Abdomen Flashcards
Benign liver cyts
Demo
RF
Complications
Developmental Common 14% Round/ovoid Fluid SI imperceptible, non CE wall Single/multiple Haemorrhage is rare
High T2 liver lesions mistaken for cysts
Cystic metastases (hypervascular tumours; colon, ovarian cystadenocarcinoma, squamous cell lung cancer.
sarcomas, melanoma.
GIST,pancreatic mucinous cystadenocarcinoma.
Metastatic NET
Haemangioma
Biliary cystadenoma/carcinoma
Liver haemangioma
demo
Common (7-20%) F>M R>L lobe Lobulated, clefted appearance T2 very bright CE follows blood pool -continues to enhance on delayed
Interrupted peripheral nodular enhancement
Homogeneous CE by 10-20”
DDx for nodular interrupted CE - liver
Treated mets
Haemangioendothelioma: paed, (periph, confluent lesions)
Angiosarcoma : heterogeneous
Focal liver fat
locations
RF
Classic locations : falciform ligament, GB fossa, periportal
Geographic low density
Geographic inc CE
No mass effect
Non displaced vessels
Out of phase signal loss
Central high density on CECT
Focal nodular hyperplasia
Demo
RF
No underlying CLCD
3% Young females (80-95%) Solitary 80% Lobulated Subcapsular
iso/low T1; iso/sl high T2
Central scar 50-75%) : low T1/high T2(cf fibrolam HCC)
Homogeneous CE
Scar : delayed CE
Do sulphur colloid if unsure (55%)
Hepatocyte specific contrast
Liver adenomas
OCP Heterogen T1/T2 Can have Haem / FAT Hetero CE Can undergo malignant Tx
Hepatic metastases
usually hypovascular
HA supply ( high CE in arterial followed by washout - no PV supply)
Hypo on CT - no normal liver cells
High T2, low T! unless blood/melanin
Calcified liver mets
2
Serous ovarian Ca
Mucinous CRC
Hypervascular liver lesions
Benign 4
Malignant 2
Haemangioma
FNH
Adenoma
Hyperplastic regenerative nodules in Budd-Chiari
HCC
Hypervascular mets
Cholangiocarcinoma
types 3
Intrahepatic (20-30%) : mass forming, periductal infiltrating, intraductal
Peripheral
Hilar
Cirrhosis - early RF 5
Maybe inapparent
Hepatomegaly
Heterogeneous perfusion
Enlargement of hilar periportal space
Expansion of intersegmental fissure
Regenerating nodules
imaging
T1/T2 low
May contain Fe ( siderotic ) - SWI artefact
May contain Cu - non fero
Cirrhosis - advanced
Measure
Atrophy - Right lobe, medial segment of left lobe ( expanded GB fossa )
Hypertrophy :
Caudate, lateral seg LL
Caudate: RL ratio > 0.65 ratio
Dysplastic nodules
RF
Classically:
T1 high,
T2 low/iso
Does NOT enhance
Focal confluent fibrosis
Anterior/medial segments Wedge shaped May involve entire segment Capsular retraction Delayed Gad enhancement
Hyperattenuating liver on CT
5
Haemachromatosis Amiodarone Type IV glycogen storage disease Wilsons Thorotrast
Haemosiderosis
Dyseryhtropoesis
Transfusion
Excessive Fe accumulation in RES ie
Liver ( kuppfer cells), spleen, bone marrow
Budd Chiari
Presentation
Causes 5
Types 3
Hepatomegaly, ascites, jaundice
Causes : Idiopathic, IVC webs, tumours, pregnancy, hypercoaguable states
Type
I : IVC +/- secondary HV
II : HV +/- secon IVC
III - venoocclusive
Budd Chiari
RF
Delayed of reversed PV inflow
Hepatomegaly Congestion - low T1/high T2 "nutmeg' liver Patchy central CE Peripheral hypoperfusion Reversed on delays
Small HV/IVC
Intravascular thrombi
Hepatic infarcts
PV thrombosis (20%)
Chronic - atrophy of liver, caudate hypertrophy
Hepatic infarction
Rare Peripheral wedge shaped/geographic Low density on CT Low T1/high T2 Changes shape with time Necrosis (air) Bile lakes Eventual atrophy
CT signs of acute appendicitis
6
Fat stranding Diameter >6mm (intraluminal air is usually normal) Focal caecal thickening Appendicolith Paracolic gutter fluid Adenopathy
CT signs of acute diverticulitis
4
Colonic wall thickening (7-10mm)
Pericolic fat infiltration
Abscesses
Extraluminal air
Epiploic appendagitis vs Omental infarction ( CT)
appendagitis : oval pericolonic dirty fat with HIGH density rim.
Signs of strangulation, closed loop or ischaemic bowel
Excess free fluid Hazy, paper thin walls Mesenteric fat stranding Distended single loop Non enhancing, thickened walls Pneumatosis (Free air)