Cumulative Flashcards

1
Q

Carotid cavernous fistula is caused by?
And what is the triad?

A

Closed head trauma, spontaneous ruptured carotid aneurysm and high pressure blood from carotid posterior to globe

Triad
1) chemosis
2) pulsatile proptosis
3) ocular bruit

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2
Q

Hyperthyroid systemic symptoms

A

Heat intolerance
Weight loss
Palpitations
sweating
Irritability

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3
Q

Thyroid function tests

A

TSH
T4
T3
TSI

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4
Q

Which hormones are increased in hyperthyroidism?

A

T3 & T4
TSI

T3: triiodothyronine
T4: thyroxine (total and free)
TSI: thyroid-stimulating immunoglobulin

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5
Q

True or false
TSH is high in hyperthyroidism

A

FALSE!!!
* TSH is low while T3, T4 and TSI are high

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6
Q

NO SPECS is a mnemonic for the eye findings in which disease?

A

Grave’s disease

NO SPECS
N: no signs or symptoms
O: only signs
S: soft tissue involvement (s/s)
P: proptosis
E: extra ocular muscle involvement
C: corneal involvement (SLK)
S: sight loss (optic nerve compression)

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7
Q

TED mostly affects what age and gender?

A

females 30-40 years old

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8
Q

True or false
Tolosa hunt syndrome leads to painful external ophthalmoplegia

A

TRUE
*inflammation of the orbital apex and anterior cavernous sinus

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9
Q

Dalrymple’s sign

A

Widened palpebral fissure due to upper eyelid retraction in thyroid disease

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10
Q

Globe lags behind upper lid on up gaze in thyroid disease

A

Kocher’s sign

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11
Q

Lid lag on down gaze in thyroid disease

A

Von Graefe’s sign

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12
Q

TED ocular signs (name at least 5)

A

Eyelid retraction
Edema
Lagophthalmos
Lid lag
Reduced blinking
Superficial keratopathy
Conjunctival injection
Exophthalmos
EOM limitations
(+) forced duction
Decreased VA
Decreased color vision
RAPD
VF defects

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13
Q

Tx for optic neuropathy secondary to Grave’s disease

A
  1. Immediate treatment with oral steroids
  2. Orbital decompression for compressive optic neuropathy
    * performed by oculoplastic surgeon
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14
Q

Tolosa-hunt syndrome

A

Rare, idiopathic orbital inflammation of cavernous sinus + superior orbital fissure = acute painful external ophthalmoplegia

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15
Q

Tx for corneal exposure due to eyelid retraction, lagophthalmos and reduced blink rate in TED

A
  • AT’s up to q1h
  • Ointment QHS
  • lid taping or moisture chamber goggles at bedtime
  • punctal occlusion for severe dry eyes
  • permanent lateral tarsorryhaphy or canthorrhaphy for lateral chemosis or widened lateral palpebral fissure
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16
Q

Steven Johnson syndrome is what type of hypersensitivity reaction?

A

Type 4

  • hypersensitivity to drug/infectious agent
  • sulfonamides= pseudoblistering of conjunctiva (BM intact, inflammation below)
  • amoxicillin and Augmentin can cause SJS
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17
Q

Basal cell carcinoma is more common in males or females?

A

Males
* fair skin
* UV exposure

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18
Q

Pt with hx of chronic unilateral chalazia should be evaluated for what?

A

Sebaceous gland carcinoma
* F>M
* malignancy of sebaceous glands, highly infiltrative + inflammatory, high mortality rate

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19
Q

What is the most lethal primary skin cancer of the melanocytes?

A

Malignant melanoma

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20
Q

True or false SCC is more aggressive than BCC

A

TRUE
* 2nd most common eyelid malignancy
* M>F

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21
Q

What conjunctival lesion is the precursor to SCC of the conjunctiva?

A

Conjunctival intraepithelial neoplasia (CIN)
* looks gelatinous and white near limbus
* can lead to SCC
* mostly in elderly white males, heavy smokers, UV radiation, HPV

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22
Q

Phlyctenules are what type of hypersensitivity reaction?

A

Type 4!!
* in teenage girls
* located at limbus
* ask about Hx TB
* hypersensitivity reaction to staph blepharitis, goes away in ~14 days
* associated with rosacea

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23
Q

Yoked and Fresnel prisms are for near or distance?

A

Near

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24
Q

Reverse telescopes are used for?

A

Ambulating
*increase field of view

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25
Q

Patient wants to be able to read 20/40 letters on a bus, what is the most appropriate power of a telescope to meet this goal?

OD: +4.00 -2.00 x090 distance 20/100, near 20/100 @ 40cm

OS: +4.00 -2.00 x 000 VA 20/400 distance, near 20/400 @ 40cm

A

2.5X telescope

Steps to solve:
1) take denominator of can read and divide it by want to read
100/40 =2.5

2) 2.5X telescope will allow pt to see 20/40 without decrease in FOV

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26
Q

What is the inheritance pattern of Coat’s disease?

A

None,
It’s idiopathic, non-hereditary

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27
Q

S/S of Coats disease (name at least 3)

A

Leukocoria
Strabismus
Nystagmus
Decreased VA
Pain
Iris heterochromia (because of NVI)

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28
Q

If Coat’s disease is left untreated, how would the disease progress?

A
  1. Lead to end stage total exudative RD and neovascular glaucoma
  2. Can cause blind and painful eye
  3. Enucleation
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29
Q

Coats disease can lead to what type of retinal detachment?

A

Exudative RD

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30
Q

What is an adverse prognostic factor related to histological features in choroidal melanomas?

