Head and neck disease (Yr3) Flashcards

1
Q

where can unilateral nasal discharge be localised to?

A

sinus or nasal passage

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2
Q

where can bilateral nasal discharge be localised to?

A

guttural pouch, larynx, pharynx, lower respiratory tract

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3
Q

what innervates the motor control of the nares?

A

facial nerve

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4
Q

what are the typical causes of facial nerve paresis/paralysis?

A

recumbency where there is pressure on nerve (GA) or iatrogenic (surgery)

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5
Q

what is another name for a nasal atheroma?

A

epidermal inclusion cyst

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6
Q

what anatomical region do epidermal inclusion cysts (nasal atheroma) form?

A

nasal diverticulum

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7
Q

what is the clinical signs associated with epidermal inclusion cysts (nasal atheroma)?

A

non-painful swelling at nasoincisive notch

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8
Q

how can epidermal inclusion cysts (nasal atheroma) be treated?

A

surgical removal (often carries a good prognosis)

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9
Q

what are the signs associated with alar fold collapse?

A

respiratory noise at exercise (fluttering) and poor performance

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10
Q

how is collapse of the alar folds treated?

A

resection

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11
Q

what is a progressive ethmoid haematoma?

A

encapsulated non-neoplastic locally invasive mass that grows in the nasal passage and paranasal sinuses

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12
Q

how are progressive ethmoid haematomas treated?

A

nasal passage - intralesional formalin or laser excision
sinuses - sinus flap surgery

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13
Q

what are the clinical signs of fungal rhinitis?

A

unilateral purulent/haemorrhagic nasal discharge
malodorous smell
nasal stertor

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14
Q

how is fungal rhinitis treated?

A

removal of plaques and necrotic bone
topical enilconazole lavage

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15
Q

how many paranasal sinuses are there?

A

7 pairs (2 functional groups - rostral and caudal)

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16
Q

what are the rostral paranasal sinuses?

A

rostral maxillary
ventral conchal

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17
Q

what are the caudal paranasal sinuses?

A

caudal maxillary
frontal
dorsal conchal
sphenopalatine
ethmoid sinus

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18
Q

what are the main two clinical signs associated with paranasal sinus disease?

A

nasal discharge
facial swelling

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19
Q

how should primary sinusitis be treated?

A

TMPS for 7-14 days
phenylbutazone
feed from ground, dust free environment, turn out

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20
Q

what is the one cause of primary sinusitis that shouldn’t be treated with antimicrobials?

A

strangles (Streptococcus equi var equi)

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21
Q

what is the most common cause of secondary sinusitis?

A

dental disease

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22
Q

what is responsible for abduction of the arytenoids to open the glottis?

A

cricoarytenoideus dorsalis (CAD) muscle

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23
Q

what innervates the cricoarytenoideus dorsalis (CAD) muscle?

A

recurrent laryngeal nerve

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24
Q

what are the two types of dorsal displacement of the soft palate?

