HTN In Pregnancy Flashcards

1
Q

Gestational HTN

A

BP > 140/90 AFTER 20wks and returns to normal w/in 6 wks postpartum

Considered chronic HTN if last longer
Don’t show any other s/s, NO PROTEINURIA
May progress to preeclampsia

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2
Q

CHRONIC HTN

A

HTN BEFORE 20wks pregnancy or lasts >12wks postpartum

Can develop superimposed preeclampsia
Concern during pregnancy blood flow to placenta

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3
Q

MILD PREECLAMPISA

A

BP increases by 30/15 or >= 140/90 AFTER 20wks GA, Proteinuria >= +1 in random urine dipstick
s/s: edema around eyes, face, & fingers, weight gain > 2lbs/ wk, hyperreflexia 3+ usually w/ clonus, CNS s/s, mild headache, IUGR (placenta doesn’t provide enough blood supply)

Multi- organ failure = thrombocytopenia (< 150,00), increase CRT (>1)

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4
Q

Patho Preeclampsia

A

placenta inserts-> poor perfusion -> endothelial cell dysfxn
* Vasospams ; decrease blood flow to organs (inc.brain) & increase BP
* Increase Peripheal resistance
* Increase endothelial cell permeability

Disease of CNS irritablility
goes away when placenta delivered
CURE = Delivery of Baby

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5
Q

BIGGEST CONCERN OF PREECLAMPSIA

A

GRAND MAL SEIZURES

Can be Fatal

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6
Q

ASSESSMENT OF MILD PREECLAMPSIA

A

VS Q4HRS, DEEP TEDNON REFLEXES, LOC

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7
Q

Interventions of Mild Preeclampsia

A

Can send home of bed rest, may hospitalized if severe
Cont. Monitoring
Left lateral bed rest
IV w/ Fluid restrictions
Insert a foley-> strict I/O > 30 ml/hr
DTR w/ clonus Qshift or Q4hrs
Early delivery -> monitor closely to determine when
* may induce labor or C-Section if HELLP suspected
Home care: montior fetal activity, BP & Weight, urinalysis for protein,light salt & protein diet
* come in ASAP if decrease in Fetal Movement
* see pt usualy every other day
* call provider w/ epigastric pain, N/V, RUQ pain

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8
Q

MED TREATMENT FOR MILD PREECLAMPSIA

A

Mg + sulfate
Given IV in low dose 1st then continuous 2g.hr
* often becomes flush & N/V w/ start of low dose

CNS irritability depressant & helps prevent seizures ( not to low BP, even though it does)
Need to be on seizure precautions !!!
TOXICITY s/s:absent DTR, change in LOC, decrease RR (< 12)
* if developing any s/s: turn off Mg+, give Ca+ gluconate (antidote), notitfy MD (stop delivering aids, like pitocin)
* Increase IV Fluids & wait till s/s goes away

Continue 24hrs after delivery to prevent seizures
Labetalol or Hydralazine-> anti-HTN
Bethamethasone, Dexamethasone -> Helps w/ fetal lung maturity

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9
Q

HELLP SYNDROME

A

Hemolysis
Elevated Liver Enzymes
Low Platelets
S/S: malaise, epigastric or RUQ pain, visual disturbance, severe headache

Can’t clot, liver is not clear toxins, could see liver, blood values sen

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10
Q

Severe Preeclampsia

A

Same as mild execpt +2 of the following:
* BP 160/110 on >= occassions
* Proteinuria >= 3 (uric acid, creatintine increased)
* edema - very puffy face (periorbital) & hands
* pitting pedal edema = normal preg finding

Admitted directly into hospital - need ASAP delivery b/c pt going to have a seizure

S/S: DTR >= +3 & clonus, proteinura >= +3, oliguria (< 100 mL/4hrs)

CNS S/S: severe HA, visual distrubance (hyperelexia) (blurry, photophobia, blind spots), epigastric pain (above umbilicus)

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11
Q

Interventions for Severe Preeclampsia

A

1st: seizure precautions
* no brain stim: dark room, tv off, no electronics, no noise, no stim ppl, quiet private room & closed door
* Pad side rails, suction @ bedside, O2
* Aim for vag birth b/c don’t want to cut open

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12
Q

Drug treatment for Severe Preeclampsia

A

Mg+ sulfate: only drug used that helps
Anti HTN agents rare, only given if DBP > 110 b/c of CVA risk
* Drug choice if needed is Apresoline Hydralazine HCL

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13
Q

Chronic HTN

A

HTN BEFORE 20 wks pregnancy or lasts >12 wks postpartum
* can develop superimposed preeclampsia
* concern during pregnancy blood flow to placenta

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14
Q

Eclampsia

A

Preeclampsia progression to generalized seizures not r/t other causes
* Worsening preeclampsia s/s
* Seizures can occur b/4, during, or after labor
* Tonic-clonic seizure

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15
Q

When To Deliver

A
  • Presistent increase in BP
  • Development of severe cerebral s/s
  • progressive thrombocytopenia ( < 100,000/mU)
  • **ROM, bleeding **
  • Abnormal liver enzymes
  • Presisent RUQ pain
  • Severe IUGR
  • Non reasurring fetal assessment
  • **Oligohydramnois ( decrease amniotic fluid) **
  • GA 38-40 wks

s/s get worse, Mild preeclampsia home on bed rest and fetal kick count

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16
Q

Etiology

Patho

Preeclampsia is a progressive disorder, with the placenta as the root cause.

A
  • Poor perfusion and endothelial cell dysfunction (contributes significantly to restriction of fetal growth)
  • Arteriolar vasospam diminishes diameter of blood vessels, which impedes blood flow to all organs and increase BP
    *

Placenta Ischemic =Endothelial cell dysfunction = vasospasm, increase peripheral resistance, increased permeability

17
Q

Mg+ Sulfate

prevent seizure

A

Monitor U/O < 30 ml/hr, RR < 12/ min, Stop if 0 DTRS (absent) , Decel FHR, mom O2 % < 95%
Toxicity:
* S/S: Lethargy, absent reflexes(q1hr), slurred speech, bradycardy , bradypnea (q1hr), mom hypoTN, Altered sensorium confusion (q1hr)
1. STOP mg!
2. IV fluid
3. Calcium Gluconate ( @ beside)
4. Notfiy MD

MOM HypoTN and confusion

2g/hr contains, stay on 24hr after delivery to decreasse the risk of postpartum seizures

18
Q

Mg+ and Pitocin

A

Need to have both one to prevent seizure and induce labor for pt who has Preeclampsia.

Want a vag birth, carefull about c-section b/c of HELLP syndrome

19
Q

Preeclampsia Meds

A

Lebatolol/Hydralazine: HTN ( no ACE)
Asones: Fetal lung maturity
Mg+ Sulf: Seizure

20
Q

DEEP TENDON REFLEXES

A

0= Absent
3= Brisk
4= Very Brisk w/ clonus

clonus= # beats