A

Large number of epithelioid cells (vs. spindle cells)

Epithelioid cells are associated with a worse prognosis compared to spindle cells.

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31
Q

What pigmentation characteristic is associated with a worse prognosis in choroidal melanomas?

A

Lack of pigmentation

Tumors that lack pigmentation tend to be more aggressive.

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32
Q

Why will an RD located superiorly be more likely to spread than an RD located inferiorly?

A

Because of gravity
* emergency! Refer to retinal specialist ASAP

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33
Q

At what gestational age does the retinal vasculature reach the nasal periphery?

A

8 months gestation

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34
Q

At what age does the retinal vasculature reach the temporal periphery in normal gestation?

A

1 month after birth

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35
Q
A

Choroidal melanoma

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36
Q
A

Choroidal nevus

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37
Q

Myelinated nerve fiber layer can be associated with what ocular conditions?

A

Amblyopia
Strabismus
Nystagmus
Myopia
Optic neuritis
Neovascularization of the retina

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38
Q

Tx for toxocariasis

A

Prednisone
* Tx intraocular inflammation caused by death of toxocara larva and eye reaction immunologically to the antigens released

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39
Q

What lab tests helps confirm toxocariasis?

A

ELISA
* toxocara ELISA testing

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40
Q

Inheritance pattern of Stargardt

A

Autosomal recessive

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41
Q

ERG and EOG in best disease

A

Normal ERG
Abnormal EOG

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42
Q

What does EOG measure?

A

Potential between bruch’s membrane (RPE) and cornea

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43
Q

ERG and EOG in Stargardt

A

Normal ERG and EOG

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45
Q

GPC shows what two types of hypersensitivity reactions?

A

Type I and Type IV

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46
Q

GPC symptoms

A

Contact lens intolerance
Blurred vision
Excessive lens movement with blinking
Pruritis and mucous production
* general discomfort develops 2-3 years after previously tolerating contacts well

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47
Q

GPC diagnosis is characterized by the presence of?

A

Large papillae located on the upper tarsal conjunctiva
*associated with Hx of CL wear

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48
Q

What is the pathophysiology of GPC?

A

Immune reaction to denatured protein deposits
Mechanical irritation for the superior tarsal conjunctiva

Discontinue CL use
Tx with pataday, switch to dailies

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49
Q

Tx and RTC for GPC

A
  1. Discontinue CL wear
  2. Mast cell stabilizer example: oloptadine 1 gtt OU bid for 2 weeks
  3. Switch to dailies or hydrogen peroxide based solution for cleaning CLs

RTC 2-4 weeks

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50
Q

Tx for contact dermatitis

A
  1. Determine and remove offending agent (ex: new make up, eye cream, lotion)
  2. Cool compress
  3. PF AT’s and topical antihistamine/ mast cell stabilizer combo
  4. Topical steroid cream for eyelids (dexamethasone 0.05% bid or tid for 5 days)
    * oral antihistamine like Benadryl can be added
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51
Q

RTC for contact dermatitis

A

1 week

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52
Q

What skin lesion is a precursor to SCC of the eyelids?

A

Actinic keratosis
* occur on inferior eyelid/ sun exposed areas of skins
* older white patients

*squamous cell carcinoma occurs due to excessive proliferation of the stratum spinosum (cell division should not occur at this layer, that’s why SCC is more invasive than BCC)

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53
Q

What are the layers of the skin epidermis from superficial to deep?

A

Superficial
Stratum corneum
Stratum lucidum
Stratum granulosum
Stratum spinosum (abnormal for cell division, SCC occurs in this layer)
Stratum basale (normal for cell division, BCC occurs in this layer)
Deep

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54
Q

What does a positive Jones 1 test mean?

A

Patent punctum and no blockage

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55
Q

If Jones 1 is negative what is the next step?

A

Irrigate with saline

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56
Q

Saline irrigation in Jones 2 demonstrates reflux of fluid through the same punctum, where is the blockage?

A

Upper/lower canaliculus blockage

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57
Q

Saline irrigation in Jones 2 shows retrograde flow through opposite canaliculus, where is the blockage?

A

Nasolacrimal duct

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58
Q

Saline irrigation in Jones 2 patient tastes saline/ recover in nose, what does that mean?

A

Blockage is cleared

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59
Q

HSV dosage for acyclovir

A

400mg po 5x/day for 7 days

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60
Q

HZO dose for acyclovir

A

800mg 5x/day for 7 days

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61
Q

HSV dosage for valacyclovir

A

500mg TID po for 7 days

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62
Q

HZO dose for valacyclovir

A

1000 mg po TID for 7 days

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63
Q

AIDS medication Retrovir may cause what ocular conditions?

A

Amblyopia
Macular edema

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64
Q

Causes of whorl keratopathy

A

CHAI T

Chloroquine
Hydroxychloroquine
Amiodarone
Indomethacin
Tamoxifen

And Fabry’s disease

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65
Q

Which drugs can cause NAION? (4)

A

Amiodarone
Imitrex
Vardenafil
Viagra

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66
Q

Causes of bull’s eye maculopathy

A

Chloroquine
Hydroxychloroquine
Progressive cone dystrophy
Stargardt’s disease
Thioridazine

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67
Q

Name 2 commonly used aminoglycosides

A

Tobramycin
Gentamicin

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68
Q

Dosage for besifloxacin

A

TID to q1h

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69
Q

Name 2 commonly used macrolides

A

Azithromycin
Erythromycin

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70
Q

Cyclosporine drug class
(Restasis)

A

Immunomodulator

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71
Q

Keflex drug class

A

Cephalosporins
* Keflex is brand name for cephalexin

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72
Q

Tx for endogenous endophthalmitis caused by fungal Candida

A
  • Systemic antifungal
  • If moderate to severe inflammation: pars plana vitrectomy and intraocular injection of antifungal and steroid
  • topical steroid and cycloplegic
  • medical consultation
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73
Q

Fungal infections such as candidiasis mostly occur in what type of patients?