A

intermittent
persistent

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25
when does intermittent dorsal displacement of the soft palate occur?
during intense exercise only
26
when will the soft palate return to normal after intermittent dorsal displacement of the soft palate?
when the horse swallows
27
what are the signs seen with intermittent dorsal displacement of the soft palate?
expiratory obstruction causing gurgling/vibrating noise, possibly causing the horse to pull up
28
what disease states can often lead to persistent dorsal displacement of the soft palate?
epiglottic entrapment sub-epiglottic ulcers sub-epiglottic cysts
29
what is needed to diagnose intermittent dorsal displacement of the soft palate?
exercising endoscopy (will only occur with strenuous exercise)
30
what conservative treatments are available for intermittent dorsal displacement of the soft palate?
often resolves with age get horse fitter (stronger respiratory muscles) change tack tongue tie (prevent caudal movement)
31
what surgical techniques are available for managing dorsal displacement of the soft palate?
tie forward (best) palatoplasty
32
how is a tie forward procedure done?
sutures placed between basihyoid bone and thyroid cartilage to position the larynx more rostrally and caudally
33
how does palatoplasty work to treat dorsal displacement of the soft palate?
uses thermal/laser cautery to stiffen the soft palate (poor evidence of efficacy)
34
when would pharyngeal lymphoid hyperplasia be of clinical significance?
when associated with dorsal displacement of the soft palate
35
when is treatment of pharyngeal lymphoid hyperplasia indicated?
when associated with intermittent dorsal displacement of the soft palate treat with anti-inflammatories
36
when is dynamic pharyngeal collapse seen?
yearling (2 year olds) sports horses (neck flexion)
37
what is the typical clinical sign of a cleft palate?
milk coming from nostrils
38
which is the most common side for recurrent laryngeal neuropathy?
left (unilateral paralysis of arytenoid cartilage)
39
what is the typical signalment of recurrent laryngeal neuropathy?
large horses (genetic predisposition)
40
what is the classic clinical signs of recurrent laryngeal neuropathy?
abnormal inspiratory noise (roaring) at exercise poor performance
41
how can resting laryngeal function be graded?
1. normal 2. can fully abduct but asynchronous 3. can't fully abduct 4. complete paralysis
42
what is the treatment options for recurrent laryngeal neuropathy?
prosthetic laryngoplasty (tie-back) ventriculocordectomy (hobday) laryngeal re-innervation
43
how is tie-back (prosthetic laryngoplasy carried out for recurrent laryngeal neuropathy?
sutures placed from cricoid cartilage to muscular process of left arytenoid cartilage to permanently abduct the left arytenoid
44
what complications can be seen with prosthetic laryngoplasty (tie-back)?
coughing seroma formation infection of implant dysphagia chondritis
45
what is a hobday procedure?
ventriculocordectomy - laryngeal ventricles (saccules) removed to reduce vibration and noise associated with recurrent laryngeal neuropathy
46
what can cause unilateral laryngeal paralysis?
4th brachial arch defect laryngeal dysplasia guttural pouch mycosis previous surgery
47
what can cause bilateral laryngeal paralysis?
hepatic disease toxicity (organophosphate, lead...) post anaesthetic complication
48
what causes congenital laryngeal dysplasia?
abnormal laryngeal cartilage development due to 4th brachial arch deformity (right side effected)
49
what laryngeal dysfunctions are seen due to laryngeal dysplasia?
limited abduction of right arytenoid cartilage rostral displacement of palatopharyngeal arch
50
what is the typical clinical sign seen with vocal cord collapse?
inspiratory whistle at exercise
51
how is vocal cord collapse treated?
vocalcordectomy
52
what is the typical sign of medial deviation of the aryepiglottic folds?
thick inspiratory noise (seen in juveniles)
53
how is medial deviation of the aryepiglottic folds treated?
laser removal of aryepiglottic folds
54
what clinical signs are seen with epiglottic entrapment?
respiratory noise cough when eating poor performance
55
what horses is sub epiglottic cysts seen most commonly in?
young horses (congenital)
56
how is arytenoid chondrites treated?
antibiotics (topical and systemic) partial arytenoid resection (guarded prognosis)
57
what clinical signs are associated with guttural pouch disease?
epistaxis dysphagia (nasal discharge) dyspnoea external swelling neurological (ataxia, head tilt, facial paralysis...)
58
what is the main clinical sign of guttural pouch mycosis?
severe epistaxis nasal discharge nerve dysfunction (dysphagia, horners, laryngeal paralysis)
59
how should guttural pouch mycosis be treated?
keep horse calm and prevent further bleeding referral needed if associated with epistaxis
60
how do guttural pouch chondroids form?
chronic infection (guttural pouch empyema) results in inspissated purulent material developing
61
what is guttural pouch tympany?
air trapanned by in guttural pouch in foals up to a year old due to a congenital defect
62
what are the clinical signs of guttural pouch tympany?
marked retropharyngeal swelling respiratory stridor dysphagia