LA 2 Flashcards

1
Q

What is the purpose of a “drug holiday” with clients who are taking narcotics on a long-term basis?

A

With narcotics, especially stronger agents, that are used long-term (e.g., ms contin, transdermyl fentanyl), a 3-day drug holiday (or as ordered by the physician) every month has shown evidence of increasing the sensitivity of the client to specific narcotics, as now the receptors have also become more sensitive.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

true or false and explain your answer:

Cancer clients should never receive strong narcotics at the beginning of any pain experience because of the fear of addiction

A

False. With cancer pain, a client’s quality of life is far more important than the fear of addiction. Clients who are living with cancer need to experience comfort and maintain quality of life as much as possible. Addiction should not even be a concern for these clients because quality of life is so important for those afflicted with the illness.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

You administer 100 mg meperidine (Demerol) intramuscularly to a client in severe post-operative pain, as ordered. What assessment data should be gathered before and after administering this drug? Explain your answer.

A

The nurse should always assess the client’s blood pressure, pulse, and respirations before administering any narcotic or, in this situation, meperidine. This allows comparison of baseline vital signs to those taken after the drug has been administered, as well as the assessment of the effect of the drug on the physiological response to pain.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Your client complains that the drugs he is receiving for severe pain are really not helping. What should be the most appropriate response to this client?

A
  • First, it is important to assess the client and his or her previous reactions to pain medications and then assess the dose, action, route, and appropriateness of the agent.
  • The client’s medical history, nursing assessment, and medication history should also be studied carefully because the client’s diagnosis may not match up with the type of analgesic ordered.
  • Once it has been determined that the client is getting the right medication and dosage, then education about pain management
  • The nurse should administer the medication at regular intervals, as ordered
  • The nurse should also always include non-pharmacological methods of pain management
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of self-preparation:
Oral:

A

Very easy if alert; easy to teach others.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of self-preparation:
IM:

A

Not easy to carry out on own; would need assistance from healthcare personnel or other trained person

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of self-preparation:
Transdermal

A

Easy to learn to use and easy to teach to others.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of administration:
Oral:

A

Very easy if alert; easy to teach others.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of administration:
IM:

A

Easy to administer medication by this route if qualified healthcare provider is there to assist

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of administration:
Transdermal

A

Easy to administer and learn how to apply. Very easy to instruct on application and administration of patch.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Onset of therapeutic effects:
Oral:

A

Varies depending on specific drug properties and chemical composition of the oral drug; some very predictable and some very unpredictable. Generally speaking, 45 minutes to 1 hour for onset of effects. Remember that some drugs with longer half-lives may take weeks to have therapeutic effects

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting

Onset of therapeutic effects:
IM

A

Generally dependable; onset of therapeutic effects within at least 15 to 30 minutes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Onset of therapeutic effects:
Transdermal

A

Dependable onset but is variable in some of the pharmacokinetics until serum steady states are achieved because of the nature of topical absorption into systemic circulation. However, once used for a few days, transdermal patches are very effective in their onset of therapeutic effects.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Serum concentrations:
Oral

A

Serum concentrations are dependent on the drug, its properties, chemical composition and dosage form (i.e., if enteric-coated. If the oral drug is taken at regular intervals, it may achieve adequate serum concentrations; however, many physiological factors provide barriers to predictable concentrations of the drug in the serum.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Serum concentrations:
IM

A

Very effective serum concentrations.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Serum concentrations:
Transdermal

A

Very effective serum concentrations that remain fairly predictable with routine usage and application.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Degree of sedation:
Oral:

A

Degree of sedation is dependent on dosage of drug and the potency of the drug. Medications such as morphine and other opioid derivatives are very sedating, but again, level of sedation depends on dose and other variables.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Degree of sedation:
IM

A

: Once again, the degree of sedation depends on drug and dosage, but because systemic absorption is more rapid, there are more CNS-depressing effects, and thus more CNS sedation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Degree of sedation:
Transdermal

A

CNS sedation depends on drug patch and dosage. Often the client builds up some resistance to the sedation, but this is generally seen with lower dosage forms of the patch.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Side-effects:
Oral:

A

Narcotics usually have many side effects, and even more with oral dosage forms owing to gastric upset related to gastric irritation.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Side-effects:
IM

A

Side effects depend on drug and dosage but with IM route, the effects are generally more profound because of increased absorption. Therefore, the side effects related to CNS depression, such as sedation and decreased vital signs, are more often associated with these dosage forms.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Side-effects:
•Transdermal

A

Side effects are very likely to occur with transdermal patches once the steady state of the drug is achieved. Because of steady absorption with consistent dosing, the side effects will depend on the dosage and drug.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of home management:
Oral

A

Very easy.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting.

