive-compulsive features
 Both related and unrelated to food (e.g., hoard food, collect recipes, obsessive weighing, counting calories)  Concerns about eating in public
 Strong need to control one’s environment

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7
Q

BULIMIA NERVOSA
DSM-V criteria

A

 Recurrent episodes of binge eating:
 A) Eating, in a discrete period of time (e.g., within any 2-hour period), an amount of food that is definitely larger than what most individuals would eat in a similar period of time under similar circumstances.
 B) A sense of lack of control over eating during the episode (e.g., a feeling that one cannot stop eating or control what or how much one is eating).
 Recurrent inappropriate compensatory behaviours in order to prevent weight gain, such as self-induced vomiting; misuse of laxatives, diuretics, or other medications; fasting; or excessive exercise.
 The binge eating and inappropriate compensatory behaviours both occur, on average, at least once a week for 3 months.
 Self-evaluation is unduly influenced by body shape and weight.

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8
Q

BULIMIA NERVOSA: CORE FEATURES
BINGE EATING

A

 Objectively large amounts of food: must ask in assessment
 Roughly 1500 calories in a binge; discomfort at fullness
 Shame, often kept secret

COMPENSATORY BEHAVIOURS
 Severity based on frequency of compensatory behaviours per week
 Mild: 1-3, Moderate: 4-7, Severe: 8-13, Extreme: 14+  In

In clinical samples, vomiting is most common
behaviour (2/3 of patients)

 In community samples, exercise and strict
diet are most common compensatory behaviours (only 1/5 vomiting)

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9
Q

BULIMIA NERVOSA: CORE FEATURES
SELF-EVALUATION

A

 Dominated by shape and weight
 Unlike AN, weight typically in normal range
Episodes often triggered by low mood, interpersonal stressors, intense hunger following restriction, or feelings re: body shape or weight
 Binges/purges can provide temporary relief from tension, dysphoria, or fullness
 Often followed by low mood, shame, self-criticism
Subtypes removed in V non purging removed

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10
Q

BN: CLINICAL CORRELATES

A

Impulsivity
 E.g., in relation to purging but also other behaviours  Emotional instability and erratic dietary pattern

Substance abuse
 Subgroup that engages in substance abuse (common overlap)

Self-injury and BPD
 Subgroup meets criteria for Borderline Personality Disorder

Childhood abuse?
 Mixed evidence from research

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11
Q

CHANGES FROM DSM-IV eating disorders ednos

A

Prior to DSM-V, many eating disorder patients were diagnosed as EDNOS (Eating Disorder Not Otherwise Specified):

 No amenorrhea
 AN symptoms but healthy weight
 Frequency of binge/purge
 Other syndromes (e.g., chew and spit)
To address this problem, changes were applied to the DSM-V criteria:
 Anorexia: removed requirement for amenorrhea
 Bulimia: reduced frequency from 2x/week to 1x/week on average
 Wilson & Sysko, 2009: literature review demonstrated that the clinical characteristics for both groups were similar
 Creation of binge eating disorder diagnosis
 Clinical correlates and course are different from those of other EDs
 Reliably diagnosed

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12
Q

BINGE EATING DISORDER

A

 Recurrent episodes of binge eating:
 A) Eating, in a discrete period of time (e.g., within any 2-hour period), an amount of food that is definitely larger than
what most individuals would eat in a similar period of time under similar circumstances.
 B) A sense of lack of control over eating during the episode (e.g., a feeling that one cannot stop eating or control what or how much one is eating).
 The binge-eating episodes are associated with 3 or more of the following:
 A) Eating much more rapidly than normal.
 B) Eating until feeling uncomfortably full.
 C) Eating large amounts of food when not feeling physically hungry.
 D) Eating alone because of feeling embarrassed by how much one is eating.
 E) Feeling disgusted with oneself, depressed, or very guilty afterward.
 Marked distress regarding binge eating is present.
 The binge eating occurs, on average, at least once a week for 3 months.
Severity based on frequency of binge eating episodes / week.
 The binge eating is not associated with the recurrent use of inappropriate compensatory behaviour.
May be overweight or not

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13
Q

BINGE EATING DISORDER correlates

A

Individuals with BED differ from in meaningful ways from individuals with AN and BN
 More likely to be obese
 Age of onset is later
 Course of BED is different e.g., dysfunctional dieting follows onset of BED whereas dysfunctional dieting often precedes onset of BN
Individuals with BED differ from individuals who are obese but do not have BED  Greater psychiatric co-morbidity
 Poorer functioning
CBT and IPT are associated with significantly greater improvement in functioning relative to weight loss programs alone in treatment of BED

