Gamesmanship Flashcards

1
Q

Mammo:

The calcifications don’t change configuration on CC and MLO views. This is the so called “tattoo sign “ for DERMAL calcifications. Next step: tangential view to prove it.

A

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2
Q

Mammo:

Remember that secretory calcifications occur after menopause. Don’t call them secretory in a premenopausal patient (no matter how much they look like them).

A

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3
Q

Mammo:

If they show you a ML view for calcifications. Think hard about milk of calcium - is it tea cupping?

A

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4
Q

Mammo:

If a test writer wants you to say DCIS, they can prompt it 3 ways:

1) suspicious calcifications (fine linear branching or fine pleomorphic),
2) non mass like enhancement on MRI,
3) multiple intraductal masses on galactography.

A

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5
Q

Mammo:

Skin thickening and trabecular thickening should get progressively better with time. It should start out worst, then better, then better. If it gets worse—this recurrent disease.

A

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6
Q

Mammo:

Gynecomastia looks like a cancer on ultrasound. This is why a male breast cancer workup (palpable finding) always begins with a mammogram.

A

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7
Q

Nukes:

Distinguishing
Tc-99 DTPA vs Xe-133.

DTPA can be done in multiple projections. DTPA tends to clump in the central airways.

A

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8
Q

IR:

Sneaky things related to TIPS.

1) CO2 run during hepatic vein wedge—blowing liver dome off bc injection pressure too strong. Anytime see CO2 run over liver, think of this.
2) TIPS placed into hepatic artery (not portal vein). Pay attention to anatomy!
3) Could say portal systemic gradient was normal (3-6). Remember—TIPS treats portal hypertension. Don’t do TIPS on someone without portal HTN!

A

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9
Q

IR:

Self Expandable: anywhere you might get external compression.
Balloon Expandable: if you need more precise placement

May Thurner Syndrome—Self
SFA—Self
Focal Atherosclerosis Stenosis in distal aorta—balloon
Renal Ostium Stenosis—balloon

A

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10
Q

Vascular:

Thoracic Angiogram: If you see an angiogram through the great vessels and aorta, think about TOS, Takayasu, and Giant Cell. The locations are classic, and helpful.
Takayasu—young (probably Asian female),
Giant Cell-old person.
AGE TRUMPS LOCATION!
If they show you TOS, they’ll show arms up and down—dead give away.
If trauma, don’t forget to check great vessels (not just aorta)

A

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11
Q

Vascular:

Aortic Dissection on Angiogram:
Can show as opacification of abdominal aortic branch vessels during aortography (cath in aortic true lumen) w/branch vessels (celiac axis, sup. mesenteric a., and renal arteries) arising out of nowhere. They appear to be floating, w/little or no antegrade opacification of aortic true lumen.

“Floating Viscera Sign”

A

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12
Q

Vascular:

Collateral Filling:
If you inject SMA and the celiac branches fill—infers a tight stenosis at celiac origin.

If you inject celiac and the SMA branches fill—infers tight stenosis at the SMA origin.

A

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13
Q

Vascular:

Hand Angiograms:
Pathology: it’s going to be either Buergers or Hypothenar Hammer Syndrome (HHS).

Ask yourself: is ulnar artery involved?
If yes, go with HHS.
If the ulnar nerve (typo?) looks ok, but the fingers are out, go w/Buergers.
Careful: fingers can be out w/HHS too (distal emboli).
Pseudo-aneurysm off ulnar artery: slam dunk HHS.

A

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14
Q

Vascular:
Renal Artery Angiogram:

Ostial narrowing—think atherosclerosis; treat with balloon + stent

Beading mid vessel—think FMD; treat with balloon only

A

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15
Q

Vascular:

Kidney Angiogram

First question should always be
“Is there an RCC or AML?”

Second question should be
“Is there PAN/Speed kidney/A bunch of little aneurysms?”

A

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16
Q

Vascular:

Kawasaki:
Two classic ways to show this:

1) CT with a coronary artery aneurysm (obvious one),
2) calcified coronary artery aneurysm shown on CXR (old oral boards favorite)

A

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17
Q

Cardiac:

Signs of left atrial enlargement:

1) Double density—superimposed over contour of the right heart
2) Splaying of the carina—Angle over 90*
3) Posterior placement of the heart—seen on lateral CXR

A

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18
Q

Upper or Lower Lobe Predominant?

Most inhaled stuff (not asbestosis!)—coal workers, silicosis. Includes progressive massive fibrosis.

