GI exam 2 Flashcards
acute pancreatitis
inflammation of pancreas
-can be mild / severe
mild acute pancreatitis
self limiting, no end organ dysfunction
-most fully recover
severe acute pancreatitis
pts may develops SIRS and end-organ dysfunction
acute pancreatitis risks
alcohol and gallstones
other acute pancreatitis risks
I GET SMASHED
I GET SMASHED
idiopathic
gallstones
ethanol
trauma
steroid use
mumps
autoimmune
scorpion stings
hypercalcemia / hypertriglyceridemia
ECRP
drugs / meds
acute pancreatitis s/s
sudden onset severe EPIGASTRIC pain!!
-radiates to flank / back / shoulder
-sharp, deep pain
n/v - bloody??
abd distention
hypotension and shock
tetany (hypocalcemia)
-trousseau’s sign
-chvostek’s sign
chvostek’s sign
twitch of facial muscles when touching someones cheek
trousseau’s sign
involuntary contraction of wrist muscle when BP cuff is inflated
severe hemorrhagic pancreatitis
from eroding blood vessels
-cullen’s sign
-grey-turner’s sign
cullen’s sign
bluish discoloration around umbilicus
grey turner’s sign
bluish discoloration of flanks
-must turn pt to see this!!
acute pancreatitis labs
CBC : wbc elevation
CMP : ast / alt elevation , direct bilirubin elevated, calcium decreased, albumin decreased
Lipase : elevated
acute pancreatitis dx test
abdominal CT with contrast!!!
abd US
EKG
CXR
acute pancreatitis US
look at gallbladder for dilated common bile duct
acute pancreatitis CT
confirming / viewing possible calcification in duct
acute pancreatitis dx
- acute onset on persistent , severe epigastric pain
- elevated lipase / amylase
- findings on dx imaging
** MUST have 2 OF 3 in order to have dx
ranson’s criteria
score > 3 indicates severe pancreatitis
Ransons admission
> 55 years old
WBC > 16,000
LDH > 350
AST > 250
glucose > 200
**KNOW THIS
Ransons at 48 hours
hematocrit decrease > 10%
BUN increase > 5
Calcium < 8
PaO2 < 60
base deficit > 4
fluid sequestration > 6
**KNOW THIS
acute pancreatitis management
fluid replacement : IV crystalloids
electrolyte replacement : hypocalcemia, hypomagnesmia, hypokalemia
acute pancreatitis fluids
IV crystalloids at 5-10 ml / kg / hr
acute pancreatitis I / O
urinary output < 50 mL / hr is early sign of HYPOVOLEMIA
acute pancreatitis BP
systolic BP > 100
MAP > 60
HR < 100
hypocalcemia s/s
widened QT interval
tetany , chvostek , trousseau
seizure PRECAUTIONS
what lab to monitor with hypocalcemia…
albumin levels
if potassium is needed for hypokalemia…
ALWAYS HANG POTASSIUM ON A PUMP
-no more than 20 mL / hr
-potassium burns going in
acute pancreatitis patients must….
remain NPO for 24 hours!!!
mild pancreatitis diet
oral feeding IF no vomiting / decreased pain / inflammatory markers improving
severe pancreatitis diet
enteral nutrition if unable to tolerate oral diet by 5 days!!!
acute pancreatitis comfort
PAIN MANAGEMENT is #1
-pain increase pts metabolism , IV opioids , PCA pump
antibiotics NOT recommended
acute pancreatitis pulmonary complications
ARDS
atelectasis , pneumonia , pleural effusion
hypoxemia
acute pancreatitis cardiac complications
cardiac dysrhythmias
hypovolemic shock
myocardia depression
acute pancreatitis GI complications
GI bleeding
pancreatic abscess
pancreatic pseudocyst
acute pancreatitis hematologic complication
DIC
coagulation abnormalities
acute pancreatitis renal complications
acute renal failure
elevated BUN
oliguria - low urine output
acute pancreatitis education
diet : small frequent meals, eats carbs , AVOID FAT AND PROTEIN
no alcohol
no smoking
acute pancreatitis improvement
lipase decreases and pain decreases
acute pancreatitis might have this in place if caused by alcohol….
seizure precautions
chronic pancreatitis
persistent inflammation , not reversible
chronic pancreatitis risks
heavy alcohol use
recurrent / severe acute pancreatitis!!!!
smoking
genetics
chronic pancreatitis s/s
-upper abd pain radiating to back : gets WORSE with eating / drinking (esp. alcohol)
-n/v
-weight loss
-pale / clay colored stool!!!
-steatorrhea (greasy , bad smelling stool)
chronic pancreatitis labs
CBC
CMP :
-alkaline phosphate : increase
-bilirubin : increase
-glucose : increase
Lipase and amylase increased
chronic pancreatitis dx test
abd CT
abd US
ERCP
*visualize pancreas and verify structural changes
chronic pancreatitis diagnosis
- chronic abd pain , recurring acute pancreatitis
- diarrhea, steatorrhea, weight loss
- pancreatogenic diabetes
- visual damage on imaging!!!
chronic pancreatitis pain control
opioids normally required
chronic pancreatitis meds
IV fluids
electrolyte replacement
histamine blocker
PPI
pancreatic enzyme replacement therapy (PERT)
provides amylase and protease
tx malnutrition and malabsorption
taken everytime pt eats
**Creon
chronic pancreatitis education
NO alcohol
avoid tobacco
pancreatic enzyme meds
limit fat
avoid irritating foods / drinks
-coffee, caffeine
pancreatic enzyme education
take with food and FULL glass of water
DO NOT chew tablets
chronic pancreatitis CT
will have MULTIPLE pancreatic calcifications
pancreatic cancer
diagnosis often occurs late in disease process
-very rapid growing
pancreatic cancer risks
smokers
diet high in fate
high meat consumption, fried food, refined sugar, nitrates
diabetes, chronic pancreatitis
family hx
> 60 years
pancreatic cancer s/s
VAGUE symptoms : present like all GI disorders
pain : dull and epigastric area
jaundice : bile duct obstruction
fatigue
weight loss