Images Flashcards by Vanessa Racal

Hollenhorst plaque
- Cholesterol
- associated with atherosclerosis and risk for stroke
- usually seen at arterial bifurcations

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Toxocariasis
* parasitic nematode Toxocara canis

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RPE defect
** window defect, appears as yellow-white. Well demarcated circular lesion present in mid-peripheral retina
** benign

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Posterior embryotoxon
*anteriorly displaced Schwalbe line
* no treatment at this time
* RTC: annual for CEE

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S/S red, itchy eyes with swollen eyelids, chemosis
Idiopathic

Angioedema
* mast cell degranulation, leading to increased vascular permeability which causes fluid and proteins to leak into subcutaneous spaces

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Myelinated nerve fiber layer
* white feathery margins typically extending from the optic disc

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Myelinated nerve fiber layer
* white feathery margins typically extending from the optic disc

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Bergmeister
*remnants of embryological posterior hyaloid artery that failed to atrophy
(Hyaloid artery supplies blood to developing eye during fetal development)

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Patient wears silicone hydrogel CLs

Mucin balls
* occur with silicone hydrogel lenses that are too flat
* mechanical rubbing of CL stimulates goblet cells to produce extra mucin which form balls that embed into corneal epithelium

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*ignore black lesion

Terrien marginal degeneration
* bilateral superior corneal thinning
* blur from high ATR astigmatism
* men in their 30’s
* longstanding may see pseudopterygium
* tx: correction, if unsuccessful tectonic or penetrating ketatoplasty

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Corneal farinata
* small white specs in the interpalpebral zone of posterior stroma
* bilateral
* no tx required

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Congenital zonular cataract

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What are the layers of the choroid and bruch’s?

Make sure you know the layers!

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Fibrous disciform scar in exudative AMD
* formed by formation of choroidal neovascular membrane

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Transillumination defects seen in patient with tyrosinase-negative albinism

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Subconjunctival hemorrhage
* painless, sectoral hemorrhage of conjunctival vessel into subconjunctival space
* clear demarcation line at the limbus of cornea

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Butterfly macular dystrophy
* pattern dystrophy
* yellow/gray deposits within macular region in a triadiate pattern
* treatment not necessary and prognosis good
* autosomal dominant, 2nd to 5th decades of life

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Conjunctival melanocytoma
* slow growing black lesion of conjunctiva
* rare
* mass not freely movable over sclera and tends to be homogenous in color and lack cystic spaces

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Advanced glaucoma visual field loss
* very small island of central vision left
* patient is considered legally blind due to there being less than 20 degrees of vision in horizontal meridian of the better seeing eye

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Lens vacuoles
* view with retro illumination
* pooling of water in lens due to aggregation of crystalline proteins which are less water-soluble now
* due to aging, cataract formation, diabetes

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Left homonymous hemianopsia
* lesion in right visual pathway distal to optic chiasm
* respects vertical midline

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What is this VF defect? And where is the lesion located?

Left homonymous hemianopsia
* lesion in right visual pathway distal to optic chiasm
* respects vertical midline

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Pt has keratoconus

Fleischer ring * epithelial iron deposit at the surrounding base of the cone in keratoconus * best seen under slit lamp with blue light

Non-ischemic central retinal vein occlusion * caused by formation of thrombus near or at the lamina cribrosa leading to compression of central retinal vein by CRA which impedes blood flow return * blood accumulates causing endothelial cell damage leading to formation of a thrombus leading to retinal hemorrhages

Preretinal hemorrhage (boat shaped) * blood trapped between vitreous cortex and ILM * associated with PDR * caused by blood loss from neovascularization of disc or elsewhere resulting in entrapment of blood between posterior aspect of hyaloid and retinal surface

Epithelial ingrowth * proliferation of epithelial cells at the flap interface after LASIK * flap may need to be relifted and cells removed if interfering with vision

Pseudoexfoliation syndrome * white flaky material deposits along pupillary margin * unilateral * transillumination defects limited to iris sphincter * material can accumulate in TM causing increase in IOP —> glaucoma

Full thickness macular hole

Supernumerary puncta * more than 1 punctum

Large mutton fat keratic precipitates * granulomatous anterior uveitis

Toxoplasmosis *Inactive infection * Protozoa: Toxoplasma gondii * contaminated raw meat or cat feces ingestion

Is this an active infection?

No, it is inactive toxoplasmosis * old retinal lesions appear as circular areas of chorioretinal atrophy surrounded by pigmentation * generally stable * no tx is necessary

Visuoscopy; what type of eccentric fixation does this patient posses?