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14
Q

PREVALENCE
ANOREXIA:

A

 Prevalence: approx. 1% (APA, 2000)
 Gender differences: more females than males
 Males sometimes underdiagnosed
 Thinness vs. athleticism
 Influence of sexual orientation? (Yelland & Tiggeman, 2003; Martins et al., 2007

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15
Q

PREVALENCE
Bulimia

A

BULIMIA:
 Prevalence: approx. 2%
 Gender differences: more females than males

16
Q

BINGE EATING DISORDER: prevalence

A

 Prevalence: approx. 1-3.5% (1.6% of adolescents)
 Gender differences: Gender ratio less skewed, but more females than males

17
Q

Heritability of eating disorders

A

Biological factors
 Based on family studies with ED probands, controls and first-degree relatives
 AN heritability: 58-76%  BN heritability: 30-83%
Part of etiology

18
Q

Etiology of eating disorders

A

Family environment
 Parent’s attitudes towards eating and weight
 Frequent dieting or encouragement of child to diet

Social influences
 Peer influence, social contagion
 Preadolescence obesity linked to later EDs (teasing)

Cultural influences
 Media, societal pressures

19
Q

CULTURAL INFLUENCE
ANOREXIA: unlike bulimia

A

Evidence for AN across cultures and history
Modest increase in AN prevalence coinciding with increasing idealization of thinness (meta-analysis by Keel & Klump, 2003)
Most prevalent in industrialized cultures, Western societies

BULIMIA:
Greater influenced by culture

20
Q

MEDIA INFLUENCES:
THE ADVENT OF TELEVISION IN FIJI
Typically difficult to study effects of media
Becker et al., 2002: The Advent of Television in Fiji

A

1990s: one reported case of anorexia
63 girls interviewed 1 MONTH AFTER TV
65 girls interviewed 3 YEARS AFTER TV
1995
Two groups did not differ on age, weight, or ethnicity.

8 out of then and then 19 laters
Suggests media had effect on eating patterns. I

MEDIA INFLUENCES:
THE ADVENT OF TELEVISION IN FIJI

63 Girls interviewed < 1 month after t.v. introduced
65 Girls interviewed 3 years after t.v. introduced
Significantly disordered eating
8
Vomiting to control weight
0
Narrative data suggested that girls admired the women they saw on TV and wanted to emulate them (hairstyles, clothing, body size)
77% reported that TV had influenced their body image

21
Q

COURSE OF EATING DISORDERS

A

Usually appear during passage to adolescence or young adulthood

Diathesis-stress model
 Dieting is often trigger

Fluctuating course, with high rates of relapse
 AN: less than 50% recover (approx. 1/3 unremitting)
 Harder to treat the longer it persists
 BN: Recovery 50-75% (higher than AN)
 Body dissatisfaction may remain high
 BED: More likely to improve than AN or BN
 Little cross-over between diagnoses

22
Q

CONSEQUENCES OF EATING DISORDERS

A

ANOREXIA
Physical symptoms
 Dry mouth, dry skin, losing hair, sensitivity to cold, loss of bone density, gastro-intestinal symptoms, heart problems, etc.
High death rate
 Mortality rate of 5-10% (highest of all clinical diagnoses)
 Death commonly from cardiovascular problems resulting from electrolyte imbalances (not starvation) electrolyte imbalance K/Ca
 Potassium and calcium critical to normal heartbeat; disrupted by purging and underfeeding
 Bradycardia (slowing of heart rate) is common cause of death

BULIMIA
Physical problems
 Puffy cheeks/enlarged salivary glands, loss of dental enamel, tears in esophagus/stomach
 Electrolyte imbalance and cardiovascular consequences
 Fatigue, headaches

23
Q

TREATMENT OF EATING DISORDERS

A

Common perceptions of treating EDs
 One of hardest disorders to treat
 Often chronic (many rounds of treatment)
 Worries about patient’s physical health
 High rate of mortality

Apprehension from therapists
Recommendation: multidisciplinary treatment team
Psychs no good at taking electrolytes need doc or nurse

24
Q

WHY ARE EATING DISORDERS SO HARD TO TREAT?

