Exam Study Mix Flashcards

1
Q

Process Approach

A
  1. Clinician writes down all answers verbatim
  2. Observes setting and bx during and between assessment tasks for strengths and weaknesses
  3. Uses qualitative data to assess PWA
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2
Q

Why should we care about Limb Apraxia?

A

Limb use can be an important strategy for helping PWA communicate with gestures and gestural communication.

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3
Q

Two Types of Word Fluency Tasks

A
  1. Divergent Naming Task
  2. Letter-Fluency Task
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4
Q

Divergent Naming Task

A

Generate words from a semantic base (category)

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5
Q

Letter-Fluency Task

A

Generate words from a lexical base (starts with letter…)

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6
Q

Why is a screener better than an informal evaluation?

A

Informal evaluations lack validity and reliability

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7
Q

Advantage of WAB

A
  1. Summary score can be completed in 30-45 minutes
  2. Uses real objects for word level tasks
  3. Bedside version
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8
Q

Problems with BDAE

A

Outdated pictures (kitchen picture & noose), takes long time to administer

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9
Q

Special Aspect of BDAE

A

First test to document syndromes in aphasia; includes short form

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10
Q

3 Features of a Good Aphasia Battery

A
  1. Comprehensive assessment of language functions
  2. Covers all 4 langauge functions/ modalities at all levels of language (reading, writing, listening, speaking)
  3. Minimizes influence of intelligence, education, or other extralinguisitic content
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11
Q

Things to Learn from Case History

A

Patient Routines
When Stroke was
Aspects of life most affected

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12
Q

Goals of Aphasia Assessment

A
  1. Identify strengths/weaknesses to inform treatment (+severity)
  2. Deduce aphasia type
  3. Discover Pt’s goals
  4. Get documentation for payers
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13
Q

How to Acknowledge Competence?

A

1.Acknowledge their frustration and tell them they know they know what they want to communicate

  1. Speak in a natural manner
  2. Let them know their thoughts and opinions are important.
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14
Q

How to Reveal Competence?

A
  1. Use resources for the PWA to communicate (letter board, Yes/No sheet, writing tools, pain scale)
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15
Q

Why is Supported Conversation Important?

A

Because PWA have a problem with language access, not cognition, and they are in charge of their health and planning. People need to know their thoughts on this.

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16
Q

4 Types of Agrpahia

A
  1. Phonological Agraphia
  2. Deep Agraphia
  3. Surface (Lexical) Agraphia
  4. Graphemic Buffer Agraphia
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17
Q

Which type of Agraphia is the sound to letter route an effective strategy?

A

Surface (Lexical) Agraphia

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18
Q

4 Types of Alexia

A
  1. Pure Alexia
  2. Surface Alexia
  3. Phonological Alexia
  4. Deep Alexia
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19
Q

Pure Alexia

A
  1. Alexia w/o Agraphia
  2. Retain ability to spell & write
  3. Impairment between visual and naming system
  4. Will use letter by letter strategy (short words easier)
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20
Q

Surface Alexia

A
  1. Impairments in the lexical semantic reading route
  2. Difficulty with irregular spellings, homophone, homograph, low frequency words
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21
Q

Phonological Alexia

A
  1. Damage to sublexical route
  2. High frequency words > low
  3. High imageability words > low
  4. Content words > grammatical words
  5. ❌ non-words
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22
Q

Deep Alexia

A
  1. Damage to semantics and sublexical route
  2. High frequency > low
  3. High imageability > low
  4. Content words > grammatical
    5 ❌ non-words
  5. ❌ semantic errors
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23
Q

Phonological Agraphia

A
  1. Damage to sublexical route
  2. High frequency > low
  3. High imageability > low
  4. Content words > grammatical
  5. Difficulty spelling non-words
  6. Spelling errors typically not phonologically plausible (flewen for flower)
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24
Q

