Hypglycemia Flashcards

1
Q

What is Whipple’s triad?

A

Blood glucose <70mg/dl
Sx of hypoglycemia
S/s resolve with glucose elevation

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2
Q

What is the range of glucose levels that sx begin and impairment of brain function?

A

Sx begin at 60mg/dl
Brain function is affected at 50mg/dl

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3
Q

What are the 2 principal types of hypoglycemia?

A

Fasting and postprandial

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4
Q

How does fasting hypoglycemia manifest?

A

Either subacute or chronic with neuroglycopenia as principal manifestation

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5
Q

How does postprandial hypoglycemia manifest?

A

Acute with sx of neurogenic autonomic d/c (sweating, palpitations, anxiety and tremulousness)

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6
Q

What are the broad categories of hypoglycemic manifestations?

A

Neuroglycopenic
Sympathomimetic

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7
Q

Why and how does neuroglycopenia manifest?

A

Decline of serum sugar, manifests with alterations of consciousness, lethargy, confusion, combativeness, agitation and unresponsiveness

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8
Q

Why and how does sympathomimetic hypoglycemia manifest?

A

Rapid fall in glucose level causes secretion of epinephrine and norepinephrine
Sx include anxiety, nervousness, irritability, nausea, vomiting, palpitations and tremor

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9
Q

What kind of hypoglycemia is common after GI surgery?

A

Postprandial hypoglycemia

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10
Q

What lab should be done for hypoglycemia?

A

Finger stick blood glucose

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11
Q

If a hypoglycemic patient has no altered mental status, how would you treat?

A

Eat or drink

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12
Q

A patient unable to eat or drink due to altered mental status, what is the treatment?

A

Glucagon 0.5mg or 1mg SC/IM
50% dextrose 50-100ml IV bolus

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13
Q

Why would a patient with hypoglycemia be MEDEVAC’d?

A

Continued or recurrent altered mental status, recurrent hypoglycemia or downward trend in glucose after therapy

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