A

Immunocompromised
IV drug abusers

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74
Q

RTC for candidiasis

A

Daily

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75
Q

Oral antibiotics used to treat MRSA

A
  • Bactrim (trimethoprim + sulfamethoxazole)
  • Doxycycline
  • Clindamycin
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76
Q

Intravenous antibiotics used to treat severe MRSA infections

A

Vancomycin

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77
Q

Bacteria that invades intact corneal epithelium

A

Corynebacterium diptheriae
Neisseria gonorrhoeae
Haemophilus
Listeria

Canadian National Hockey League invades intact corneal epithelium

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78
Q

Etiology of acanthamoeba keratitis

A

Contact lens wearers using nonsterile water or have poor CL cleaning habits
* patients who wore CL while in fresh water environments like lakes or in a hot tub

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79
Q

Signs for acanthamoeba keratitis

A

Early
1. Mild SPK, pseudo dendrite (will stain with FL)
*misdiagnosed as herpes simplex!

Late stages (3-8 weeks)
2. Ring ulcer and corneal melt

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80
Q

Tx for acanthamoeba keratitis

A
  1. Discontinue CL wear
  2. Polyhexamethylene biguanide q1h, chlorhexidine q1h, propamidine isethionate q1h or dibromopropamidine isethionate ointment
  3. Can also use antifungal
  4. Topical cycloplegic and oral NSAID for pain

RTC 1-4 days, then 1-3 weeks
Treatment for 3-12 months

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81
Q

What does ERG measure?

A

Electrical currents across the eye

*Cornea is positive relative to RPE
*when retina is stimulated, specific retinal cells become excited and firm the electrical wave that creates ERG

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82
Q

What is ocular histoplasmosis?

A

A retinal condition linked to a type of fungus known as Histoplasma capsulatum

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83
Q

What type of organism causes ocular histoplasmosis?

A

Histoplasma capsulatum

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84
Q

How is Histoplasma capsulatum contracted?

A

Through inhalation

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85
Q

Multiple sclerosis s/s

A

S/S:
Numbness
Fatigue
Muscle weakness
Optic neuritis

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86
Q

Syndrome with optic atrophy in one eye and disc edema in the fellow eye

A

Foster-Kennedy syndrome

*vision loss from intracranial mass compressing optic nerve, and papilledema in the other eye due to increased intracranial pressure

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87
Q

Tx for optic neuritis associated with MS

A

IV steroids
Followed by oral steroids along with interferon beta-1a treatment

*stabilize condition and reduced risk of progression of optic neuropathy for 2-3 years

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88
Q

What VF defects can optic neuritis cause?

A

Altitudinal defect
Overall depression central 30 degrees
Central/centrocecal scotoma

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89
Q

Demographic for MS

A

18-45 females
Raised in northern hemisphere

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90
Q

Hutchinson sign

A

Herpes zoster lesion on tip or side of nose
*signal development of ocular involvement

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91
Q

Shafer’s sign

A

Release of pigment into the vitreous chamber from retinal tear
* noted with sudden increase in floaters

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92
Q

Psoriasis Skin scale is removed and pinpoint bleeding occurs

A

Auspitz sign

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93
Q

What diagnostic testing do you order if you suspect ankylosing spondylitis?

A

1) CBC
2) ESR
3) HLA-B27
4) Sacroiliac spine radiograph

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94
Q

In which layer of the cornea do deposits accumulate in whorl keratopathy?

A

Epithelium
*yellow/brown or white
*located inferior to visual axis, spins outward

Associated with drug use:
amiodarone class 3 anti-arrhythmic
Chloroquine
Hydroxychloroquine
Indomethacin
Tamoxifen

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95
Q

Red cap desaturation test procedure

A
  1. Pt wears best near correction, fully lit room
  2. Better seeing eye is tested first, occlude weaker eye
  3. Show patient red cap bottle and tell them this is 100%, then switch to weaker eye
  4. Ask patient to compare level of saturation of red between the two eyes as a percentage from 100%
  • perceived difference between the eyes of 20% or more = APD likely
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96
Q

True or false
Aspirin can make symptoms of tinnitus worse

A

TRUE
* tinnitus is caused by damage to hair cells in inner ear from exposure to excessive noise, medications (aspirin), aging and diseases

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97
Q

DBL + A =?

A

Frame PD

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98
Q

Minimum blank size equation

A

MBS = ED + TLD

ED: effective diameter
TLD: total lens decentration (TLD = |(A + DBL) - distance PD)

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99
Q

Which layer of the cornea is affected in acute hydrops in patients with keratoconus?

A

Descemet’s membrane

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100
Q

Transpose plus cyl into minus cyl form
OD: - 4.25 + 0.75 x 077
OS: - 4.00 + 1.00 x 098

A

OD: -3.50 - 0.75 x 167
OS: -3.00 - 1.00 x 008

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101
Q

What is the average annual progression rate for childhood myopia?

A

0.3 to 0.5 diopters per year

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102
Q

Is Positive relative accommodation indirect or direct measure of accommodation?