A

Upper

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19
Q

Upper or Lower Lobe Predominant?

CF

A

Upper

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20
Q

Upper or Lower Lobe Predominant?

RB-ILD

A

Upper

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21
Q

Upper or Lower Lobe Predominant?

Centrilobular Emphysema

A

Upper

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22
Q

Upper or Lower Lobe Predominant?

Ankylosing Spondylitis

A

Upper

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23
Q

Upper or Lower Lobe Predominant?

Asbestosis

A

Lower

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24
Q

Upper or Lower Lobe Predominant?

Primary Ciliary Dyskinesia

A

Lower

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25
Upper or Lower Lobe Predominant? Most Interstitial lung diseases (UIP, NSIP, DIP)
Lower
26
Upper or Lower Lobe Predominant? Panlobular Emphysema (Alpha 1)
Lower
27
Upper or Lower Lobe Predominant? Rheumatoid Lung
Lower
28
Upper or Lower Lobe Predominant? Scleroderma
Lower
29
Obtuse margin with lung. Is it mediastinal or pulmonary origin?
Mediastinal
30
Acute margin with lung. Is it mediastinal or pulmonary origin?
Pulmonary
31
Ground glass nodule on PET: HOT GGO = _______
Infection
32
Ground glass nodule on PET: COLD GGO = _______
Cancer (BAC)
33
Collapse— Always be on the lookout for collapse. Anytime see anything that could be, at least entertain idea. -post intubation -placement of central line -ICU patient w/no other details (mucous plugging) -outpatient w/no other history (cancer)
.
34
Pulmonary edema— After chest tube placement: re-expansion edema After crack or heroin: drug induced edema After head injury: neurogenic edema After lung transplant: reperfusion edema related to ischemia/reperfusion (peak day 4)
.
35
Collagen vascular tricks: RA in shoulders on frontal CXR
Lower lobe UIP pattern
36
Collagen vascular tricks: Ankylosing Spondylitis on lateral CXR
Upper lobe fibrobullous disease
37
Collagen vascular tricks: Dilated esophagus on CT
Scleroderma with NSIP lungs
38
“Frozen hemithorax”—lack of contralateral mediastinal shift in association with massive pleural effusion . Diagnosis?
Mesothelioma. It’s due to encasement of the lung (and fissures) by cancer
39
Infections in AIDS by CD4: >200
Bacterial infections, | TB
40
Infections in AIDS by CD4: <200
PCP, | Atypical Mycobacterial
41
Infections in AIDS by CD4: <100
CMV, Disseminated Fungal, Mycobacterial
42
AIDS: Lung cysts = (diagnosis?)
LIP LIP is AIDS defining in a pediatric patient
43
AIDS: Lung cysts + ground glass + pneumothorax = (diagnosis?)
PCP
44
AIDS: hypervascular nodes = (diagnosis?)
Castleman’s or Kaposi
45
AIDS: Most common airspace opacity
Strep pneumonia
46
AIDS: CT with ground glass = (diagnosis?)
PCP
47
AIDS: Flame-shaped perihilar opacity = (diagnosis?)
Kaposi sarcoma
48
AIDS: Persistent opacities= (diagnosis?)
Lymphoma
49
If they show you a varicocele, regardless of side (right being more suspicious than left), if it’s a next step question, look for the ____
Abdominal cancer
50
If “hyperemesis” is in question stem (repro), think ______
Things that give you elevated B-hCG—like moles and multiple pregnancy (twins)
51
If it’s a GYN case and has “history of abdominal surgery”, keep in mind _______
Peritoneal inclusion cysts (from adhesions)
52
Met to vagina in anterior wall upper 1/3 is “always” (90%) from _______
Upper genital tract
53
Met to vagina in posterior wall lower 1/3 is “always” (90%) from _______
GI tract
54
Fluid in endometrial canal in post menopausal woman should make you think ______
Cervix is obstructed (cancer, or more commonly stenosis)
55
Repro: combo of ovarian mass and thickened endometrium should make you think _____
Granulosa Cell Tumor
56
IUGR: Symmetrical -baby or placenta issue?
