Head & neck Flashcards
Bad signs of acute subdural haematoma
4
Heterogeneity
Convexity
Inc Mass effect
SAH
Features of isodense SDH
Further imaging
Unexplained mass effect
Effaced sulci
“thick’ cortex”
WM buckling
Do contrast
MR diagnostic
Traumatic brain haematoma - ring enhancing lesion
Diagnostic aids/clues
4
Bilateral
Orbitofrontal
Haemosiderin ring
Other injuries
Diffuse axonal injury
Grading - 3
Deeper lesions worse prognosis
1 : lobar WM. Lesions bloom
2: involves corpus callosum typically splenium
3: Dorsolateral midbrain
Worrying features of skull fractures
7
Overlies a dural sinus Overlies MMA Overlies eloquent cortex Depressed > table width Open/compound Traverses ICA canal Temporal bone # ( opacification of mastoid air cells) Skull base # ( blood in sphenoid sinus)
Carotico-cavernous fistula CT findings
5
Proptosis Enlarged extra ocular muscles Convex ipsilateral CS Enlarged superior opthalmic vein Retrobulbar fat stranding Check Brain - venous infarcts
Early NCCT signs of stroke
3
Penumbra on PWI : at risk area
DWI : infarct core
Loss of GW differentiation : insular ribbon sign, BG effacement (less delineation)
Dense artery sign (30%) - asymm, correlates c Sx
Hypodensity
Subacute : gyriform CE
Distal Carotid artery dissection features
mid to distal ICA below cavernous segment
Bulb spared
NO false lumen
Subintimal haematoma ( do MRI T1 FS - methaemaglobin in wall)
DSA may be normal!
Proximal carotid artery dissection features
Progression of aortic dissection CCA ICA extension uncommon False lumen May be asymptomatic
ICA dissection imaging signs
location unusual for atherosclerosis ( bulb spared) string sign aneurysmal dilatation occlusion intimal flap may be seen
Amyloid angiopathy
features
B-amyloid peptide deposition
Lobar haemorrhage in elderly (15% >60y, 50%>70y)
Subcortical microhaemorrhages, spares BG cf HTN
Non traumatic ICH
causes
Underlying lesion: tumour, AVM, cavernoma, aneurysm Amyloid angiopathy Dural sinus thrombosis Hypertension Coagulopathy
Hypertensive haemorrhage
locations & pattern
Putamen/EC - 60%
Thalamus - 15-25%
Pons/CBL - 10%
Lobar 5-15%
Acute focal haematoma
Subacute/ch microbleeds
CTA dot sign - active bleed
Cavernous malformation
features
Discrete collections of endothelial lined sinusoids
Vascular hamartoma
ICH risk - 0.15-1% annually
Sporadic- 75%, familial 10-30% ( fam cavernomatosis)
Angiographically occult
Associated DVA
COMPLETE haemosiderin rim
Variable SI locules
Popcorn appearance
Capillary telangiectasia in brain
Brainstem Ass with radiation No T2 abnormality Brush like Ca+ GRE blooming Do not touch
Spetzler-Martin Grading AVM
Size
Eloquence
Venous drainage
<3cm - 1
3-6cm -2
>6cm - 3
Eloquence 1 - 0
Venous drainage superficial/deep
RF for intracranial aneurysms
9
Smoking Cocaine Age > 50y Female > Male FMD Anatomic variants eg trigem artery Type IV Ehlers Danlos NF 1 ADPCKD
Trigeminal artery vs Persistent fetal PCA
Trigeminal originates from CAVERNOUS ICA to BA
persist fetal from supraclinoid ICA (above sella)
PseudoSAH
causes
4
Cellular material ( infection, carcinomatosis, granulomatous etc, clean ventricles)
Iodinated contrast (myelogram, leak due to RF etc)
CSF Hypotension
Anoxia
Pseudopseudo SAH
Global anoxia/hypoxia due to dense venous stasis (white cerebellum, indistinct BG, no hydrocephalus)
CSF hypotension : ?crowded normal blood vessels, SD collections and large dural sinuses
No hydrocephalus
Idiopathic intracranial hypertension
( BIH, pseudotumour cerebri)
Features
RF
Women 3rd decade Obese/weight gain May be drug induced(steroids) Papilloedema Headaches, visual obs, diplopia, pulsatile tinnitus
Look for secondary causes Empty sella Flat post globe Vert tortuous , distended ON sheath Slit like ventricles - rare Focal compression mid transverse sinuses (pseudostenosis)