A

Indirect
* measures ability to increase accommodation while maintaining a clear, single binocular vision
* how much negative lens power tolerate before blur occurs at near target

Normal PRA -3.00
low PRA (early blur) = reduced accommodative ability (accommodative insufficiency or convergence excess)
High PRA (excessive minus tolerated) = overactive accommodation or accommodative spasm

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103
Q

Neomycin + poly B + dexamethasone

A

Maxitrol

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104
Q

Which IOP lowering drug class is contraindicated in patients with sickle cell?

A

Carbonic anhydrase inhibitors
*because they can cause metabolic acidosis, increase risk for hypoxia and exacerbate sickle cell retinopathy

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105
Q

Can a patient with sickle cell use acetazolamide to lower IOP?

A

NO!!!
Because it can cause metabolic acidosis
*this would make blood pH acidic and promotes red blood cell sickling

106
Q

What is the Americans with Disabilities Act? (ADA)

A

Civil rights law that prohibits discrimination against individuals with disabilities. Ensures equal opportunities and access to public spaces, employment, and transportation.

107
Q

Which drugs should be avoided in children and pregnant women because they damage blood marrow?

A

Methotrexate (immunomodulator)
Pyrimethamine (tx toxo)
Trimethoprim (+ sulfamethazine = Bactrim)

108
Q

Methotrexate is used to treat what autoimmune and inflammatory diseases?
(MOA: folate antagonist)

A

Rheumatoid arthritis
Psoriasis and psoriatic arthritis
Inflammatory bowel disease
Lupus
Uveitis and scleritis

*can also be used as cancer treatment for lymphomas and leukemias

109
Q

Which infectious disease is caused by inhalation of fungal spores from bird and bat droppings?

A

Histoplasmosis

110
Q

What conditions can cause both unilateral and bilateral swollen nerves?

A
  • Sarcoidosis
  • TB
  • Syphilis
  • Lupus

These conditions affect the optic nerves.

111
Q

What are the causes of premature cataracts?

A

Atopic Dermatitis, Diabetes, Myotonic Dystrophy, and Wilson’s Disease

Mnemonic: Wilson is MAD about early cataracts
M: myotonic dystrophy
A: atopic dermatitis
D: Diabetes

These conditions can lead to early cataract formation.

112
Q

How can swollen nerves be categorized?

A

Based on if the swelling is unilateral or bilateral

This classification helps in diagnosis.

113
Q

What causes bilateral swollen optic nerves (papilledema)?

A

Increased intracranial pressure

This can be due to various systemic conditions.

114
Q

What causes unilateral swollen nerves (papillitis)?

A

Complications anterior to the optic chiasm such as:
* Vascular (Diabetes, Central Retinal Vein Occlusions)
* Ischemic (NAION, AION)
* Compressive (Thyroid)
* Optic Disc Drusen

These factors lead to localized nerve swelling.

115
Q

What medications can cause Pseudotumor Cerebri?

A

Accutane, Idiopathic, Nalidixic Acid, Oral Contraceptives, Tetracyclines, and Vitamin A

These substances are linked to increased intracranial pressure.

116
Q

What causes retinal neovascularization and subsequent tractional retinal detachments?

A

Diabetic retinopathy, ocular ischemic syndrome, retinopathy of prematurity, sickle cell retinopathy, and vein occlusions

These conditions lead to abnormal blood vessel growth in the retina.

117
Q

What are the causes of transillumination defects?

A
  • Pigment dispersion syndrome
  • pseudoexfoliation syndrome
  • albinism
  • herpetic uveitis
  • trauma
  • PCIOL intraocular surgery
118
Q

What causes whorl keratopathy?

A

Amiodarone, Chloroquine, Hydroxychloroquine, Fabry’s Disease, Indomethacin, and Tamoxifen, Amiodarone

These substances can lead to characteristic corneal changes.

119
Q

What are the causes of hypopyon?

A

Bechet’s Disease, VKH Syndrome, Endophthalmitis, Bacterial Keratitis, and Blebitis

Retinoblastoma may also cause a pseudohypopyon.

120
Q

Which bacteria can invade the intact corneal epithelium?

A

Corynebacterium diphtheria, Haemophilus, Listeria, Neisseria gonorrhea, and Pseudomonas aeruginosa

These pathogens can lead to serious ocular infections.

121
Q

Nalidixic acid MOA

A

Inhibits DNA gyrase
* can cause increased intracranial pressure —> pseudotumor cerebri

122
Q

DEPRIVENS is the mnemonic for diseases that can cause cystoid macular edema, name them.

A

D: Diabetes
E: Epinephrine
P: Pars Planitis
R: Retinitis pigmentosa
I: Irvine-Gas’s
V: Vein Occlusions
E: E2-Prostaglandin
N: Nicotinic acid and Niacin
S: Surgery

123
Q

Imitrex drug class, MOA, indication and ocular sequelae

A

Serotonin receptor agonists

MOA: vasoconstrictor of cranial blood vessels

Indicated for migraines and cluster headaches

Can cause NAION

124
Q

What are the risk factors for retinal breaks?

A

High myopia, lattice degeneration, aphakia/pseudophakia, and trauma

These factors increase the likelihood of retinal detachment.

125
Q

What mnemonic can be used to remember the causes of Angioid Streaks?

A

PEPSI: Pseudoxanthoma Elasticum, Ehlers Danlos, Paget’s, Sickle Cell Disease, Idiopathic

Each letter of PEPSI corresponds to a specific condition that causes Angioid Streaks.