Baby problem (placenta is fine)
57
IUGR: Symmetrical -head spared or not?
head NOT spared
58
IUGR: Symmetrical -when first apparent?
Early, including first trimester
59
IUGR: Symmetrical -causes?
TORCHS, Fetal EtOH, Chromosomal abnormalities
60
IUGR: Asymmetrical -baby or placenta issue?
Placenta problem
61
IUGR: Asymmetrical -head spared or not?
Head spared
62
IUGR: Asymmetrical -when first apparent?
Normal until third trimester
63
IUGR: Asymmetrical -causes?
Maternal hypertension, Severe malnutrition, Ehler-Danlos
64
prostate CA v BPH: ______ is usually in the *peripheral* zone. When it is in central zone, T2 is “smudgy” or charcoal.
Prostate CA
65
prostate CA v BPH: _______ is usually in central zone. Have a sharp border. Can “draw a line around them with a pencil”
BPH nodules
66
Gartner duct cyst v Bartholin cyst ______ is above pubic symphysis. _______ is below.
GARTNER DUCT CYST is above pubic symphysis. BARTHOLIN is below it.
67
Bicornuate v septate uterus Distinguish the two by:
Apex of the fundal contour
68
Bicornuate v septate uterus Apex of fundus >5mm above tubal Ostia
Septate
69
Bicornuate v septate uterus Apex of fundus <5mm above tubal Ostia
Bicornuate
70
Bicornuate v septate uterus Which has established increased 1st trimester loss?
Septate | Bicornuates have a lot less problems. Maybe no increased risk, depending who you ask
71
IVPs (GU section): Haven’t been used since 1970s, but few tricks. If have *medial* deviation of the ureters, think______
Retroperitoneal fibrosis
72
IVPs (GU section): Haven’t been used since 1970s, but few tricks. If have *lateral* deviation of the ureters, think______
Psoas hypertrophy, or lymph nodes
73
RCC v oncocytoma: ___ is typically colder than surrounding renal parenchyma on PET
RCC
74
RCC v oncocytoma: ___ is typically hotter than surrounding renal parenchyma on PET
Oncocytoma
75
Oncocytoma: if they wanted to ask it, can be shown 3 ways: - CT solid mass with central scar - ultrasound “spokewheel” vascular pattern - PET CT it will be hotter than surrounding renal cortex
.
76
Renal cysts syndromes: Cysts in liver, kidneys are BIG Diagnosis?
ADPKD
77
Renal cysts syndromes: Cysts in pancreas Diagnosis?
VHL
78
Renal cysts syndromes: kidneys are small Diagnosis?
Acquired (uremic)
79
Renal cancer syndromes: Subtype: Clear Cell Syndrome/Association?
Von Hippel-Lindau
80
Renal cancer syndromes: Subtype: papillary Syndrome/Association?
Hereditary papillary renal carcinoma
81
Renal cancer syndromes: Subtype: chromophobe Syndrome/Association?
Birt Hogg Dube
82
Renal cancer syndromes: Subtype: Medullary Syndrome/Association?
Sickle cell trait
83
If you’re shown a unilateral renal agenesis case, remember association with _______ in men, and _______ in women
Men: absent vas deferens, and ipsilateral seminal vesicle Cyst Women: mullarian anomalies (unicornuate uterus)
84
Showing persistent nephrograms —either by plain film or CT is the classic trick for ATN—usually contrast induced nephropathy.
.
85
CJD restricts diffusion (MRI).
.
86
Dilated esophagus on CT, ground glass in the lung bases (and maybe sub-pleural sparing). Diagnosis?
Scleroderma with NSIP
87
Benign Liver Masses Hemangioma: -ultrasound appearance?
Hyperechoic
88
Benign Liver Masses Hemangioma: -CT appearance?
Peripheral nodular discontinuous enhancement
89
Benign Liver Masses Hemangioma: -MR appearance?
T2 Bright
90
Benign Liver Masses Rare in cirrhotics. Which one?
Hemangioma
91
Benign Liver Masses FNH: -ultrasound appearance?
Spoke wheel
92
Benign Liver Masses FNH: -CT appearance?
Homogeneous arterial enhancement
93
Benign Liver Masses FNH: -MR appearance?
“Stealth Lesion —Iso on T1 and T2”
94
Benign Liver Masses Central scar, Bright on delayed eovist (Gd-EOB-DTPA). Which is it?
FNH
95
Benign Liver Masses Hepatic adenoma : -ultrasound appearance?