Ease of home management:
IM

A

Not easy without medical or nursing assistance.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Compare and contrast the effectiveness of oral, intramuscular, and transdermal routes for narcotic administration, considering ease of self-preparation and administration, onset of therapeutic serum concentrations, degree of sedation, side effects, and ease of management in the home setting. Ease of home management: Transdermal
Very easy.
26
When is spinal anesthesia the method of choice?
There are many conditions besides labour and delivery for which spinal anaesthesia would be indicated. Conditions for which one might have a “spinal” rather than general anaesthesia include any that would be exacerbated, causing possible harm to the client—such as a client with severe respiratory or cardiac disease who could not tolerate the effects of general anaesthesia. It is important to remember that spinal anaesthesia is often used with specific types of surgeries or in clients in poor health or with conditions/diseases that would be exacerbated by general anaesthesia.
27
What is the purpose of adding epinephrine to a local anesthetic?
Epinephrine is added to topical and local anesthetics to enhance blood vessel constriction at the area and decrease the risk of possible systemic absorption.
28
What are potential complications of local anesthetics used in dental offices, such as lidocaine with epinephrine, in a client with a variety of cardiac or vessel diseases?
there is concern regarding the absorption of epinephrine, which could lead to blood vessel constriction and rapid elevation in blood pressure and pulse. For the “at-risk” client who might have vessel disease and weakened vessels, there is concern regarding the subsequent rupture of a vessel and cerebrovascular accident or death. Rapid acceleration of pulse rate could also be detrimental to at-risk clients because of the potential for ischemia from excessive oxygen demand oversupply
29
pain that originates from organs or smooth muscles
visceral pain
30
pain that originates from skeletal muscles, ligaments, or joints
somatic pain
31
pain that is psychological in nature but is truly pain in terms of actual pain impulses that travel through nerve cells
psychogenic pain
32
pain experienced in a body part that has been surgically or traumatically removed
phantom pain
33
pain that results from a disturbance of function or pathological change in a nerve
neuropathic pain
34
Enflurane is a(n) ______ anaesthetic.
inhalation general
35
Halothane is a(n) ____________ anaesthetic.
inhalation general
36
Nitrous oxide is primarily indicated for use in __________?
dental procedures (such as removal of “wisdom teeth”) or as a useful supplement to other, more potent anaesthetics.
37
Three major complications of general anaesthesia include _____________________
respiratory and/or cardiac depression, cardiac and/or respiratory arrest, irregular heart rhythms, malignant hyperthermia, aspiration pneumonia, and central nervous system depression
38
pain that results from any disorder that causes central nervous system damage
central pain
39
These contraindications are for??? i) known drug allergy ii) severe asthma or other respiratory insufficiency, especially in the absence of resuscitative equipment, iii) conditions involving elevated intracranial pressure, and iv) pregnancy, especially in long-term or high doses
Narcotics
40
An antidote to an overdose of morphine is ____________________.
naloxone hydrochloride
41
Opioid analgesics bind to an opioid pain receptor in the brain and cause an analgesic response—the reduction of the pain sensation.
mechanism of action of the opiates
42
What are the neurotransmitters that are produced by the body to fight pain. They bind to opioid receptors and inhibit the transmission of pain by closing the "GATE" to the pain impulse
Endogenous enkephalins and endorphins
43
Any of a group of highly addictive analgesic drugs derived from opium or opium like compounds. Narcotics can cause drowsiness and significant alterations of mood and behavior.
Narcotic
44
Medications that relieve pain without causing | loss of consciousness (sometimes referred to as painkillers).
Analgesic
45
