Adult Cardiac Flashcards
Acute Coronary Syndromes
umbrella term :
-stable angina
-unstable angina
-non ST elevation ( NSTEMI )
-ST elevation MI ( STEMI )
stable angina
chest pain / discomfort with physical activity , ALLEVIATED with rest and medications
-nitroglycerin and rest
unstable angina
chest pain that occurs at rest , most concerning , NOT RELIEVED with rest or medications
-precursor for MI , tx as emergency
prinzmetal’s angina
caused by coronary artery spasm , normally occurs at night
-type of unstable angina
ACS dx test
- 12 lead EKG
- Cardiac Biomarkers
- Lipid profile
- Exercise Stress test
- Stress Echo
- coronary angiography
Cardiac Biomarkers (cardiac enzymes)
creatinine kinase
troponin I or T
myoglobin
levels rise after myocardial injury , measured every 6 hours after admission
lipid profile
triglycerides
total cholesterol
LDL
HDL
stress echo
those unable to use treadmill or bike , DOBUTAMINE can be used to increase HR mimicking effect of exercise
coronary angiography
GOLD STANDARD for ACS
-L sided heart cath
ACS medications
- OXYGEN
- Nitroglycerin
- Beta blockers
- CCB
- Antiplatelet drugs
- Morphine
- Antithrombin agents
- ACE inhibitors
ACS oxygen
ACUTE pain from angina is r/t decreased oxygen supply!!!
ACS nitroglycerin
causes vasodilation , given topically / sublingual / transdermal / IV
ACS beta blockers
used to tx angina , MI , dysrhythmias , HF , hypertension
-oral or IM
Beta blocker examples
Metoprolol
Labetalol
Carvedilol
ACS CCB
inhibits calcium ion flow across cellular membrane
Verapamil and Cardizem
calcium channel blockers
ACS antiplatelets
inhibits clotting or prevents platelet aggregation , used with UNSTABLE angina
Plavix and Aspirin
Antiplatelet agents
ACS antithrombins
inhibits antithrombin III , prevents fibrinogen to fibrin conversion
-IM or Sub q
Lovenox and Heparin
antithrombin agents
ACS morphine
used for pain relief and anxiety
-given IV
ACS ACE inhibitors
prevent angiotensin I to angiotensin II conversion
tx HTN and HF afteer MI
Enalapril / Captopril / Lisinopril
ACE inhibitors
myocardial infarction
leading cause of death among men and women in US
-increased risk b/t 6:00am and 12:00pm
MI modifiable risk factors
smoking
high LDLs
type II DM
obesity
HTN
MI non-modifiable risk factors
male gender
postmenopausal female
family hx
why are MI more common in the morning
blood pressure is at it’s highest and blood is most viscous
NSTEMI MI
partial occlusion of major coronary vessel
ST depression or T wave inversion without Q wave
will have elevated cardiac markers
STEMI MI
complete occlusion of major coronary vessel resulting in irreversible full thickness heart muscle damage
ST elevations
elevated cardiac markers
complete occlusion MI s/s
chest pain
shoulder / arm pain ( LEFT )
jaw / tooth pain
upper back pain
SOB
n/v
sweating
fatigue
R coronary MI s/s
JVD
hypotension
bradycardia : damaged SA node
n/v
L coronary artery MI ( WIDOW MAKER ) s/s
worst prognosis w / high risk of sudden death
dyspnea
tachycardia
hypertension
Women MI s/s
fatigue
diaphoresis
indigestion
arm / shoulder pain
nausea
vomiting
geriatric MI s/s
dyspnea
confusion
weakness
syncope
MI tests
- CK
- Troponin I and Troponin T
- Myoglobin
- Complete Metabolic profile
- CBC
- Coagulation Studies
- ABG
- EKG : GOLD STANDARD
- echo
- L sided heart cath
CK lab with MI
rise 6 hours post MI
peak at 18 hours
baseline 10-14 days after
Troponin MI test
preferred blood test to dx an MI , proteins are almost exclusively in heart
increase 6 hours post MI
peak 10-24 hours
baseline 10-14 days
EKG MI
GOLD STANDARD
