interventions midterm Flashcards
what is pne utilized for
healthier responses to pain
nociceptive pain
short duration
well localized
neuropathic pain
pain travels
neuroplastic plain
persistant, chronic
spread in non dermatomal pattern
intensity out of proportion
pain neuroscience education
change belief that pain is marker of injury or disease
is there a pain signal or center in the brain?
no
pain is an experience
what is pain?
appraisal made by our protection system based on neural representation of threat/danger
our knowledge of pain is limited
its still in infancy
its complex - opioid epidemic didn’t solve it.
still learning about chronic pain
MOM (mature organism model of pain)
we evolved to tolerate pain
helps us to survive and pass on genes
stress response
pain helps us avoid bad things
What would happen if we didnt feel pain?
may not survive to adulthood
harm themself
dont realize they are hurt
what is the brain’s role in MOM?
continual sampling of the outside environment, own body, and relevant past experiences
experiences coded into memories
through what process does the brain scrutinize during MOM?
somatic: muscles
autonomic: subconscious control of body
neuroendocrine
immune
3 dimensions of pain in MOM
sensory: anatomical descriptions
cognitive: thoughts
affective: emotional reaction
how does MOM guide treatment?
therapeutic interventions (change tissue inputs)
therapeutic interactions (change environmental input - sleep better, stimulate parasympatetic)
therapeutic reframing (change brain’s self-sampling input via therapeutic neuroscience education)
input not mandatory
some neural processes can act in the absence of any inputs
stress and depression can cause physical pain
phantom pain in amputees
role of neural networks
suggests origins of patterns which underlie experiences lie in brain neural networks
the self
body perceived in unity
pain breaks the unity
genetic compenent
may explain why some are more at risk for chronic pain or maladaptive pain
neuroignature/neurotag
synaptic arrangement of multiple centers in the brain
modulated by protection being highest priority
fired together are wired together
thoughts affect ability
failed homeostasis equals
conditions producing chronic pain
cortical smudging
poor localization
blurring of pain map
need to retrain where the body is
biomedical approaches
medical/pharm treatments
anatomic education
emphasis on pain not function
facilitating avoidance
all have poor - modest results
biopsychosocial approach
emphasize PNE
develop therapeutic alliance with pt
focus less on diagnosis and more on changing maladaptive behaviors
central sensitization
become efficient at feeling pain
increase perception of pain in other regions
changes in properties in neurons
CNS is now on higher alert