Variable
96
Benign Liver Masses Hepatic adenoma : -CT appearance?
Variable
97
Benign Liver Masses Hepatic adenoma : -MR appearance?
Fat containing on In/Out phase
98
Benign Liver Masses OCP use, Glycogen storage Disease, can explode and bleed. Which is it?
Hepatic adenoma
99
Benign Liver Masses Hepatic angiomyolipoma : -ultrasound appearance?
Hyperechoic
100
Benign Liver Masses Hepatic angiomyolipoma : -CT appearance?
Gross fat
101
Benign Liver Masses Hepatic angiomyolipoma : -MR appearance?
T1/T2 Bright
102
Benign Liver Masses 50% don’t have fat (unlike renal AML); tuberous sclerosis. Which is it?
Hepatic angiomyolipoma
103
GI location: H. pylori gastritis
Antrum (usually)
104
GI location: Zollinger-Ellison
Jejunal ulcer is buzzword. Duodenal bulb is actually the most common location for ZE ulcers
105
GI location: Crohns
Antrum when in stomach (but uncommon in stomach)
106
GI location: Menetrier’s
Fundus (classically spares antrum)
107
GI location: Lymphoma
“Crosses the pylorus “ Classically describes as doing so, although in reality adenocarcinoma does it more
108
GI location: Giardia
Duodenum
109
GI location: Strongyloides
Duodenum
110
GI location: TB
Terminal ileum
111
GI location: Yersinia
Terminal ileum
112
Herpes esophagitis v CMV and AIDS: Multiple small ulcers
Herpes esophagitis
113
Herpes esophagitis v CMV and AIDS: Solitary large ulcer
CMV and AIDS
114
Esophageal cancer: Black guy who smokes and drinks—mid esophagus. Diagnosis?
Squamous cell
115
Esophageal cancer: White guy with reflux (history of PPIs)—Lower esophagus. Diagnosis?
Adenocarcinoma
116
Uphill v downhill varices: Caused by portal hypertension
Uphill Varices
117
Uphill v downhill varices: Confined to bottom half of esophagus
Uphill Varices
118
Uphill v downhill varices: Caused by SVC obstruction (Catheter or tumor related)
Downhill Varices
119
Uphill v downhill varices: Confined to top half of esophagus
Downhill Varices
120
Traction v Pulsion Diverticulum: Triangular
Traction
121
Traction v Pulsion Diverticulum: Will empty
Traction
122
Traction v Pulsion Diverticulum: Round
Pulsion
123
Traction v Pulsion Diverticulum: Will NOT empty (contain no muscle in their walls)
Pulsion
124
Esophageal hernias, Sliding v Rolling: GE junction ABOVE diaphragm
Sliding
125
Esophageal hernias, Sliding v Rolling: GE junction BELOW diaphragm
Rolling
126
Carney’s *Triad*, parts
- Extra-Adrenal Pheochromocytoma - GIST - Pulmonary Chondroma (hamartoma)
127
Carney’s *Complex*, parts (vaguely)
-*C*ardiac Myxoma (C is for Complex. Not Carney’s Triad!) - skin stuff - endocrine stuff
128
Benign v malignant ulcers (on barium): Width>depth
Malignant
129
Benign v malignant ulcers (on barium): Depth > width
Benign
130
Benign v malignant ulcers (on barium): Located within lumen
Malignant
131
Benign v malignant ulcers (on barium): Project behind expected lumen
Benign
132
Benign v malignant ulcers (on barium): Nodular, irregular edges
Malignant
133
Benign v malignant ulcers (on barium): Sharp contour
Benign
134
Benign v malignant ulcers (on barium): Folds adjacent to ulcer
Malignant
135
Benign v malignant ulcers (on barium): Folds radiate to ulcer
Benign
136
Benign v malignant ulcers (on barium): Aunt Minnie: Carmen Meniscus Sign
Malignant
137
Benign v malignant ulcers (on barium): Aunt Minnie: Hampton’s Line
Benign
138
Inguinal Hernia: direct v indirect Less common
Direct
139
Inguinal Hernia: direct v indirect Medial to inferior Epigastric
Direct
140
Inguinal Hernia: direct v indirect Defect in Hesselbach Triangle
Direct
141
Inguinal Hernia: direct v indirect NOT covered by internal spermatic fascia
Direct
142
Inguinal Hernia: direct v indirect More common
Indirect
143
Inguinal Hernia: direct v indirect Lateral to inferior epigastric
Indirect
144
Inguinal Hernia: direct v indirect Failure of processus vaginalis to close