Deep Agraphia

A
  1. Damage to Semantics and Phonological
  2. High frequency > low
  3. High imageability > low
  4. Content words > grammatical
    ❌ non-words
    ❌ semantic errors
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25
Q

Surface Agraphia

A
  1. Lost or degraded orthographic representations
  2. Difficulty with irregular spellings, homophones, and low frequency words
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26
Q

Graphemic Buffer Agraphia

A
  1. A short-term memory issue
  2. Lexical-semantic and sublexical routes intact
  3. Long words more difficult than short words
  4. Types of Errors: Omissions (sweatr), Substitutions (peanul), Transpositions (painetr), additions (flowaer)
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27
Q

Allographic Conversion Agraphia

A
  1. Well-formed but incorrect letters or disturbed letter formation
  2. Preserved oral spelling
  3. Print vs. Cursive
  4. Person will have preserved copy, but impaired transcoding (e.g., ask to transcode from upper to lower case)
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28
Q

Treatment for Allographic Conversion Agraphia

A

Use self-dictation!

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29
Q

Types of Alexia with Trouble with lexical semantic route?

A

Surface & Pure Alexia & Deep

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30
Q

Types of Alexia with trouble with sublexical route?

A

Phonological and deep alexia

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31
Q

What is the sublexical route?

A

The route that skips the semantic route straight to the phonological buffer. It does not depend on the activation of words in the lexicon.

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32
Q

Prognosis

A

Prediction of the progression of a condition over time and recovery likelihood

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33
Q

First thing to assess besides language deficits/skills?

A

1.Swallowing ability and safety
2.Educating and counselling
3.Teaching communication

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34
Q

Semantic vs lexical access for PWA with lots of circumlocutions

A

Indicates poor lexical access but good semantic access

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35
Q

Would a PWA have more or fewer word finding deficits in picture naming vs narratives?

A

Word finding narratives would have fewer deficits

36
Q

Order of Events for Neuropsychology model of object naming

A
  1. See object
  2. Picture Recognition
  3. Conceptual Semantic System
  4. Phonological Output Lexicon
  5. Articulatory Processes
  6. Spoken Name
37
Q

Confrontational Naming Task: Lexical Impairment vs Semantic Impairment

A
  1. Lexical: Has trouble recalling words from the spelling and sounds, word form
  2. Has trouble recalling words from the category and meaning
38
Q

Visual Agnosia

A

Nonaphasic misnaming

39
Q

Argument Structure

A

Grammar component that entails who is doing what to whom in a sentence

40
Q

Semantic concept

A

Representation of a word without letters, such as features of the object or idea

41
Q

3 things that make a sentence more challenging

A
  1. Complexity (Active easier than passive)
  2. Reversibility (reversible sentences harder)
  3. Canonicity (deviating from easiest to understand structure (agent-action-recepient); embedded clauses)
42
Q

3 ways to assess sentence level comprehension

A
  1. Follow simple commands (listening)
  2. Follow simple instruction (reading)
  3. Point to a picture after reading a sentence.
43
Q

Syntactic Parsing Mechanism

A

Assigning structure to a string of words

44
Q

Evidence for semantic networks

A

Demonstrated by the semantic priming effect, where a lexical decision is faster when preceded by a semantically related prime

45
Q

What is more important than type of aphasia?

A
  1. How it affects life participation
  2. Symptoms of aphasia
  3. Dissociation vs. Syndrome
  4. Describe speech and strengths, levels and severity
46
Q

Parasylvian Lesion

A
  1. Near Sylvian fissure
  2. Affects repetition
47
Q

Extrasylvian lesion

A
  1. Far from Sylvian fissure
  2. Connected to better repetition
48
Q

Crossed Aphasia

A

Aphasia from right-hemisphere stroke

49
Q

Subcortical aphasia

A
  1. Aphasia from damage to subthalamic structures
  2. Thalamic = Word finding deficits
  3. Non-thalamic (incl. basal ganglia)= global, Broca’s, and Wernicke’s
50
Q