126
Q

What are the causes of blue sclera? (4)

A
  • Minocycline
  • Ehlers Danlos Syndrome
  • Rheumatoid Arthritis
  • Scleritis

Blue sclera: scleral appears blue due to thinning and increased transparency of the scleral collagen, allowing the underlying darker choroid vasculature to show through

127
Q

Name some causes of exudative retinal detachments. (5)

A
  • Central Serous Retinopathy
  • VKH Syndrome
  • Optic Pits
  • Morning-Glory Syndrome
  • Coats Disease

Exudative retinal detachments can result from several syndromes and diseases.

128
Q

What can cause bull’s eye maculopathy?

A
  • Chloroquine
  • Hydroxychloroquine
  • Progressive Cone Dystrophy
  • Stargardt’s Disease
  • Thioridazine

Bull’s eye maculopathy is associated with drug toxicity and genetic conditions.

129
Q

List some causes of keratoconus.

A

T-DOME

  • Turner Syndrome
  • Down’s Syndrome
  • Osteogenesis Imperfecta
  • Marfan’s Syndrome
  • Ehlers Danlos Syndrome
  • Atopy, excessive eye rubbing

Keratoconus is a progressive eye disease that can be associated with these syndromes.

130
Q

What are the causes of lens subluxation? (Name 4)

A
  • Blunt trauma
  • Marfan’s Syndrome
  • Homocystinuria
  • Ectopia Lentis et Pupillae
  • Hyperlysinemia
  • Microspherophakia
  • Sulfite Oxidase Deficiency
  • Simple Ectopia Lentis

Lens subluxation can occur due to genetic conditions or trauma.

131
Q

What can lead to macular thickening?

A
  • Chronic intraocular inflammation
  • Intraocular surgery
  • Intraocular tumor
  • BRVO
  • Best’s disease
  • Retinitis pigmentosa
  • Neuroretinitis

Rhegmatogenous retinal detachments do NOT cause macular thickening.

132
Q

Name conditions associated with nyctalopia? (Name at least 3)

A
  • Choroideremia
  • Gyrate Atrophy
  • Enhanced S-Cone Syndrome/Goldmann-Favre Syndrome
  • Thioridazine Retinopathy
  • Retinitis Pigmentosa
  • Leber’s congenital amaurosis

Nyctalopia: night blindness

133
Q

What can cause papillae?

A
  • Allergies
  • Bacteria
  • Idiopathic

The presence of papillae can be due to allergic reactions or infections.

134
Q
A

Capillary hemangioma
* most common BENIGN orbital tumors in children
* will spontaneously resolve/disappear by when the patient turns 10

135
Q

Patient unhappy with vision through new glasses, what is the FIRST thing you should do?

A

Verify the prescription is correct via lensometer

136
Q

Inheritance pattern for juvenile retinoschisis

A

X-linked recessive
*mostly affects males, females are carriers

137
Q

4.5 year old white male
OD: +0.75 DS
OS: +1.00 DS
BCVA 20/50 OD, OS

A

Juvenile retinoschisis
* X-linked recessive

138
Q

Pathophysiology of retinoschisis

A

Separation of nerve fiber layer from outer retinal layers

139
Q

True or false
Highest incidence of retinoschisis is in African Americans

A

FALSE
highest incidence in caucasians

140
Q

What are some causes of photophobia?

A
  • Albinism
  • Aniridia
  • Aphakia/Pseudophakia
  • Cataracts
  • Macular degeneration
  • Retinitis pigmentosa

Photophobia, or light sensitivity, can arise from various ocular conditions.

141
Q

What is the legal doctrine known as respondeat superior?

A

A doctrine meaning ‘let the master answer’ where an employer is responsible for the actions of an employee.

142
Q

Can a patient sue an optometrist directly for breaching confidentiality?

A

Yes, a patient can sue the optometrist for breaching confidentiality even if an employee made the disclosure.

143
Q

What federal act can a patient file a complaint under for a violation of confidentiality?

A

Health Insurance Portability and Accountability Act of 1996 (HIPAA)

144
Q

True or False: Patients can file private lawsuits under HIPAA.

145
Q

Who may pursue a claim against an optometrist’s office for a HIPAA violation?

A

The federal government

146
Q

What may some states do in response to professional misconduct by an optometrist?

A

Take action against the optometrist personally.

147
Q

What should be done if a breach of patient confidentiality occurs?

A

Get immediate legal counsel.

148
Q

What is the best treatment for CME s/p yag cap

A

Topical steroid + topical NSAID

Example: 1 gtts ketorolac QID and 1 gtts prednisone acetate QID

149
Q

What is the fluorescein angiography pattern for CME?

A

Small HYPERfluorescent spots in early phase with “flower petal” pattern of HYPERfluorescent in late stage

150
Q

Chronic long standing CME can lead to?

A

Lamellar hole
*small fluid filled cystic spaces may fuse to form larger fovea cysts and coalesce and form a lamellar macular hole
* lamellar hole has poorer acuity and worse prognosis because it can cause irreversible damage to central vision

151
Q

White without pressure is commonly observed in which region of the retina?

A

Inferior temporal
*at the location of the vitreous base and Ora serrata

152
Q

Retinal dialysis

A

Retina torn from its insertion site at the Ora serrata

153
Q

Signs of blunt trauma to eye

A

Retinal dialysis
Choroidal rupture
Commotio retinae
Berlin’s edema
Hyphema
Lens subluxation
Iridodialysis

154
Q

Which population has the highest occurrence of white without pressure?

A

African Americans

155
Q

Which ancillary procedure should be performed if you have a patient that has white without pressure?

