GU exam 2 Flashcards
acute glomerulonephritis
caused by infection!!
-typically Strep that is untreated
-10 days after infection s/s
-seen in young adult and children
acute glomerulonephritis
edema / fluid retention
hematuria
decrease UO
mild HTN
acute glomerulonephritis labs
urinalysis : protein , blood , WBC
CMP :
-BUN increase
-Creatinine increase
-GFR decreased
-Albumin decreased
CBC : WBC increase
acute glomerulonephritis dx
history
presentation
lab findings
acute glomerulonephritis complications
fluid overload
HTN
acute glomerulonephritis potential complication
acute or chronic kidney disease with no tx
acute glomerulonephritis tx
- abx - penicillin
- diuretics - HTN
- fluid / sodium restrict
- low protein
- steroids
- plasmapheresis
acute glomerulonephritis management
monitor vitals
diet intake
meds
acute glomerulonephritis education
disease
meds : FINISH ALL ABX!!!
dietary restriction
infection prevention
polycystic kidney disease
excessive growth of fluid filled cysts in kidneys
polycystic kidney disease s/s
HTN!!!
hematuria
flank / low back pain
HA
UTI!!!
urinary frequency!!!
urinary calculi
polycystic kidney disease labs
urinalysis : blood and bacteria
CBC : H and H decrease
CMP :
-BUN increase
-Creatinine increase
-potassium increase
-calcium decrease
-phosphorous increase
polycystic kidney disease tests
renal US : shows renal cysts
abd. CT
polycystic kidney disease complications
HTN
hematuria
polycystic kidney disease potential complications
renal calculi / UTIs
heart valve abnormality
aneurysm
liver / GI cysts
RENAL FAILURE
polycystic kidney disease tx
ACE inhibitors / ARBS
non-narcotics - acetaminophen
palliative nephrectomy
UTI abx
RENAL TRANSPLANT
polycystic kidney disease education
immediately report INFECTION
diet
medication adherence
acute kidney injury
rapid loss of renal function that is reversible when treating underlying cause
azotemia
collection of nitrogenous wast products in blood
prerenal AKI
caused by decrease blood flow to kidneys
-ex : hypovolemia!!!
intrarenal AKI
direct damage to renal tissue impairing nephron functioning
-ex : prolonged ischemia , contrast dye , aminoglycoside antibiotics
***acute glomerulonephritis
**acute tubular necrosis
postrenal AKI
caused by obstruction below kidneys
-ex : urinary tract stones , tumors , BPH
**urine is backing up
oliguric phase
urine output < 400 mL / day
-does not respond to fluid challenges or diuretics
diuretic phase
urine output increases rapidly - up to 5L / day
**dehydration common and electrolyte imbalance
AKI s/s
volume overload s/s
-edema
-SOB
-HF
-JVD
-HTN
-chest pain
anorexia / nausea
constipation / diarrhea
confusion
seizure
AKI labs
CBC : H and H decreased
CMP :
-increase potassium
-increase phosphorous
-increase phosphate
-increase BUN
-increase creatinine
-decrease calcium
-decrease sodium
ABG : metabolic ACIDOSIS
AKI complications
HYPERKALEMIA
fluid overload
hyperkalemia EKG
PEAK T WAVES
-worsening = QRS complex is widening and loss of P waves
hyperkalemia protocol
- Calcium Gluconate
- D50 (IV glucose)
- insulin
- albuterol
- bicarb
- kayexalate
AKI tx
eliminate the cause
diuretics
sodium restriction
potassium restriction
dialysis = short term
AKI management
CARDIAC MONITOR
fluid balance
positioning / deep breathing
chronic kidney disease cause
diabetes and hypertension
-IRREVERSIBLE
chronic kidney disease s/s
HTN
yellow / grey skin color
HF
arrhythmia
n/v
HA
fatigue
amenorrhea
chronic anemia
confusion
seizures
chronic kidney disease labs
CBC : H and H decrease
CMP :
-decrease sodium
-increase potassium
-increase phosphate
-decrease calcium
-decrease CO2
-increase creatinine
-increase BUN
ABG : ACIDIC
urinalysis : protein
chronic kidney disease tests
CT and renal US
chronic kidney disease complications
fluid overload
electrolyte imbalance
dysrhythmias
anemia
fragile bones
chronic kidney disease tx
- hyperkalemia protocol
- HTN
- anemia - erythropoietin IV only when active bleed or symptomatic
- dyslipidemia - STATINS
- hypocalcemia - Vit D and calcium supplement
- renal osteodystrophy - phosphate binders (administer with each meal)
renal replacement therapy
hemodialysis
peritoneal dialysis
renal transplant
CKD education
disease process
DO NOT miss dialysis
dietary restriction : protein / phosphorous / sodium / potassium
no nephrotic meds
renal transplant
improve life function NOT A CURE
renal transplant candidate
free of medical problems that increase risk
-cardiac disease
-cancer (metastatic)
-chronic infection
-alcoholism
-chemical dependency
NOT considered
post op renal transplant
- evaluate function : I / O
- immunosuppressive therapy : prevent tissue rejection , increases infection risk
- ASSESS URINE OUTPUT hourly for 48 hours
-abrupt decrease = rejection or acute tubular 4. necrosis - urine color : pink is normal , NOT FRANK RED
- monitor fluid status : NO FLUID OVERLOAD
-weigh daily