Intro to Acquired Language Disorders Flashcards

1
Q

What is Aphasia?

A

-Acquired
-Neurological Cause
-Affects reception and production of language, across modalities
-NOT a sensory, motor, psychiatric or intellectual disorder

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2
Q

What is often the problem of aphasia?

A

-Problem is access to stored linguistic representations, not the loss of representations themselves

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3
Q

brain pathology can cause other patterns of impairment and perseveration

A

-TBI
-Right-hemisphere dysfunction
-dementias, includeing PPA

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4
Q

Unidimensional vs Mult-dimensional Frameworks

A

-Uni: A whole, so a deficit anywhere will affect the language system
-If a patient needs to work on speaking, also work on other things to stimulate the language system
-Multi: understanding the different classifications
-Understand what kinds of things might help a particular person

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5
Q

Medical Frameworks

A

-Impairment label/deficit approach
-Don’t have to focus on medical approach if in a medical setting

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6
Q

Cognitive neuropsychological, psycholinguistic, and neurolinguistic models

A

-Think about language as a process
-See the object, visual processing, semantic concept
-If a pt can’t name an object there are multiple places where they system could be interrupted

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7
Q

Biopsychosocial model

A

-Environmental factors, social support, motivators

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8
Q

Social Models

A

-Understanding aphasia and disability as a social condition
-Subset of biopscychosocial
-Aphasia will affect everything in a person’s life

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9
Q

Social Detriments of Health

A

-Inequities and inequalities in health care; and these affect people with aphasia as well; no two people will be the same due to the social determinants of health

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10
Q

WHO Framework

A

-Health condition (stroke)
-Body functions & structures (impairments) [language]
-Activities (limitations) [how much is communication affected; talking to family members coworkers]
-Participation (restrictions) [occupation, sports, tiktok]
-Environmental factors
o Personal factors
 Own personal motivation
 Depression
 Identity

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11
Q

Language vs. Cognition

A

o Use a multimodality approach
-Assess all modalities
-Which are more or less impacted
o Disorder of language, not intellect
-E.g., someone with aphasia may recognize an individual but not come up with name

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12
Q

Diagnosing Aphasia

A

o Includes
 Naming (name the item or point to it from choice of 3)
 Follow direction (touch nose, ceieling, move toes_
 Answer questions (do you peel a banana before you eat it)
 Orientation questions (your name? date? Where you are?)

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13
Q

Multimodality deficit: example

A

-Most common deficit with aphasia
-Word-finding difficulty
-“I know the word I just cant say it”

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14
Q

Diagnosing aphasia: goal

A

-Word level: Listen to word, point to object; read a word “; name objects (speaking); write names of objects

-Sentence level: follow simple commands; follow simple instruction (reading); describe simple actions (speaking); describe actions (writing)

-Discourse/text level: Listen to a story/answer questions; read a paragraph and answer questions; describe a complex picture (speaking); writes a letter (writing)

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15
Q

Challenges of Diagnosis

A
  1. Is it aphasia and motor speech disorder or just aphasia
  2. only neurologist can diagnose pathology; SLP diagnoses speech and language disorder by bx
  3. damage of aphasia is in ONE location
    -Think of it as a “Central Disorder”
    -i.e., “one grammar” is underlying “language” whether it is for talking or writing
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16
Q

Deficit of Propositional Language

A

-Statements about the world either true or false
-Use language to intentionally talk to people
-Every time you speak you are getting words putting them together, you are using your knowledge of speaking
-Automatic language is sometimes preserved (subpropositional language)
-Can’t use language to be as creative
-e.g., counting, singing a song, or producing routine greetings such
as , “How are you?” or “I’m fine”

17
Q

Symptoms of Omission

A

whats missing? (certain words; grammatical words)

18
Q

Symptoms of Commission

A

– whats wrong? Have the right words but something is wrong

19
Q

Anomia

A

Problem of finding and retrieving words
-Not to be confused with anomic aphasia
-Omissions and Commisions

20
Q

Circumlocution

A

-Talk around the word (error of commission)
-Describing item can be a useful strategy

21
Q

Paraphasia

A

A word substitution error
(Think about the relationship between the PWA’s production and the target)

22
Q

4 Types of Paraphasia

A
  1. Phonemic: Substitutes as sound in the word (e.g., kiger for tiger)
  2. Semantic: substitutes a related word for the intended word (e.g., lion for tiger)
    3.Unrelated: e.g., flag for tiger
  3. Neologism: nonword error (e.g., floosis for tiger)
23
Q

Non-fluent

A

Individual speaks with effort and with fewer words than normal

24
Q

Fluent

A

Individual talks with easy flow using complete sentences

25
Agrammatism
-limited grammar -omitted units are grammatical morphemes -The agrammatic individual produces many open-class or content words
26
Fluent Aphasia
-Main Issue: producing appropriate content words such as nouns, verbs, and adjectives -May have (significant or mild) word-finding errors -May use vague/general terms or circumlocutions -words come easily, but not necessarily the right words -High use of jargon and paraphasias (word salad/gibberish) -Neologism one word, jargon is many
27
Stereotypic utterances
-Some PWA can only produce involuntary or subpropositional language -Repetitions of the same word, phrase, or syllable -Can be dictionary words, or neologism, or phrase
28
Why do some PWA have agrammatism and others speak fluently (but not always comprehensibly)?
-Multidimensional framework: syndromes correspond to lesions -Cognitive, psycholinguistic neurolinguistic framework: based on models of representation -We need to understand each approach
29
Sylvian Fissure
Separates Parietal and temporal lobes
30
frontal lobe
executive functioning, personality, motor, decision making, planning
31
Problems with Classical Model
o lesion doesn’t predict aphasia type o underestimates the number for distribution of brain regions involved in language o researchers don’t agree where these regions (specifically) are
32
Explaining aphasia: neurology
-Neurological explanation: link lesions to symptoms -Wernicke: comprehension deficit ~ damage in the left temporalcortex
33
Explaining aphasia: cognition
-Knowledge: what we know -Process: activity of the mind/quick mental response -Mental representation: How information is represented in the brain -Layers of knowledge * Mental representation * Cat: how do you conceive of this?
34
Memory: cognitive functions need memory
-LTM: long term memory (address, phone number) how to type, things you’ve learned how to do -WM: working memory, shorter, thinking about it -does brain damage affect working memory in a way that aggravates or even causes symptoms of aphasia? * automatic processing vs. controlled processing
35
Aphasia: Cognition
aphasia is an impairment of processing rather than a loss of linguistic knowledge
36
Aphasia Treatment Settings
-Acute: Immediate and short-term -Subacute: transition between acute hospitalization and home (inpatient rehabilitation unit, SNF) -Chronic: long term, outpatient
37
Ways to treat aphasia
Impairment-based: focus on linguistic/cognitive deficits Communication-based: focus on activity/participation limitations
38
Central Disorder
one anatomical location for the lesion that is causing all of the problems