A

Scleral depression
*check for retinal holes, tears, or retinal detachments

156
Q

Cobblestone degeneration commonly found in which area of the retina?

A

Inferior temporal

157
Q

Potential surgical treatments for keratoconus

A

Corneal cross-linking
Deep anterior lamellar keratoplasty
Penetrating keratopasty

158
Q

Initial management of acute hydrops

A

1) Cycloplegic (reduce pain/photophobia)
2) Antibiotic (prophylactic to prevent infection)
3) hypertonic solution (reduce corneal edema)

atropine
ciprofloxacin
sodium chloride (use once defect has healed)

159
Q

Tx interstitial keratitis

A

Strong steroid (Pred acetate)

*If herpes simplex keratitis treat with topical antiviral like trifluridine

160
Q

Ocular HTN

A
  • IOP > 21 mmHg with open angle + without optic neuropathy
161
Q

Fill in the blank: The American Optometric Association (AOA) states that a breach of patient confidentiality can be the basis for a _______.

A

malpractice claim

162
Q

What is MEGALOCORNEA?

A

Rare, x-linked condition in males with horizontal corneal diameter greater than 13 mm.

Common in Marfan’s syndrome, Ehlers-Danlos, osteogenesis imperfecta. Associated with myopia, lens subluxation, angle abnormalities (glaucoma).

163
Q

What is MICROCORNEA?

A

Rare, autosomal dominant condition with horizontal corneal diameter < 10 mm.

Associated with hyperopia and risk for angle closure.

164
Q

What characterizes CORNEA PLANA?

A

Corneal curvature equal to scleral curvature (K ~ 20-30 D).

Associated with hyperopia, shallow anterior chamber, sclerocornea.

165
Q

What is ANIRIDIA?

A

Partial or complete loss of iris tissue.

Associated with glaucoma, foveal hypoplasia, disc hypoplasia, choroidal coloboma.

166
Q

What are HAAB’S STRIAE?

A

Horizontal cracks in Descemet’s membrane due to congenital glaucoma.

Indicate increased intraocular pressure.

167
Q

What is AXENFELD-RIEGER SYNDROME?

A

Developmental arrest of neural crest mesectoderm affecting anterior segment structures.

Type 1 has iridocorneal adhesions; Type 2 has lenticular-corneal adhesions.

168
Q

What is POSTERIOR EMBRYOTOXON?

A

Anteriorly displaced Schwalbe’s line.

Found in up to 15% of normal eyes.

169
Q

What characterizes AXENFELD ANOMALY?

A

Posterior embryotoxon plus angle abnormalities.

Increases risk for glaucoma.

170
Q

What is RIEGER ANOMALY?

A

Posterior embryotoxon plus angle abnormalities and iris stromal hypoplasia.

May include polycoria, corectopia, anterior synechiae.

Polycoria: more than 1 pupillary opening
Corectopia: pupil displacement
Anterior synechiae: iris sticks to cornea

171
Q

What is RIEGER SYNDROME?

A

Rieger anomaly plus systemic deformities affecting face, teeth, and bones.

172
Q

What is PETER’S ANOMALY?

A

Autosomal recessive condition with white corneal opacities and iris adhesions.

Can lead to secondary glaucoma.

173
Q

What is DELLEN?

A

Area of poor wetting leading to stromal dehydration and corneal thinning.

Risk factors include contact lens wear and filtering bleb.

174
Q

What is EXPOSURE KERATOPATHY?

A

Condition resulting from eyelid issues such as Bell’s palsy or ectropion.
*lagophthalmos, incomplete eyelid closure

175
Q

What is FILAMENTARY KERATOPATHY?

A

Chronic irritation or dryness resulting in filaments formed by degenerated epithelial cells and mucous.

176
Q

What is THYGESON’S SPK?

A

Acute recurrent bilateral inflammation with raised central corneal lesions.

Typically seen in younger patients.

177
Q

What is NEUROTROPHIC KERATITIS?

A

Loss of corneal epithelial repair due to damage to the trigeminal nerve.

Conditions include HSV, HZO, and diabetes.

178
Q

What is RECURRENT CORNEAL EROSION (RCE)?

A

Condition often associated with corneal dystrophies and trauma.

179
Q

What causes THERMAL/UV KERATOPATHY?

A

Prolonged sun exposure or welding without protective eyewear.

Results in apoptosis of corneal cells.

180
Q

What is KERATOCONJUNCTIVITIS SICCA?

A

Dry eye disease characterized by decreased tear production.

181
Q

What is the Schirmer 1 test?

A

Measures basal + emotional + reflex aqueous secretions; < 10 mm in 5 min indicates dry eye.

182
Q

What is AQUEOUS DEFICIENT dry eye?

183
Q

What is EVAPORATIVE dry eye?

A

Condition characterized by meibomian gland dysfunction or low blink rate.

184
Q

What is SUPERIOR EPITHELIAL ARCUATE LESION (SEAL)?

A

Condition due to tight contact lenses causing upper lid chafing.

185
Q

What is DIMPLE VEILING?

A

Occurs with RGP lenses that have excess inferior edge lift.

186
Q

What is CL ACUTE RED EYE?

A

Inflammatory reaction to Gram-negative bacterial toxins.

187
Q

What is CORNEAL WARPAGE (OVERWEAR SYNDROME)?

A

Condition seen in long-term PMMA or RGP wearers with poorly fitting lenses.

188
Q

What is SUPERIOR LIMBIC KERATOCONJUNCTIVITIS (SLK)?

A

Condition due to contact lens hypersensitivity or poor fitting lenses.

189
Q

What is TIGHT LENS SYNDROME?