3 prism diopters nasal and 1 prism diopter superior eccentric fixation *each mark is 1 PD, count from center of target and the direction is opposite

EBMD (epithelial basement membrane dystrophy) * aka map-dot-fingerprint dystrophy or cogan microcystic dystrophy * fibrillation protein deposits between bowman’s and epithelial basement membrane

What congenital retinal vascular anomalies is apparent?

Cilioretinal artery * anomalous direct or indirect branch of posterior ciliary arteries * emerge from temporal aspect of optic disc * positive because allows for secondary route of blood flow in cases of CRAO

Corneal topography I

Keratoconus *asymmetrical figure “8” * high K values >48 = steep

Herpes simplex virus keratitis * dendrites terminal end bulbs that stain with rose bengal * main portion of dendrites stain with sodium fluorescein

S/S; acute, painless, unilateral vision loss

CRVO * caused when central retinal artery compresses central retinal vein causing blood flow to be impeded and forms a thrombus which leads to an occlusion

Pt was welding without proper eye protection

Image 1? Image 2? What condition does this patient have?

1. Krukenberg spindle 2. Transillumination defects Patient has PDS (pigment dispersion syndrome) —-> lead to pigmentary glaucoma

Neovascular AMD * left untreated can lead to leakage of blood and protein under macula, scarring from abnormal blood vessels eventually cause irreversible damage to retinal photoreceptors leading to rapid vision loss

Amelanotic choroidal melanoma * eye cancer where melanocytes in choroid proliferate but lose ability to produce pigment * amelanotic means without pigment

Retinal detachment * lesion is inferior nasal, patient will report phototopsias superior temporally

Retinal detachment

Lattice degeneration * ~1% will progress and lead to retinal detachment

14 month old Asian male “Eyes have become cloudy”

Primary congenital glaucoma * eye appears cloudy due to corneal edema secondary to high IOP * most common form of infantile glaucoma

39 WM, saxophonist Unilateral, Acute, painless loss of lower portion of vision

Valsalva retinopathy * one or more well- rounded or dumbbell shaped hemorrhages beneath the posterior hyaloid of vitreous or the internal limiting membrane

56 WF T2DM with A1c: 9.1% with FBG: 171 mg/dL

Severe non-proliferative diabetic retinopathy * CWS *hemes in all 4 quadrants But no neovascularization, venous beading or IRMA present

Optic pit * rare, congenital * unilateral * pit temporally * asymptomatic

Advanced primary open angle glaucoma * ONH damage. Retinal NFL defects * bilateral, asymmetrical

Fuch’s endothelial dystrophy *beaten metal appearance

50 AF Subjective Rx OD: -17.00 -1.25 x 090 OS: -15.00 -1.25 x 090

Degenerative myopia * vertically elongated optic nerves * Chorioretinal atrophy with temporal myopic conus * confirm dx with A-scan *normal Axial length is 22-25mm *abnormal >26 mm

Optic nerve melanocytoma (benign) * variant of melanocytic nevus * deep pigmented lesion on optic disc * congenital, non-hereditary

Plaquenil maculopathy

Choroideremia * X-linked recessive (females are carriers and do not display reduction in VA) * degeneration of RPE and choriocapillaris

Optic nerve drusen * optic nerve elevation and irregular disc margins *diagnostic imaging: B-scan (turn down gain to visualize high acoustic drusen) OCT imaging * no tx

White without pressure * seen in patients with axial elongation * most frequently seen inferior and temporally * mostly seen in AA pts

CHRPE * lacunae: areas of depigmentation within or around borders * flat with well-defined borders

Mutton fat keratic precipitates in pt with sarcoidosis *idiopathic inflammation *middle aged AA females * need ACE and chest x-ray (90% lung involvement, granulomas )

Y suture cataract

Retinitis pigmentosa * Bone spiculues *autosomal recessive

Presumed ocular histoplasmosis * peripapillary atrophy * atrophic histoplasmosis spots Histoplasmosis triad: PPA, punched out lesions, maculopathy)

Retinal hole with surrounding cuff of edema * caused by retraction of retina by vitreous, pulls a small piece of retinal tissue *operculum seen hovering in vitreous looks like a disc shaped floater, smaller than retina defect

Aniridia * autosomal dominant *majority of iris is lacking

Fundus flavimaculatus with geographic atrophy

Choroidal rupture *tear in choroid, brunch’s and RPE * associated with blunt ocular trauma * can cause CNVM