Any of various sedative narcotics containing opium or one or more of its natural or synthetic derivatives. or A drug, hormone, or other chemical substance having sedative or narcotic effects similar to those containing opium or its derivatives: a natural brain opiate. Also called opioid
Opiate
46
Is it a narcotic and non-narcotic analgesics? -Acts centrally-
Narcotic Analgesics
47
Is it a narcotic and non-narcotic analgesics? Does not produce CNS depression
Non-Narcotic Analgesics
48
Is it a narcotic and non-narcotic analgesics? Cause addiction
Narcotic Analgesics
49
Is it a narcotic and non-narcotic analgesics? Produce CNS depression
Narcotic Analgesics
50
Is it a narcotic and non-narcotic analgesics? Do not produce gastric irritation
Narcotic Analgesics
51
Is it a narcotic and non-narcotic analgesics? Has anti-inflammatory effect
Non-Narcotic Analgesics
52
Is it a narcotic and non-narcotic analgesics? Act peripherally
Non-Narcotic Analgesics
53
A synthetic analgesic drug that is used as a substitute to treat morphine and heroin addiction
Methadone
54
pain that continues or recurs over a prolonged period, caused by various diseases or abnormal conditions. Chronic pain may be less intense than acute pain. The person with chronic pain does not usually display increased pulse and rapid respiration because these autonomic reactions to pain cannot be sustained for long periods. Some factors that can complicate the treatment of persons with chronic pain are scarring, continuing psychologic stress, and medication
Chronic Pain
55
Acute pain is sudden and usually subsides when treated—for example, postoperative pain or the sudden onset of a headache.
Acute Pain
56
pain anywhere in the body of unknown cause
Idiopathic pain
57
What medications relieve pain without causing loss of consciousness?
“Painkillers” Opioids Nonsteroidal anti-inflammatory drugs (NSAIDs)
58
Pain felt in a different are of the body
Referred Pian
59
Persistent or recurring Lasting 6 weeks or longer Often difficult to treat
Persistent pain/Chronic Pain
60
It uses the analogy of a gate to describe how impulses from damaged tissues are sensed in the brain Many current pain management strategies are aimed at altering this system
Gate Theory
61
What are the four processes to the gate theory?
Transduction Transmission Perception Modulation
62
What are the two types of nerves stimulated?
A fibres | C fibres
63
real pain of psychological orgin eg. Fibromyalgia, chest pain from anxiety
Psychogenic Pain
64
Pain felt from touching a hot stove
superficial pain
65
The amount of pain a patient can endure without its interfering with normal function
Pain tolerance
66
The level of stimulus needed to PRODUCE THE PERCEPTION OF PAIN
Pain Threshold
67
What are the two endogenous neurotransmitters the body produces in pain modulation?
Endorphins & Enkephalins
68
In pain modulation, rubbing or applying liniment to a painful area stimulates the?
large sensory fibres
69
Pain relievers that contain opium, derived from the opium poppy or chemically related to opium:
Opioid Analgesics
70
What are the three Chemical Classification of Opioids
Meperidine-like drugs Methadone-like drugs Morphine-Like drugs
71
What are the actions based on the three classifications?
Agonist- cause analgesic response Partial agonist Antagonist
72
Often given with adjuvant analgesic drugs to assist the primary drugs with pain relief NSAIDS AND CORTISONES reduce
INFLAMMATION
73
Often given with adjuvant analgesic drugs to assist the primary drugs with pain relief Antidepressants and Anticonvulsants
Are used for NERVE PAIN
74
What kind of drug can be used for Cough centre suppression,Treatment of diarrhea (causes constipation), & Procedural pain during surgery as an adjunct to anaesthesia
Opioids
75
What are some contraindications to opioid use?
Known drug allergy Severe asthma or other respiratory insufficiency Elevated intracranial pressure Pregnancy
76
Hypotension, palpitations, flushing Sedation, disorientation, euphoria, lightheadedness, dysphoria, lowered seizure threshold, tremors Nausea, vomiting, constipation, biliary tract spasm Urinary retention Itching, rash, wheal formation **Respiratory depression and aggravation of asthma**
ADVERSE
77
What is the most common Opiate Antagonists?
Naloxone (Narcan) Used for complete or partial reversal of opioid-induced respiratory depression
78