A

Condition where contact lenses do not move with blinking, leading to redness and edema.

190
Q

Which hypersensitivity reaction is Staph marginal keratitis?

A

Type 3
Ag-Ab complex—-> complement response

191
Q

Hudson-Stahli line is iron deposits in which layer of the cornea?

A

Epithelium
* horizontal corneal epithelial iron line at the inferior third of the cornea at the level of tear film caused by normal aging

192
Q

Reis-Buckler

A

Rare, autosomal dominant corneal dystrophy
* donut shaped gray reticular opacities, painful episodes of RCE
*honeycombs

193
Q

Cone dystrophy

A

Slow progressive decrease central vision
* severe photophobia
* blue/yellow color loss
* central geographic atrophy of RPE —> bulls eye maculopathy
* temporal ONH pallor

194
Q

Tx for IIH

A

If pt obese, initiate program for weight loss (5-10%)

195
Q

Tx papilledema

A

Oral acetazolamide 1000-2000 mg po QD in divided doses, up to 4000 mg qd

196
Q

Pseudotumor cerebri may be associated with what conditions/ medications?

A

Conditions:
Venous sinus thrombosis
COPD
Pregnancy

Medications:
Tetracyclines
Vitamin A
Isotrentinoin (Accutane)
Contraceptives
Fluoroquinolones
Levothyroxine
Nalidixic acid

197
Q

Malignant HTN

A

Systolic >180 mmHg
Diastolic > 120 mmHg

198
Q

What are the phases of FA?

199
Q

Parinaud’s oculoglandular syndrome

A

Unilateral conjunctivitis with conjunctival granulomas and preauricular or submandibular lymphadenopathy
*may have fever, malaise and rash
* caused by cat-scratch fever, TB, syphilis, Epstein-Barr Virus, fungi, sarcoidosis

200
Q

Cat scratch disease caused by?

A

Bartonella henselae infection

201
Q

Wilson’s disease pathophysiology

A

Abnormal accumulation of copper in body
*affects pt 5-40 years old
*hepatolenticular disease

202
Q

Signs of Wilson’s disease

A

Kayser-Fleischer ring (copper at descemet’s)
Sunflower cataract

203
Q

In which layer of the cornea is copper deposited in Kayser-Fleischer ring?

A

Descemet’s membrane

204
Q

Chronic Alcoholism ocular signs

A

1) Optic nerve pallor
2) Up and downbeat nystagmus due to nerve hydrocephalus and demyelination
*alcoholism leads to nutritional deficit

205
Q

True or false
Staph marginal keratitis is a type III hypersensitivity reaction

206
Q

What are the most common cancers in men?

A

Prostate, lung, colon

207
Q

Most common cancers that lead to death in men and women?

A

Men: lung, prostate, colon
Women: lung, breast, colon

208
Q

Randot stereoacuity of < 40 arcseconds

A

Normal binocular stereoacuity

209
Q

50-100 arc seconds in randot stereoacuity means?

A

Good stereo vision

210
Q

Randot stereoacuity of 100-400 arc seconds means?

A

Fair stereo vision, mild impairment

211
Q

> 400 arc seconds or unable to perceive on Randot stereoacuity means what?

A

Poor or absent stereo vision (possible strab or amblyopia)

212
Q

Reye syndrome ocular manifestations
(Brain swelling)

A
  1. Dilated pupils
  2. Cortical blindness
  3. Papilledema
213
Q

SEI

A

Subepithelial infiltrates

214
Q

Tx for SLK

A

Silver nitrate solution
Bandage CL
Mast cell stabilizer (cromolyn QID, olopatadine BID)

215
Q

Syphilis is caused by what organism?

A

Treponema pallidum

  • sphirochette bacteria
  • 3 stages
  • end stage: neurosyphilis
  • congenital or acquired
  • congenital (Hutchinson’s triad: peg-shaped teeth, deafness and interstitial keratitis)
  • interstitial keratitis is inflammation of corneal stroma without prior epi or endo involvement
216
Q

Pulfrich phenomenon

A

Perception of Stereopsis (elliptical motion of pendulum) caused by difference in nerve conduction times between eyes and cortex
*seen in multiple sclerosis

217
Q

What is the phenomenon where a patient perceives elliptical motion of a pendulum? It is associated with multiple sclerosis

A

Pulfrich phenomenon

218
Q

Visual field anomaly in which a moving object can be seen but not a static one

A

Riddoch phenomenon

219
Q

Hyper mature cortical cataract in which liquified cortex allows nucleus to sink inferiorly

A

Morgangnian cataract

220
Q

Increased pigmentation anterior to Schwalbe line in pseudoexfoliation syndrome

A

Sampaoelesi line

221
Q

Scheie line

A

Pigment on lens equator and posterior capsule in pigment dispersion syndrome

222
Q

Forced vergence fixation disparity curve for eso fixation disparity and show poor adaptation to BI

223
Q

Does this forced vergence fixation disparity curve show eso or exo?

224
Q

What is the purpose of forced vergence fixation disparity curves?

A

Evaluate the efficiency of a patient’s Vergence system by observing how fixation disparity changes when different amounts of base-in and base-out prisms are introduced to the eyes

225
Q

Fusional vergence dysfunction symptoms

A
  1. Asthenopia at near
  2. Eye strain and headaches after short periods of reading, blurred vision, difficulty concentrating, sleepiness when performing near tasks
226
Q

What condition does this patient have?