Cortical cataracts

Symblepharon * partial or complete adhesion between palpebral conj and bulbar conj * lower lid

MGD *frothy tear film *aka posterior blepharitis * caused by dysfunction of the sebaceous glands

Traumatic cataract * from blunt or penetrating ocular trauma, intraocular foreign body Or following cataract surgery * rosette cataract

Posterior synechiae *adhesions between posterior surface of iris and anterior capsule of crystalline lens * result of intra ocular inflammation

56 WF Cc: FBS, sandy, gritty feeling Gradual onset Watery discharge Similar episodes occurring over last few years

Superior limbic keratoconjunctivitis

BRAO

CRVO

Iris nevi * proliferation of benign melanocytes * can cause secondary glaucoma

Iris melanoma

31 WF, Cc: CL very uncomfortable, itchy eyes, ropy discharge, blurred vision, can only wear CL for a few hours

GPC Giant papillary conjunctivitis *large papillae on superior palpebral conjunctiva S/s: itching, stringy mucus, excessive movement on CL

55 year old white female

Birdshot choroidopathy * multiple, small ovoid creamy yellow-white spots (looks like a birdshot blast from a shotgun in the mid periphery) *spares the macula

Iridodyalisis * Iris tear from Iris root * caused by trauma/blunt force * large tear causes glare, photophobia and monocular diplopia * a lot of damage to TM can cause IOP to increased * gonio will show angle recession

ERM

VMT with partial thickness macular hole

Conjunctival nevus * unilateral * cystic spaces, can be freely moved over the underlying sclera * adjacent to limbal region or caruncular area

Wet AMD

Polypoidal Choroidal Vasculopathy * associated with HTN * ICGA to view polyps

Pt with keratoconus

Acute corneal hydrops * cornea progressively thins to the extent that Descemet’s membrane ruptures

What chart is this? And what age range do you use this on to check VA?

Landolt C * most accurate VA in young children

What chart is this?

Allen figures * may overestimate or underestimate true VA in peds

What is top picture? What is the bottom picture?

1. Aponeurotic ptosis (high eyelid crease, LPS functional) 2. Congenital ptosis (no eyelid crease, LPS not functional)

What does this test assess?

How well a pt processes and integrates auditory and visual information *important for reading, speech perception and multitasking * helps assess visual processing disorders in kids with reading difficulties Average score is 100, 50th percentile

What does this testing assess?

Pt’s ability to coordinate visual perception with motor control (hand movements) * visual motor integration (handwriting, drawing, copying shapes) In this example, pt has average scores

What does this testing assess?

Pt’s ability to coordinate visual perception with motor control (hand movements) * visual motor integration (handwriting, drawing, copying shapes)

What does theDEM test assess?

Developmental eye movements * assesses saccades (Ocular motor function) * And automaticity (ability to recognize and verbalize numbers) Type I: V&H normal timing, Normal ratio = no Oculomotor dysfunction Type II: V normal timing, H slow, high ratio = Oculomotor dysfunction (issues with tracking) Type III: V slow, H slow, but ratio normal = automaticity dysfunction Type IV: V slow, H slow, ratio HIGH = automaticity AND Oculomotor dysfunction Type V: V normal, H normal, HIGH ratio = Oculomotor dysfunction V/H slow = abnormal Ratio high = abnormal *difference between type III and type IV is if ratio is normal or abnormal Score interpretation: <16 percentile = FAILS 16th - 31st = suspect >31st percentile = normal *compare to people their age *50% is average

What is this test and what does it assess?

King Devick test * assess Oculomotor function on the basis of speech * does not screen for automaticity

What does this test assess?

Visual perceptual skills * assess how well a patient interprets and makes sense of visual information 1. Visual discrimination: differentiate between similar objects, shapes and symbols 2. Visual memory: recall visual details after brief presentation 3. Visual-spatial relationships: understanding position of objects in space (left vs right) 4. Visual form constancy: recognizing objects despite changes in size, position or orientation 5. Visual sequential memory: remembering the order of symbols or patterns 6. Visual figure-ground: distinguishing object from its background 7. Visual closure: identifying objects when only part of it is visible

Corneal intraepithelial neoplasia Appears: - gelatinous or fleshy - elevated - grayish - at the limbus within interpalpebral fissure * unilateral * light skin, middle age or older patients exposed to UV light * occurs more frequently in immunosuppressed pts (HIV) * high incidence in HPV

Images Flashcards

(107 cards)