Indirect
145
Inguinal Hernia: direct v indirect Covered by internal spermatic fascia
Indirect
146
Crohns v UC: Slightly less common in USA
Crohns
147
Crohns v UC: Discontinuous “Skips”
Crohns
148
Crohns v UC: Terminal ileum—String Sign
Crohns
149
Crohns v UC: Ileocecal valve “stenosed”
Crohns
150
Crohns v UC: Mesenteric Fat Increased “creeping fat”
Crohns
151
Crohns v UC: Lymph nodes are usually enlarged
Crohns
152
Crohns v UC: Makes fistula
Crohns
153
Crohns v UC: Slightly more common in USA
UC
154
Crohns v UC: Continuous
UC
155
Crohns v UC: Rectum
UC
156
Crohns v UC: Ileocecal valve “Open”
UC
157
Crohns v UC: Perirectal fat Increased
UC
158
Crohns v UC: Lymph nodes NOT usually enlarged
UC
159
Crohns v UC: Doesn’t usually make fistula
UC
160
Volvulus, sigmoid v cecal: Old person (constipated)
Sigmoid
161
Volvulus, sigmoid v cecal: Young person (mass, prior surgery, 3rd trimester pregnancy)
Cecal
162
Volvulus, sigmoid v cecal: Points to the RUQ
Sigmoid
163
Volvulus, sigmoid v cecal: Points to the LUQ
Cecal
164
Liver Nodules, Regenerative, Dysplastic, HCC: Contains Iron
Regenerative
165
Liver Nodules, Regenerative, Dysplastic, HCC: T1 Dark, T2 Dark
Regenerative
166
Liver Nodules, Regenerative, Dysplastic, HCC: Does NOT enhance
Regenerative
167
Liver Nodules, Regenerative, Dysplastic, HCC: Contains Fat, glycoprotein
Dysplastic
168
Liver Nodules, Regenerative, Dysplastic, HCC: T1 bright, T2 dark
Dysplastic
169
Liver Nodules, Regenerative, Dysplastic, HCC: Usually does not enhance
Dysplastic
170
Liver Nodules, Regenerative, Dysplastic, HCC: T2 Bright
HCC
171
Liver Nodules, Regenerative, Dysplastic, HCC: Does enhance
HCC
172
Central Scars, FNH v FL HCC T2 Bright
FNH
173
Central Scars, FNH v FL HCC Enhances on delay
FNH
174
Central Scars, FNH v FL HCC Mass is sulfur colloid avid (sometimes)
FNH
175
Central Scars, FNH v FL HCC T2 Dark (usually)
FL HCC
176
Central Scars, FNH v FL HCC Does NOT enhance
FL HCC
177
Central Scars, FNH v FL HCC Mass is gallium avid
FL HCC
178
Hepatic Adenoma v FNH: Usually >8cm
Hepatic Adenoma
179
Hepatic Adenoma v FNH: No bile ducts
Hepatic adenoma
180
Hepatic Adenoma v FNH: No kupffer cells
Hepatic adenoma
181
Hepatic Adenoma v FNH: Sulfur colloid cold
Hepatic adenoma
182
Hepatic Adenoma v FNH: Usually < 8cm
FNH
183
Hepatic Adenoma v FNH: Normal bile ducts
FNH
184
Hepatic Adenoma v FNH: Normal kupffer cells
FNH
185
Hepatic Adenoma v FNH: Sulfur colloid hot (sometimes)
FNH
186
HCC v FL HCC: Cirrhosis
HCC
187
HCC v FL HCC: Older (50-60s)
HCC
188
HCC v FL HCC: Rarely calcifies
HCC
189
HCC v FL HCC: Elevated AFP
HCC
190
HCC v FL HCC: No cirrhosis
FL HCC
191
HCC v FL HCC: Younger (30s)
FL HCC
192
HCC v FL HCC: Calcifies sometimes
FL HCC
193
HCC v FL HCC: Normal AFP
FL HCC
194
Hemochromatosis—primary v secondary: Genetic—increased absorption
Primary
195
Hemochromatosis—primary v secondary: Liver, Pancreas
Primary | P for Pancreas and Primary
196
Hemochromatosis—primary v secondary: Heart, thyroid, pituitary
Primary
197
Hemochromatosis—primary v secondary: Acquired—chronic illness, and multiple transfusions
Secondary
198
Hemochromatosis—primary v secondary: Liver, Spleen
Secondary | S is for Spleen and Secondary
199
Heterotaxia Syndromes: Right-sided or left-sided? Two fissures in left lung
Right
200
Heterotaxia Syndromes: Right-sided or left-sided? Asplenia
Right
201
Heterotaxia Syndromes: Right-sided or left-sided? Increased cardiac malformations
Right
202
Heterotaxia Syndromes: Right-sided or left-sided? Reversed aorta/IVC
Right
203
Heterotaxia Syndromes: Right-sided or left-sided? One fissure in right lung
Left
204
Heterotaxia Syndromes: Right-sided or left-sided? Polysplenia
Left
205
Heterotaxia Syndromes: Right-sided or left-sided? Less cardiac malformations
Left
206