What is a common physiological result of chronic opioid treatment?
Opioid tolerance
79
Definition The physiological adaptation of the body to the presence of an opioid
Physical Dependence
80
Definition A pattern of compulsive drug use characterized by a continued craving for an opioid and the need to use the opioid for effects other than pain relief
Psychological Dependence
81
What is expected with long-term opioid treatment and should not be confused with psychological dependence (addiction)?
Opioid tolerance and physical dependence Misunderstanding of these terms leads to ineffective pain management and contributes to the problem of undertreatment
82
What occurs when opioid are abruptly discontinued or when an opioid antagonist is administered
``` Opioid withdrawal (opioid abstinence syndrome) because the body has a physical dependence on them ```
83
Anxiety, irritability, chills and hot flashes, joint pain, lacrimation (teary), rhinorrhea(runny nose), diaphoresis, nausea, vomiting, abdominal cramps, diarrhea are symptoms of?
Withdrawal
84
What drug Has analgesic and antipyretic effects Has little to no anti-inflammatory effects Is available over-the-counter (OTC) Is a component of many combination products with opioids?
Acetaminophen
85
Overdose, whether intentional or due to chronic unintentional misuse, can cause?
hepatic necrosis (liver)
86
Long-term ingestion of large doses of acetaminophen can also cause?
nephropathy (kidneys damage)
87
What is the recommended antidote to acetaminophen?
Acetylcysteine regimen
88
What is the Maximum daily dose of acetaminophen for healthy adults?
4000 mg per day
89
What are some interactions to Acetaminophen?
Alcohol use Liver dysfunction Possible liver failure Other hepatotoxic drugs
90
Opioid Analgesics: Nursing Implications What is the most significant nursing implication to opioid use?
Withhold dose and contact physician if there is a decline in the patient’s condition or if vital signs are abnormal, ESPECIALLY IF THE RESPIRATORY RATE IS LESS THAN 12 BREATHS/MIN
91
What is the most common adverse effect and may be prevented with adequate fluid and fibre intake
CONSTIPATION
92
Should vital signs change, patient’s condition decline, or pain continue a nurse should
contact physician immediately
93
Manifested by respiratory rate of less than 12 breaths/min, dyspnea, diminished breath sounds, or shallow breathing
Respiratory Depression
94
Definition- | *Drugs that depress the central nervous system (CNS)*
Anaesthetics
95
What are the two types of anaesthesia?
Two types: General anaesthesia Local anaesthesia
96
What are the effects of anaesthesia?
Depression of consciousness Loss of responsiveness to sensory stimulation (including pain) Muscle relaxation
97
``` Definition: Drugs that induce a state in which the CNS is altered to produce varying degrees of Depression of consciousness Skeletal muscle relaxation Visceral smooth muscle relaxation ```
General Anaesthetics
98
How is general anaestheia administered?
Inhaled anaesthetics-Volatile liquids or gases that are vaporized in oxygen and inhaled Injectable anaesthetics-Administered intravenously
99
Inhaled Anaesthetics | What is an example of Inhaled gas
nitrous oxide (“laughing gas”)
100
Inhaled Anaesthetics | What is an example of Inhaled volatile liquids
halothane (Halothane) | isoflurane (Forane)
101
When would an Injectable Anaesthetics be used?
To induce or maintain general anaesthesia To induce amnesia To reduce anxiety As an adjunct to inhalation-type anaesthetics
102
Which theory explains the more fat soluble the better it works?
Overton-Meyer theory
103
During surgical procedures general anaesthetics is used to produce
Unconsciousness Skeletal muscular relaxation Visceral smooth muscle relaxation
104
What are some General Anaesthetics: Contraindications?
Known drug allergy Pregnancy (depending on drug type) Narrow-angle glaucoma (depending on drug type) Known susceptibility to malignant hyperthermia- genetic condition from prior experience with anaesthetics (depending on drug type)
105
General Anaesthetics: What are some possible Adverse Effects?
Primarily affect: Heart, peripheral circulation, liver, kidneys, respiratory tract Commonly include myocardial depression Sudden elevation in body temperature (greater than 40°C) Tachypnea, tachycardia, muscular rigidity Life-threatening emergency Very dangerous when given anasthesia