Decreased NRA and PRA
Decreased NFV and PFV
Deceased accommodative facility
Normal phorias at distance and near
Normal AC/A

A

Fusional vergence dysfunction

227
Q

Vision therapy Exercises for convergence insufficiency

A

Pencil push ups
Eccentric circles free-space fusional cards
Brock string

228
Q
A

Persistent pupillary membrane
*congenital
* remnants of anterior tunica vasculosa lentis (did not fully disintegrate)
* does not affect vision

229
Q

Which tests directly measure the accommodative system?

A

Monocular facility testing with +/- 2.00 lenses
Monocular estimation method
Monocular amplitudes

230
Q

BIM is hard for exo or eso?

A

ESO
* difficult to clear with base in and minus power lenses

231
Q

True or false
Lag on MEM in basic esophoria

232
Q

Inheritance pattern for osteogenesis imperfecta

A

Autosomal dominant
* congenital
* brittle bones because abnormal production of type 1 collagen
* blue sclera

233
Q

What is the most common accommodative dysfunction observed in the general population?

A

Accommodative insufficiency

234
Q

Herbert pits

A

Scarred limbal follicles
*chalmydial conjunctivitis

235
Q

What caution should be exercised when prescribing tiotropium (Spiriva)

A

Caution with narrow angles/ hyperopic patients
*because drug causes mydriasis and can lead to angle closure—> increase IOP —> glaucoma

236
Q

Tilted disc syndrome typically causes what visual field defect?

A

Bilateral superior temporal defects
*disc insertion at an oblique angle inferior nasal —> superior temporal defect that does not respect the midline

237
Q

Crouzon syndrome is associated with what optic nerve condition?

A

Tilted disc syndrome
* Crouzon syndrome is autosomal dominant, results in proptosis due to shallow ocular orbits, maxillary hypoplasia, abnormal craniofacial formation, hypertelorism, and strabismus
*monitor for exposure keratitis because all affected with proptosis

238
Q

Average axial length

239
Q

Ocular signs of isolated complete cranial nerve 3 palsy

A

EOM limitations in ALL fields of gaze except temporally

240
Q

Partial EOM limitation in all field of gaze except temporally described what external ophthalmoplegia?

A

Incomplete isolated cranial nerve 3 palsy

241
Q

What is 3 AND 9 O’CLOCK STAINING?

A

Common complication of GP lens wear due to low-riding lenses that leave parts of the cornea uncovered.

242
Q

What is the definition of external ophthalmoplegia?

A

Extraocular muscle impairment

This refers to the inability to move the eyes properly due to muscle dysfunction.

243
Q

What characterizes complete 3rd nerve palsy?

A

Extraocular muscle limitation in all fields of gaze except temporally

This means the patient cannot move their eyes in any direction except outward.

244
Q

What distinguishes incomplete 3rd nerve palsy from complete 3rd nerve palsy?

A

Partial extraocular muscle limitation in all fields of gaze except temporally

Incomplete palsy allows for some movement in certain directions.

245
Q

What is a superior division palsy?

A

Inability to look up only

This type of palsy affects the muscles responsible for upward eye movement.

246
Q

What is an inferior division palsy?

A

Inability to look nasally or inferiorly, with a dilated pupil

This indicates that the muscles controlling downward and inward movement are affected.

247
Q

What does internal ophthalmoplegia refer to?

A

Pupil reaction

This involves the response of the pupil to light and accommodation.

248
Q

What is a pupil-involving condition?

A

Fixed, dilated, poorly reactive pupillary response to light

This indicates significant nerve damage affecting pupillary function.

249
Q

What is a pupil-sparing condition?

A

Pupil is not dilated; normal response to light

This suggests that the pupil is functioning normally despite other symptoms.

250
Q

What characterizes a relative pupil-sparing condition?

A

Partially dilated pupil; sluggish reaction to light

This indicates some impairment in pupillary function, but not complete loss.

251
Q

What is the first step in evaluating patients with an acute isolated third nerve palsy?

A

Immediate CNS imaging should be completed

This is to rule out a space-occupying lesion or aneurysm.

252
Q

What type of third nerve palsy requires immediate CNS imaging?

A

Pupil-involving 3rd nerve palsies

This includes either relative or complete involvement.

253
Q

List the associated factors for pupil-sparing third nerve palsies that necessitate CNS imaging.

A
  • Patients <50 years old (unless long-standing diabetes or hypertension is present)
  • Incomplete third nerve palsies
  • Patients with additional cranial nerve abnormalities or neurologic symptoms
  • Children <10 years old (regardless of pupil status)

Incomplete palsies may evolve into complete pupil-involving palsies.

254
Q

What is a situation where prompt CNS imaging should be obtained?

A

When aberrant regeneration develops

This excludes regeneration in post-traumatic third nerve palsies.

255
Q

When should imaging be obtained for pupil-sparing third nerve palsies?

A

If they have not resolved within a period of 3 months

Close monitoring is necessary for signs or symptoms progression.

256
Q

Is imaging typically required for complete pupil-sparing third nerve palsy?

A

No, it is not typically required

Especially in patients >50 years old with a known history of vascular disease.

257
Q

In order to determine unknown etiology of anisocoria, which drop would you instill if anisocoria was greater in dim illumination?

A

10% cocaine
*will dilate normal pupils only

258
Q

In order to determine unknown etiology of anisocoria, which drop would you instill if anisocoria was greater in bright conditions?

A

0.125% pilocarpine
*check if enlarged pupil will constrict
*0.125% pilocarpine will not dilate normal pupil

259
Q

True or False: Imaging is required for all patients with third nerve palsy.

A

False

Imaging is not typically required for complete pupil-sparing third nerve palsies.