Heterotaxia Syndromes: Right-sided or left-sided? Azygous continuation of the IVC
Left
207
Intralobular v Extralobular Sequestration: No pleural covering
Intralobular
208
Intralobular v Extralobular Sequestration: More common
Intralobular
209
Intralobular v Extralobular Sequestration: Presents later with recurrent infection
Intralobular
210
Intralobular v Extralobular Sequestration: Has its own pleural covering
Extralobular
211
Intralobular v Extralobular Sequestration: Less common
Extralobular
212
Intralobular v Extralobular Sequestration: Presents early with other bad congenital things (heart etc)
Extralobular
213
Location: Intralobular sequestration
Left lower lobe
214
Location: Congenital lobar emphysema (CLE)
Left UPPER lobe
215
Location: CCAM
No lobar preference
216
Baby liver, Age 0-3: Diagnosis? Endothelial growth factor elevated
Hemangioendothelioma
217
Baby liver, Age 0-3: Diagnosis? High flow heart failure, big heart on CXR
Hemangioendothelioma
218
Baby liver, Age 0-3: Diagnosis? AFP elevated
Hepatoblastoma
219
Baby liver, Age 0-3: Diagnosis? Associated with Wilms
Hepatoblastoma
220
Baby liver, Age 0-3: Diagnosis? Associated with prematurity
Hepatoblastoma
221
Baby liver, Age 0-3: Diagnosis? Can cause precocious puberty
Hepatoblastoma
222
Baby liver, Age 0-3: Diagnosis? AFP negative
Mesenchymal hamartoma
223
Baby liver, Age 0-3: Diagnosis? “It’s really cystic.”
Mesenchymal hamartoma
224
How often is it bilateral? Blount’s Disease
“Often”—some sources say 50-60%
225
How often is it bilateral? SCFE
33% (1/3)
226
How often is it bilateral? Perthes
10%
227
How often is it bilateral? Wilms
5-10%
228
How often is it bilateral? DDH
20%
229
Neuroblastoma: - age - calcifies how often - encase or invade vessels? - Well circumscribed? - mets to bone?
- age: usually less than 2 (can occur in utero) - calcifies 90% - ENCASES vessels (no invasion) - NO. Poorly marginated. - yes, mets to bones
230
Wilms: - age - calcifies how often - encase or invade vessels? - Well circumscribed? - mets to bone?
- age: usually around 4 (NEVER BEFORE 2 mo) - calcifies rarely (<10%) - INVADES vessels (no encasing) - YES well circumscribed - no, doesn’t usually met to bone (unless clear cell Wilms variant)
231
Neuroblastoma v Adrenal Hemorrhage: Anechoic and avascular
Neuroblastoma
232
Neuroblastoma v Adrenal Hemorrhage: Low on T2
Neuroblastoma
233
Neuroblastoma v Adrenal Hemorrhage: Will grow on follow up
Neuroblastoma
234
Neuroblastoma v Adrenal Hemorrhage: Echogenic and vascular
Adrenal hemorrhage
235
Neuroblastoma v Adrenal Hemorrhage: High on T2
Adrenal hemorrhage
236
Neuroblastoma v Adrenal Hemorrhage: Should shrink on follow up
Adrenal hemorrhage
237
Cardiac: Cyanotic congenital heart diseases (6)
“Think the 6 Ts “ ``` TOF TAPVR Transposition Truncus Tricuspid atresia ``` (That’s only 5, and I don’t know why)
238
Which MRI sequence is best? Cardiac Myxoma
Low T1, High T2 | High myxoid content
239
Which MRI sequence is best? Acute vs Chronic MI
Look at T2–Bright on Acute, Dark on chronic (fibrous scar)
240
Which MRI sequence is best? Arrhythmogenic Right Ventricular Dysplasia (ARVD)
T1 Bright
241
Which MRI sequence is best? Microvascular Obstruction
First pass perfusion (25 sec post Gad)
242
Which MRI sequence is best? Infarct
Delayed Enhancement (10-12 min post Gad)
243
Constrictive v restrictive Cardiomyopathy: Pericardium is usually thickened in which?
Constrictive
244
Constrictive v restrictive Cardiomyopathy: Diastolic septal bounce is seen in which?
Constrictive (sigmoidization of the septum)
245
True v False Ventricular Aneurysm: Mouth is wider than body
True
246
True v False Ventricular Aneurysm: Myocardium intact
True
247
True v False Ventricular Aneurysm: Anterior-lateral wall
True
248
True v False Ventricular Aneurysm: Mouth is narrower than body
False
249
