Kell, Duffy, Kidd And Lutheran Blood Groups

Question 1

Rated as second to D antigen in immunogenicity

Answer 1

Kell antigen (K)

Question 2

Kell antigen is destroyed orinactivated by

Answer 2

Sulfuhydryl reagents like
AEY
DTT
ZZAP

Question 3

Most common antibody seen in blood bank

Answer 3

Anti-K

Question 4

Anti-K chromosome location

Answer 4

7

Question 5

Lacks Kell antigen and have no membrane abnormality

Answer 5

KO or K null phenotype

Question 6

Lacks Kx antigen (might precursor for Kell Antigen)

Answer 6

McLeod Phenotype

Question 7

Rare phenotype that has abormal RBC morphology

Answer 7

McLeod Phenotype

Question 8

Common among males from Chronic Granulomatous Disease

Answer 8

McLeod Phenotype

Question 9

Duffy antigens

Answer 9

Fya and Fyb

Question 10

Most important antigen associated with transfusion medecine in duffy blood group

Answer 10

Fya and Fyb

Question 11

Well developed at birth and can eaily destroyes by common proteolytic enzyme

Answer 11

Fya and Fyb

Question 12

Important anthropological marker for African blacks

Answer 12

Fy (a-b-)

Question 13

Fy (a-b-) were shown to resist infection caused by

Answer 13

Plasmodium vivax and Plasmodium knowlesi

Question 14

Common antibodies in duffy

Answer 14

Anti-Fya

Question 15

Kidd antigens

Answer 15

Jka and Jkb

Question 16

Resist lysis in 2M urea and occurs in Mongoloids

Answer 16

Jk(a-b-)

Question 17

Reactivity enhanced by enzyme treatment

Answer 17

Jka and Jkb

Question 18

Common cause of delayed HTR

Answer 18

Anti-Jka and Anti-Jkb

Question 19

Lutheran genes are located on

Answer 19

Chromosome 19

Question 20

lutheran genes closely linked with

Answer 20

Secretor locus

Question 21

Lutheran antige

Answer 21

Lua and Lub

Question 22

Rare phenotype with decreased Kell system antigen expression and abnormal red cell morphology, which has been associated with X-linked chronic granulomatous disease (CG), which is a rare disorder affecting only the males

Answer 22

McLeod Phenotype

Question 23

develop a slow, progressive form of muscular dystrophy between ages 40 and 50, which i: associated with areflexia (a lack of deep tendon flexes), choreiform movements (well-coordinated but involuntan
movements), and cardiomegaly leading to cardiomyopathy, they have elevated serum creatine phosphokinase (CPK) levels of the MM type and carbonic anhydrase Ill levels

Answer 23

McLeod Phenotype

Question 24

Parasities that are associated with Fya and Fyb

Answer 24

Plasmodium vivax
Plasmodium knowlesi