106
General Anaesthetics: Interactions General anaesthetics are associated with a wide array of drug interactions varying in severity More common drug–drug interactions occur with:
ANTIHYPERTENSIVES β-blockers Tetracycline **Blood pressure drugs**
107
What would a Local Anaesthetics be used for?
Used to render a specific portion of the body insensitive to pain *Do not cause loss of consciousness*
108
What are ways to administer Local Anaesthetics?
Topical-(Applied directly to skin or mucous membranes) Parenteral- outside the GI tract-(Injected intravenously or into the CNS)
109
What are the two types of Local Anaesthetics?
* Central - Spinal or intraspinal - Intrathecal - Epidural * Peripheral - Infiltration - Nerve block - Topical
110
What is a common form of a Parenteral Anaesthetic Drug?
lidocaine (Xylocaine)-freezing, used in combination with ephinephrine to keep it local
111
In what order would Local Anaesthetic Drugs Effect Paralysis?
First, autonomic activity is lost, then sensory, and motor last As local drugs wear off, recovery occurs in reverse order (motor, sensory, and then autonomic activity)
112
What are Local anaesthetics are used for:
Surgical, dental, and diagnostic procedures | Treatment of certain types of chronic pain
113
What are some Local Anaesthetics: Contraindications
Contraindications include known drug allergy | Ophthalmic use requires specially designed dosage forms
114
What is a Local Anaesthetics: Adverse Effects
May include “spinal headache,” which is treated by an “epidural blood patch”
115
What does NMBAs stand for?
Neuromuscular Blocking Drugs
116
A neuromuscular blocking drug is used to?
Prevent nerve transmission in certain muscles, resulting in paralysis of the muscle
117
True or False | NMBAs cause sedation or relief of pain
FALSE
118
TRUE OR FALSE | When NMBAS are used during surgery, artificial mechanical ventilation is required
TRUE
119
TRUE or FALSE | Patient may be paralyzed yet conscious when NMBAs are used
TRUE
120
Drugs ending in AM such as diazepam, | lorazepam are what types of drugs
anti anxiety
121
Drugs ending in IUM such as | atracurium
nondepolarizing
122
Nondepolarizing NMBAs prevent ______ from acting at the neuromuscular junctions
ACh
123
A Short-acting NMBAs is mainly used for ?
endotracheal intubation
124
What are some contraindications to NMBAs?
Known drug allergy Previous history of malignant hyperthermia Penetrating eye injuries Narrow-angle glaucoma
125
Overdose of a NMBA can cause?
prolonged paralysis
126
What are the main things to know about NMBAs?
- Lots of drugs interact with neuromuscular drugs - Causes Paralysis - Can feel pain - Doesn’t treat pain
127
Moderate Sedation is also known as?
conscious sedation
128
During moderate/conscious sedation a combination of ______________and an _______ is often used.
intravenous (IV) Benzodiazepine and Opiate analgesic
129
Used for diagnostic procedures and minor surgical procedures that do not require deep anaesthesia
Moderate sedation
130
What are some adverse effects to NMBAs
Hypotension (blockade of autonomic ganglia) Tachycardia (blockade of muscarinic receptors) Hypotension (release of histamine)
131
Assessment is vital during pre-, intra-, and postoperative phases of sedation. What would you assess for?
``` Vital signs Baseline laboratory tests, electrocardiogram Pulse oximetry ABCs (airway, breathing, circulation) All body systems ```
132
Nursing considerations during the perioperative phase include what three phases?
Preoperative phase Intraoperative phase Postoperative phase
133
What do we monitor during recovery?
During recovery, monitor for cardiovascular depression, respiratory depression, and complications of anaesthesia
134
During conscious sedation it preserves the patient’s ability to maintain____________________.
own airway and to respond to verbal commands
135
-First sensation felt is muscle weakness -This is followed by total flaccid paralysis Small, rapidly moving muscles (fingers, eyes) are affected first, and then limbs, neck, trunk -Finally, intercostal muscles and the diaphragm are affected, resulting in cessation of respirations -Recovery of muscular activity usually occurs in reverse order -Transient muscle fasciculations may result in later muscle soreness
NMBAs