Broca’s Aphasia

A

Non-fluent and effortful

51
Q

Global Aphasia

A

Non-fluent, poor comprehension,

52
Q

Conduction Aphasia

A

Fluent, good comprehension, worse repetition

53
Q

Transcortical Motor Aphasia

A

Non-fluent, repetitions intact

54
Q

Transcortical Sensory Aphasia

A

fluent, repetitions intact

55
Q

Transcortical Mixed Aphasia

A

non-fluent, poor comprehension; repetition intact

56
Q

Wernicke’s Aphasia

A

Fluent, poor comprehension, jargon

57
Q

Anomic aphasia

A

Mild, fluent, comprehension intact

58
Q

Hematoma

A

Accumulation of blood that floods the brain’s surface or brain tissue

59
Q

Arteriovenous Malformation:

A

In which arterial walls weaken and the capillary network between arteries and veins is absent

60
Q

Spontaneous Reperfusion

A

Flow to the uninfarcted side improves spontaneously and dramatically within two or three weeks after onset

61
Q

Diaschisis

A

Suspension of functions that depend on structures remote from the infarct

62
Q

Hypoperfusion

A

Reduced bloodflow to the area surrounding the infarction

63
Q

Penumbra

A

Area surrounding the infarction, a grayish margin where blood flow is reduced, but tissue is intact

64
Q

Aneurysm

A

A dilated blood vessel that stretches and weakens the vessel wall

65
Q

Intracerebral hemorrhage

A

invades deep interior regions of the brain (thalamus, basal ganglia)

66
Q

Subarachnoid Hemorrhage

A

occurs in the meningeal membranes protecting the brain

67
Q

Thrombosis

A

accumulation of atherosclerotic platelets and fatty plaque on the vessel wall, causing narrowing (stenosis)

68
Q

Embolism

A

Platelets and fatty plaque break off a vessel wall and travel until the cerebral artery becomes too small for it to travel

69
Q

Ischemic Stroke

A

Blockage of the vessel

70
Q

Hemorrhage

A

Rupture of the vessel with bleeding

71
Q

Blockage of which artery will most likely lead to aphasia?

A

MCA because it supplies blood flow to the left hemisphere where language centers are located

72
Q

Aphasia Causes

A
  1. CVA
  2. Tumor
  3. TBI
  4. Hydrocephalus
  5. Anoxia
  6. Degenerative Diseases
73
Q

Open-Class Words

A

Content words like Nouns, Verbs, Adj.

74
Q

Closed-Class words

A

Limited in number and mainly used for grammatical meaning

75
Q

Problem with classical model

A

The problem with the classical model is that it does not consider the complex ways in which brain regions work together to produce and comprehend language.

76
Q

Jargon

A

non-words or made-up words

77
Q

Errors of commission

A

Errors in the use of words

78
Q

Errors of omission

A

omitting words from sentences

79
Q

Agrammatism

A

Difficulties with creating grammatically correct sentences and utilizing grammar

80
Q

Circumlocution

A

Talking around the word a person wants to say

81
Q

Anomia

A

Difficulty with word finding

82
Q

Biopsychosocial model

A

A biopsychosocial framework considers the PWA’s medical condition, the affect on their emotions and mental health, as well as the impact on their social participation, which gives a fuller comprehensive look at how aphasia impacts their lives.

83
Q

Propositional Language

A

the intentional, creative use of language.

84
Q

Subpropositional Language

A

The automatic use of language with words and phrases

85
Q

Aphasia as a Central Disorder

A

Damaging that causing aphasia is in ONE location; ie, one grammar is underlying language whether it’s for talking or writing

86
Q

What is Aphasia Not?

A

Aphasia is not a sensory, motor, psychiatric, or intellectual disorder

87
Q

Aphasia Definition

A

Aphasia is a speech disorder that affects the access to language. It is not the loss of linguistic concepts, but the ability to retrieve them.