General Inhalational Anesthesia Flashcards

1
Q

These changes occur during GA due to the stimulant or depressant effects of the agents used.

A

CV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Reason why ether is not used as an inhalational agent anymore.

A

Explosive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Stage of anesthesia when a patient vomits, goes to laryngospam, etc.

A

II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Stage of excitement/delirium in anesthesia.

A

II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Stage of analgesia

A

I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

From induction to loss of consciousness.

A

I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

From loss of consciousness to onset of automatic breathing.

A

II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q
  • Irregular respiration
  • Breath-holding
  • Vomiting
  • Struggling
A

II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Should keep the pt in this stage anesthesia in your office.

A

I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

A good stage I anesthetic.

A

Nitrous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Pt is hyperreflexing in this stage of anesthesia.

A

II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Stage of surgical anesthesia.

A

III

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

From onset of automatic breathing to respiratory paralysis.

A

III

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

From respiratory paralysis to death.

A

IV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

This can be used if the pt is allergic to LA.

A

General anesthesia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Contraindication to office sedation.

A

Obstructive sleep apnea.

Basically any type of compromised airway.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

This facilitates complete control of the airway, breathing, and circulation.

A

GA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Can be administered without moving the patient from a supine position.

A

GA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Dangerous reaction to GA

A

Malignant hyperthermia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Changes in cognition and psychomotor performance are altered for how many after GA?

A

24-36 hours.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

During GA, this INCREASES bc there’s a decreased tidal volume.

A

Respiratory Rate

22
Q

Respiratory effects of GA.

A
  • Increased arterial CO2
  • Depressed response to increases in arterial CO2.
  • Depressed response to decreases in arterial O2.
  • Decreased tidal volume.
  • Increased respiratory rate.
23
Q

A decrease in this type of resistance occurs with GA.

24
Q

You produce apnea with greater depths of anesthesia.

25
You reduce function of this with GA.
tracheal ciliary function
26
This cardiovascular thing increases in GA.
Heart rate, but net decreased arterial BP.
27
CV effects of GA.
- Increased HR - Decreased arterial BP - Decreased sympathetic tone (decreased peripheral resistance). - Decreased CO. - Preservation of coronary blood flow.
28
Types of Catecholamines.
Epi NE Dopamine
29
Volatile General Anesthetic that sensitizes the myocardium to catecholamines.
Halothane
30
This occurs in up to 60% of patients on general anesthesia.
Cardiac dysrrhythmias
31
GA increases blood flow to these organs.
Brain Muscle Skin
32
Decreased levels of this when using GA.
Insulin | Testosterone
33
This anesthetic has little to no effect on Insulin levels.
Sevoflurane
34
Potential toxicities with GA.
Renal toxicitiy
35
Anesthetic that has the greatest risk of hepatic necrosis.
Halothane
36
Most common hepatotoxic drug in the US.
Alcohol
37
An inherited disorder of muscle metabolism where Calcium remains bound to myosin, causing muscle rigidity. Energy is generated as heat and these patients' temperatures skyrocket.
Malignant Hyperthermia
38
This is released when inhalational drugs are metabolized, causing damage to the kidneys.
Fluorine
39
All halogenated hydrocarbons used for anesthesia have these attached.
Fluorine or Bromine
40
Determines if a patient is breathing properly.
Capnography.
41
Rapid induction bc it has low blood and tissue solubility.
Desflurane
42
Agent that has less depression of diaphragmatic function than other agents.
Sevoflurane
43
What is emergence.
Waking the patient up from general anesthesia.
44
Muscle relaxants prevent these from shutting.
Vocal cords
45
Induction of GA combines these two drugs.
Sedative + Muscle relaxant.
46
IV induction involves this.
Pre-oxygenation Done before intubation
47
You should not be sedating your patient if you can't do this.
Intubate them.
48
These should be at the same level when intubating.
Ear and Sternal notch
49
Function of the laryngeal mask.
Blocks the esophagus if you're scared on endotracheal intubation.
50
This occurs when you take the tube out at Stage 2.
Laryngospasm (Hyperreflex).