A

Eating disorders are ego-syntonic
 In line with desires/goals of the ego; consistent with one’s ideal self-image
 Denial that there is a problem
 Patients “like” some aspects of their disorder,
 Secondary gains: thinness, attention, “badge of honor”, regulation of affect, control…b(music sense of control)

Affects first part of treatment
 Patients are often very ambivalent about treatment
 External sources of motivation
 Enhancing motivation to change is key to any treatment
 Motivational interviewing: non-coercive, help patient decide what is in their best interest :)

25
Q

EFFECTIVE TREATMENTS FOR EDS

A

Effectiveness
 CBT found to be effective in treating bulimia
 Limited evidence for effective treatments for anorexia
 Ethical problem of random assignment to conditions
 Rare incidence of disorder: difficult recruitment
 Robin et al., 1999: Recruited 37 participants for a study between 1988 and 1994

Common approaches:
 Individual treatment (typically cognitive-behavioural therapy)
 Family therapy FOR YOUTH

26
Q

CBT-E FOR BULIMIA

A

CBT-E (Chris Fairburn): 2 phases of treatment
PHASE 1
Phase 1 is primarily behavioural
 Explain structure of treatment
 Set goals for treatment
 Formulation of the problem
 Psychoeducation
Events and associated mood change

Over-evaluation of shape and weight and their control
Strict dieting; non-compensatory weight-control behavior
Binge eating
Compensatory vomiting/laxative misuse

Regular eating (3 meals/day + 3 snacks, every 4 hours)
 Portions, exposure to forbidden foods
 Replace bingeing with pleasurable activities
Weekly weigh in and food journal, nor stand triggers

27
Q

CBT FOR BULIMIA
Phase 2 deals with cognitions

A

 Identify maladaptive cognitions
 “If I gain weight, my swim times will be slower,” or “If I eat white rice, I will become fat.”

 Cognitive restructuring
 Thought records & behavioural experiments
 Examining the evidence: create hypotheses and test them

28
Q

CBT FOR BULIMIA
 Identifying and addressing maintaining mechanisms  Excessive weighing
 Body checking
 Highly restrictive rules for eating
 Target FEAR OF WEIGHT GAIN

A

 Emotional regulation
 “Fat” is not an emotion – what is really going on?
 Event-contingent symptoms  Problem-solving

29
Q

CBT FOR BULIMIA
Outcome evidence

A

 50% of BN patients cease bingeing and purging following 16-20 weeks of CBT

Best prognosis:
 Large initial reductions in bingeing and purging

Worst prognosis:
 Borderline personality disorder
 Substance abuse

30
Q

FAMILY THERAPY FOR ANOREXIA

A

AN as symptom of family problem AN as developmental setback
 Goals: restore patient’s weight before complications arise or development is disrupted Patient cannot control symptoms
Parents must step in to save starving child

Weight first for children, then cognitions, due to dev. Issues.

31
Q

FAMILY THERAPY FOR ANOREXIA

A

Phase 1: Parental refeeding
PHASE 1
PHASE 2
PHASE 3
 Teenager is identified as unable to care for herself
 Parents are coached to work as a team to develop and maintain a plan to feed child  Efforts to prevent purging (e.g. remove bathroom door)
 Eat all meals as a family
 High calorie diet (to restore patient to adequate weight)
 Treatment continues until patient begins to gain weight regularly

Phase 2: Cognitive distortions and family structure
 Target patient’s distorted body image and beliefs about food
 Problematic family patterns
 Role of eating symptoms in family life
 E.g. Does it maintain secondary gains? Does it express conflicts within the family?

Phase 3: Adolescent issues
 Return responsibility for eating to the adolescent  Foster adolescent autonomy
 Teach problem-solving skills

32
Q

FAMILY THERAPY FOR ANOREXIA
Outcome evidence

A

 Data is promising for adolescent AN: 39-68% moderate or good outcome
Appropriate candidates for family therapy  Younger than 18 years old
 Live with their parents
 Inappropriate for adult AN
Poor prognosis:
 Parental criticism
 Peers and parents may be contributing to symptoms: take this in account during treatment

33
Q

COMMON CHALLENGES IN TREATMENT

A

Motivation
 “Lack” of motivation?
 Need to align therapist and patient goals as
much as possible
 “What is in your best interest?”**
Resistance to weighing and food journals
Medical complications
 Fainting, blood in vomit, low heart rate, low potassium
 Regular blood tests
 Liaison with medical departments and psychiatrists
‘Plateau’ of gains
Change in diagnosis, but continued symptoms
 E.g., AN → BN, but still binging/purging
Chronicity
 Relapse prevention
 End of treatment often unplanned