-reveal ST depression or elevation
***WOMEN ARE LESS likely than men to have typical EKG change
echo MI
used to evaluate ventricular function like ejection fraction
MI L sided heart cath
GOLD STANDARD FOR FLOW DX
-inserted through radial or femoral
MI medications
- Oxygen
- MONA
- Heparin
- Beta Blockers
MONA
Morphine
Oxygen
Nitroglycerin
Aspirin
MI nitroglycerin
dilates coronary arteries
one tablet administered every 5 minutes w/ maximum of 3 doses
MI aspirin
help prevent platelets from enlarging or forming
MI morphine
control pain, relax coronary arteries, improve blood flow
MI heparin
started to prevent new clot formation
MI beta blockers
decrease heart workload
DO NOT USE WITH R CORONARY ARTERY MI w/ bradycardia
MI reperfusion therapy
- Fibrinolytic therapy
- percutaneous coronary intervention
MI Fibrinolytic Therapy
dissolve clot in coronary artery , symptoms MUST be present for less than 12 hours
NOT tx for unstable angina or NSEMI
3 IV sites
fibrinolytic therapy complications
excessive bleeding and hemorrhagic stroke
MI percutaneous coronary intervention
preferred method for opening blocked vessels causing and MI
-catheter with small balloon is inserted , inflated and deflated to open blocked artery
-stent may be placed
-radial or femoral insertion site
MI surgery
CABG
CABG
revascularization bypassing blockages
-typically uses saphenous vein or internal thoracic artery is used
CABG complications
bleeding
dysrhythmias
MI
stroke
nonunion of sternum
sternal infection
renal failure
HF
CABG : bypass complication
induction of systemic inflammatory response = shock
heparin induced thrombocytopenia
activation of platelets
complications of cross clamping aorta
post CABG care
monitor HR and BP continuously
hemodynamic monitor
cardiac monitor
s/s of infection
assess heart tones
core temperature
hourly I / O
skin color , pulses , edema
chest tube output / color / volume
hgb / hct / electrolytes / creatinine / BUN
post MI teaching
cardiac rehab
low cholesterol and sodium diet
avoid stress
report s/s of MI
angina
purpose / dose / SE of meds
nitroglycerin edu
no smoking
weight
cardiomyopathy
heart muscle becomes weak, enlarged, thickened and develop structural changes
cardiomyopathy risk factors
diabetes
hypertension
high cholesterol
high fat
obesity
family hx heart disease
sedentary lifestyle
smoking
alcohol
cocaine
viral infection
lyme disease
nutritional deficit
pregnancy
respiratory failure
ischemic cardiomyopathy
reduced ejection fraction caused by CAD such as MI or loss of myocardium
nonischemic cardiomyopathy
- dilated
- hypertonic
- restrictive
dilated cardiomyopathy
most common cause of HF , dilation of muscle in L ventricle causing enlargement and poor CO
hypertrophic cardiomyopathy
excessive myocardial hypertrophy , heart muscle thickens and enlarges , contraction is not weakened but filling is impaired
restrictive cardiomyopathy
stiff ventricular muscle resulting in impaired filling , results from another disease
cardiomyopathy angina s/s
chest pain
dizziness
indigestion
n/v
sweating
palpitations
SOB
fatigue
cardiomyopathy HF s/s
rapid / irregular HR
SOB
edema of legs , feet , abd.
pulmonary congestion
abnormal heart sounds
JVD
enlarged liver
fatigue
loss of appetite
cough
cardiomyopathy labs
- BNP
- CMP
- thyroid function
- CBC
- iron level
- CXR
- EKG
- R heart cath
- doppler echo
CMP lab
evaluates liver / renal function
Heart cath ejection fraction
55 - 65 % = normal
< 45 % = some disease
< 30 % = severe disease
cardiomyopathy medications
- ACE inhibitors
- ARBS
- Beta blocker
- Digoxin
- CCB
- diuretics
- nitrates