General Inhalational Anesthesia Flashcards

1
Q

These changes occur during GA due to the stimulant or depressant effects of the agents used.

A

CV

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2
Q

Reason why ether is not used as an inhalational agent anymore.

A

Explosive

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3
Q

Stage of anesthesia when a patient vomits, goes to laryngospam, etc.

A

II

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4
Q

Stage of excitement/delirium in anesthesia.

A

II

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5
Q

Stage of analgesia

A

I

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6
Q

From induction to loss of consciousness.

A

I

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7
Q

From loss of consciousness to onset of automatic breathing.

A

II

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8
Q
  • Irregular respiration
  • Breath-holding
  • Vomiting
  • Struggling
A

II

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9
Q

Should keep the pt in this stage anesthesia in your office.

A

I

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10
Q

A good stage I anesthetic.

A

Nitrous

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11
Q

Pt is hyperreflexing in this stage of anesthesia.

A

II

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12
Q

Stage of surgical anesthesia.

A

III

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13
Q

From onset of automatic breathing to respiratory paralysis.

A

III

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14
Q

From respiratory paralysis to death.

A

IV

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15
Q

This can be used if the pt is allergic to LA.

A

General anesthesia

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16
Q

Contraindication to office sedation.

A

Obstructive sleep apnea.

Basically any type of compromised airway.

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17
Q

This facilitates complete control of the airway, breathing, and circulation.

A

GA

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18
Q

Can be administered without moving the patient from a supine position.

A

GA

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19
Q

Dangerous reaction to GA

A

Malignant hyperthermia

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20
Q

Changes in cognition and psychomotor performance are altered for how many after GA?

A

24-36 hours.

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21
Q

During GA, this INCREASES bc there’s a decreased tidal volume.

A

Respiratory Rate

22
Q

Respiratory effects of GA.

A
  • Increased arterial CO2
  • Depressed response to increases in arterial CO2.
  • Depressed response to decreases in arterial O2.
  • Decreased tidal volume.
  • Increased respiratory rate.
23
Q

A decrease in this type of resistance occurs with GA.

A

airway

24
Q

You produce apnea with greater depths of anesthesia.

A

True

25
Q

You reduce function of this with GA.

A

tracheal ciliary function

26
Q

This cardiovascular thing increases in GA.

A

Heart rate, but net decreased arterial BP.

27
Q

CV effects of GA.

A
  • Increased HR
  • Decreased arterial BP
  • Decreased sympathetic tone (decreased peripheral resistance).
  • Decreased CO.
  • Preservation of coronary blood flow.
28
Q

Types of Catecholamines.

A

Epi
NE
Dopamine

29
Q

Volatile General Anesthetic that sensitizes the myocardium to catecholamines.

A

Halothane

30
Q

This occurs in up to 60% of patients on general anesthesia.

A

Cardiac dysrrhythmias

31
Q

GA increases blood flow to these organs.

A

Brain
Muscle
Skin

32
Q

Decreased levels of this when using GA.

A

Insulin

Testosterone

33
Q

This anesthetic has little to no effect on Insulin levels.

A

Sevoflurane

34
Q

Potential toxicities with GA.

A

Renal toxicitiy

35
Q

Anesthetic that has the greatest risk of hepatic necrosis.

A

Halothane

36
Q

Most common hepatotoxic drug in the US.

A

Alcohol

37
Q

An inherited disorder of muscle metabolism where Calcium remains bound to myosin, causing muscle rigidity.

Energy is generated as heat and these patients’ temperatures skyrocket.

A

Malignant Hyperthermia

38
Q

This is released when inhalational drugs are metabolized, causing damage to the kidneys.

A

Fluorine

39
Q

All halogenated hydrocarbons used for anesthesia have these attached.

A

Fluorine or Bromine

40
Q

Determines if a patient is breathing properly.

A

Capnography.

41
Q

Rapid induction bc it has low blood and tissue solubility.

A

Desflurane

42
Q

Agent that has less depression of diaphragmatic function than other agents.

A

Sevoflurane

43
Q

What is emergence.

A

Waking the patient up from general anesthesia.

44
Q

Muscle relaxants prevent these from shutting.

A

Vocal cords

45
Q

Induction of GA combines these two drugs.

A

Sedative + Muscle relaxant.

46
Q

IV induction involves this.

A

Pre-oxygenation

Done before intubation

47
Q

You should not be sedating your patient if you can’t do this.

A

Intubate them.

48
Q

These should be at the same level when intubating.

A

Ear and Sternal notch

49
Q

Function of the laryngeal mask.

A

Blocks the esophagus if you’re scared on endotracheal intubation.

50
Q

This occurs when you take the tube out at Stage 2.

A

Laryngospasm (Hyperreflex).