GI exam #2 Flashcards
What is the most common GI problem?
N&V
What are the 3 types of N&V
- pathogenic- R/T disease process
- iatrogenic- R/T disease Tx
- psychogenic- R/T psychological state (stress)
Patho of vomiting:
stimulation of chemo-receptor trigger zone (CTZ) which then stimulates the vomiting center (VC) in the medulla
Objective data of emesis:
color, consistency and amount
Tx for N&V:
NPO until able to tolerate oral intake
HOB elevated to prevent aspiration
replace fluid and electrolytes
Acid-base balance
vomiting leads to _______?
diarrhea leads to _______?
vomiting = alkalosis diarrhea = acidosis
What is gastritis?
Inflammation of the gastric mucosa
What causes gastritis?
long-term NSAID use
H. Pylori or other bacteria
Chronic gastritis is closely associated with development of _______.
Gastric CA
Chronic Gastritis
What is chronic duodenal reflux?
When the pyloric sphincter allows alkaline juices to leak into stomach
Chronic Gastritis
Introduction of alkaline juices into stomach destroy which 2 kinds of cells in the stomach?
Parietal and chief cells
Chronic Gastritis
What will develop if there is no intrinsic factor produced?
pernicious anemia b/c B12 cannot be absorbed
Chronic Gastritis
Why do antacids not relieve pain associated with chronic gastritis?
b/c problem is alkaline not acidotic.
melena?
dark and tarry stool
hematochezia?
bright red blood in stool
What are the 2 origins of upper GI bleeding?
arterial and venous
which type of upper GI bleeding is more severe?
arterial
Hematoemesis?
bloody vomit
What kind of bleeding does melena indicate?
slow, oozing bleed in upper GI tract
What kind of bleeding does hematochezia indicate?
bleeding in lower GI tract
What is the most important intervention for upper GI bleed?
maintain airway
What is a mallory-weiss tear?
tear in esophagus caused by severe, violent vomiting
Peptic Ulcer Disease
Are gastric secretions increased, normal or decreased with gastric ulcer?
normal to decreased
Peptic Ulcer Disease
Are gastric secretions increased, normal or decreased with duodenal ulcer?
increased
what is Peptic Ulcer Disease?
erosion of GI mucosa from digestive action of HCl & Pepsin
Peptic Ulcer Disease
Acute vs. Chronic
Acute- superficial erosion
Chronic- long duration, erosion though muscular layer
1 cause of Peptic Ulcers:
H. Pylori
Peptic Ulcer Disease
S/Sx:
Burning, gnawing pain that is worse on empty stomach. Relieved by food, but recurs when stomach empties again.
Peptic Ulcer Disease
Tx:
relieve pain, avoid stressors
physical & emotional REST
Peptic Ulcer Disease
Drug Therapy:
Antacids H2 receptor agonists Proton pump inhibitors (PPI's) anticholenergics reglan (increase gastric motility) sucralfate (coats ulcer, protects from erosion)
Peptic Ulcer Disease
action of antacids & when to admin?
antacids neutralize gastric acid. give 1-3 hours after meals & at bedtime
Peptic Ulcer Disease
action of H2 blockers?
reduce HCl acid secretions (Tagamet, Zantac, Pepcid)
Peptic Ulcer Disease
action of PPI’s?
stop secretion of HCl acid to raise pH of stomach (Protonix, Prilosec, Prevacid)
Peptic Ulcer Disease
action of Anticholinergics?
block SNS- so they slow everything down. inhibit gastric secretions, decrease gastric motility. high incidence of s/e: dry mouth, urinary retention
What is the most common complication of Peptic Ulcer Disease?
hemorrhage.
What is the most serious complication of Peptic Ulcer Disease?
perforation (high mortality)
Peptic Ulcer Disease Complications:
What happens when perforation occurs?
gastroduodenal contents spill into peritoneal cavity= peritonitis & septicemia
Peptic Ulcer Disease Complications:
S/sx of perforation?
sudden onset sever upper abd pain, rigid, boardlike abd, absent bowel sounds, rapid & shallow resp
Peptic Ulcer Disease Complications:
What is gastric outlet obstruction?
occurs when inflammation & edema around ulcer (primarily duodenal) result in narrowing of pyloric sphincter = food can’t escape stomach
Peptic Ulcer Disease
S/sx of gastric outlet obstruction:
long Hx of pain, belching, vomiting of undigested food, weight loss
Peptic Ulcer Disease
Tx of gastric outlet obstruction:
gastric decompression by large lumen NG tube, then surgery.
Gastric Surgery
gastrectomy?
partial removal of stomach (70-80%), anastomosis to either duodenum (Bilroth I) or jejunum (Bilroth II)
Gastric Surgery
Vagotomy?
severe vagus nerve to decrease gastric secretions
Gastric Surgery
Pyloroplasty?
enlargement of the pyloric sphincter
Gastric Surgery
Post-op drainage will be bright ___ for first __ hours. Then return to normal _____ color in __ hours.
bright red 1st 12 hours. yellow-green after 36 hours.
Gastric Surgery Complications
What is dumping syndrome?
rapid emptying of gastric contents into sm intestines.
Gastric Surgery Complications
dumping syndrome S/sx:
weakness, syncope, dizziness b/c of increased blood flow to GI tract to facilitate digestion