True v False Ventricular Aneurysm: Myocardium is NOT intact
False Pericardial adhesions contain rupture
250
True v False Ventricular Aneurysm: Posterior-lateral wall
False
251
True v False Ventricular Aneurysm: Higher risk of rupture
False
252
Stunned v hibernating myocardium v infarct/scar: Wall motion normal or abnormal for each?
Abnormal wall motion for all
253
Stunned v hibernating myocardium v infarct/scar: Perfusion normal or abnormal for each?
Stunned: normal perfusion (thallium or sestamibi) Hibernating: abnormal fixed perfusion Infarct/scar: abnormal fixed perfusion
254
hibernating myocardium v infarct/scar: Will it redistribute with delayed thallium? (decide for each) Will it take up FDG? (For each)
Hibernating: -WILL redistribute w/delayed thallium -WILL take up FDG Infarct/Scar: - will NOT redistribute - will NOT take up FDG
255
Stunned v hibernating myocardium v infarct/scar: What is each associated with?
Stunned: Acute MI Hibernating: chronic high grade CAD Infarct/Scar: chronic prior MI
256
Left Atrial Myxoma v Clot: Which will enhance?
Myxoma
257
Vegetations v Fibroelastoma: How do you tell difference?
Look for valvular damage (seen with vegetations). Contrast enhancement is NOT reliable bc how small these things are.
258
Internal v External Carotid For each: -resistance
Internal: Low resistance External: high resistance
259
Aortic coarctation : Infantile—pre-ductal - can have pulmonary edema. - Usually long segment. - blood supply to descending aorta via PDA Adult—ductal - not symptomatic until later childhood - often differential arm-leg pressure - usually short segment
.
260
Internal v External Carotid For each: -systolic velocity
Internal: low systolic velocity External: high systolic velocity
261
Internal v External Carotid For each: -diastolic velocity
Internal : does not return to baseline External: approaches zero baseline
262
Internal v External Carotid For each: -color flow
Internal: continuous color flow is seen throughout the cardiac cycle External: color flow is intermittent during cardiac cycle
263
HIV Encephalitis v PML: Which is symmetric, T2 bright, T1 normal?
HIV Encephalitis
264
HIV Encephalitis v PML: Which is asymmetric, T2 bright, T1 dark?
PML
265
# Choose HIV Encephalitis, PML, CMV, Toxo, or Cryptococcus Symmetric, T2 bright
HIV Encephalitis
266
# Choose HIV Encephalitis, PML, CMV, Toxo, or Cryptococcus Asymmetric T2 bright, T1 dark
PML
267
# Choose HIV Encephalitis, PML, CMV, Toxo, or Cryptococcus Periventricular, T2 bright, Ependymal enhancement
CMV
268
# Choose HIV Encephalitis, PML, CMV, Toxo, or Cryptococcus Ring enhancement, thallium cold
Toxo
269
# Choose HIV Encephalitis, PML, CMV, Toxo, or Cryptococcus Dilated perivascular spaces , basilar meningitis
Cryptococcus
270
Toxo v lymphoma: For each: - ring enhancing? - hemorrhage after treatment common? - thallium cold/hot? - PET cold/hot? - MR perfusion ?
Toxo: - ring enhancing - hemorrhage after treat MORE common - *Thallium COLD* - PET COLD - MR perfusion: DECREASED CBV Lymphoma: - ring enhancing - hemorrhage LESS common - *Thallium HOT* - PET HOT - MR perfusion: Increased (or decreased) CBV
271
LeFort unique components: LeFort 1? LeFort 2? LeFort 3?
LeFort 1—lateral nasal aperture LeFort 2—inferior orbital rim, orbital floor LeFort 3–zygomatic arch, lateral orbital rim/wall
272
Temporal bone fractures: longitudinal v transverse For each: -which axis of T-bone?
Longitudinal: long axis of t-bone Transverse: short axis
273
Temporal bone fractures: longitudinal v transverse For each: -more or less common?
Longitudinal: More Transverse: less
274
Temporal bone fractures: longitudinal v transverse For each: -ossicular dislocation or vascular injury?
Longitudinal: More ossicular dislocation Transverse: more vascular injury (carotid/jugular)
275
