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PTRS 882 - Exam 1 and 2 > Inflammation and Healing > Flashcards

Inflammation and Healing Flashcards

1
Q

Non-specific Physical Barrier (1st Line)

A

Skin
Mucous membrane
Hair
Blood clots

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2
Q

Non-specific Fluids (1st Line)

A

Saliva
Tears
Gastric juices
Sweat

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3
Q

Phagocytosis (2nd Line)

A

Neutrophils and macrophages

Includes inflammation

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4
Q

3rd line of defense

A

Specific
Antibody production
Cell-mediated immunity

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5
Q

Inflammation

A

Localize, neutralize, and remove harmful agents and dead tissue

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6
Q

Inflammation Causes

A

Physical damage
Foreign bodies
Infection
Ischemia

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7
Q

Cardinal Signs of Inflammation

A

Redness (Dilation of small blood vessels)
Warmth (Increased blood flow)
Swelling (Increased capillary wall permeability)
Pain (Release of chemicals stimulating pain receptors)

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8
Q

Acute Inflammation

A

Release of chemicals from mast cells and platelets

Chemotaxis of cells to site of injury

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9
Q

Chronic Inflammation

A

Continued tissue destruction
Fibrous scar tissue
Granuloma could form

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10
Q

Agranulocytes

A

WBC

Mono/lymphocytes

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11
Q

Cytokines

A

Mediator
Affect behavior of cells
Signaling molecules

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12
Q

Interferons

A

Cytokine

Non-specific agent produced to prevent virus replication

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13
Q

Histamine

A

Mediator
Vasodilation
Increased capillary permeability

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14
Q

Bradykinin

A 
Mediator
Vasodilation
Increased capillary permeability
Pain
Chemotaxis
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15
Q

Leukotrienes

A

Mediator

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16
Q

Granulocytes

A

WBC

Neutro/baso/eosinophils

17
Q

Mast Cells

A

WBC

Release chemicals in response to injury

18
Q

Macrophages

A

WBC

Phagocytic monocytes

19
Q

Prostaglandins

A

Mediator
Vasodilation
Increased cell permeability
Pain

20
Q

Glucocorticoid Effect

A

Decrease capillary permeability

Reduces number of leukocytes and mast cells

21
Q

Glucocorticoid Adverse Effects

A

Delayed healing
Atrophy of lymphoid tissue
Reduced WBC production
Increase tissue breakdown

PTRS 882 - Exam 1 and 2 (6 decks)

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