Temporal bone fractures: longitudinal v transverse For each: -less or more facial nerve damage?
Longitudinal: LESS facial nerves damage (20%) Transverse: MORE facial nerve damage (>30%)
276
Temporal bone fractures: longitudinal v transverse For each: -type of hearing loss
Longitudinal: CONDUCTIVE Tranverse: SENSORINEURAL
277
Open lip schizencephaly: cleft lined by grey matter (malformation) Porencephalic cyst: Hole from prior ischemia
.
278
Vocal cord paralysis v cancer: - Affected side dilated w/Vocal cord paralysis - opposite side dilated w/Cancer
.
279
Syndromes w/tumors (neuro)— Name associated tumors: NF-1
Optic nerve gliomas
280
Syndromes w/tumors (neuro)— Name associated tumors: NF-2
MSME: Multiple Schwannomas Meningiomas Ependymomas
281
Syndromes w/tumors (neuro)— Name associated tumors: VHL
Hemangioblastoma (brain and retina)
282
Syndromes w/tumors (neuro)— Name associated tumors: TS
Subependymal Giant Cell Astrocytomas, Cortical tubers
283
Syndromes w/tumors (neuro)— Name associated tumors: Nevoid Basal Cell Syndrome (Gorlin)
Medulloblastomas
284
Syndromes w/tumors (neuro)— Name associated tumors: Turcot
GBM, Medulloblastoma (Note: nevoid basal cell Syndrome also has medulloblastoma)
285
Syndromes w/tumors (neuro)— Name associated tumors: Cowdens
Lhemitte-Dulcos (dysplastic cerebellar gangliocytoma)
286
Meningioma v Schwannoma: For each: -enhance homogenous or not?
Meningioma: enhance homogenously Schwannoma: LESS homogenous
287
Meningioma v Schwannoma: For each: -invade IAC?
Meningioma: do NOT usually invade IAC Schwannoma: INVADE IAC
288
Meningioma v Schwannoma: Which calcifies more often?
Meningioma: calcify MORE often
289
Meningioma v Schwannoma: Which IAC can have “trumpeted” appearance?
Schwannoma
290
Where is blood coming from? Match blood location to aneurysm location ACOM
Interhemispheric fissure
291
Where is blood coming from? Match blood location to aneurysm location PCOM
Ipsilateral basal cistern
292
Where is blood coming from? Match blood location to aneurysm location MCA Trifurcation
Sylvian fissure
293
Where is blood coming from? Match blood location to aneurysm location Basilar tip
Interpeduncular cistern OR intraventricular
294
Where is blood coming from? Match blood location to aneurysm location PICA
Posterior fossa OR intraventricular
295
Skull hole contents: Foramen ovale
CN V3 | Accessory meningeal artery
296
Skull hole contents: Foramen rotundum
CN V2 | “R2V2”
297
Skull hole contents: Superior orbital fissure
CN 3 CN 4 CN V1 CN 6
298
Skull hole contents: Inferior orbital fissure
CN V2
299
Skull hole contents: Foramen spinosum
Middle meningeal artery
300
Skull hole contents: Jugular foramen
Jugular vein, CN 9 CN 10 CN 11
301
Skull hole contents: Hypoglossal Canal
CN 12
302
Skull hole contents: Optic canal
CN 2 | Ophthalmic Artery
303
Forearm Fractures: Fracture of the radial head + Anterior dislocation of the distal radial ulnar joint
Essex-lopresti
304
Forearm fractures: Radial shaft fracture with anterior dislocation of the ulna at the DRUJ
Galeazzi fracture (MU*GR*)
305
Forearm fracture: Fracture of proximal ulna, with anterior dislocation of the radial head
Monteggia Fracture (*MU* GR)
306
Femoral neck stress fractures: - medial side: - stress fracture, - compressive side, - does well - lateral side: - bisphosphonate - tensile side - does terrible
.
307
Bankart spectrum shoulders: Which has disrupted periosteum?
True Bankart
308
Avulsions—what attaches to... Iliac crest
Abdominal muscles
309
Avulsions—what attaches to... ASIS
Sartorius | A*S*IS....*S*artorius
310
Avulsions—what attaches to... AIIS
Rectus femoris
311
Avulsions—what attaches to... Greater trochanter
Gluteal muscles
312
Avulsions—what attaches to... Lesser trochanter
Illiopsoas
313
Avulsions—what attaches to... Ischial tuberosity
Hamstrings
314
Avulsions—what attaches to... Symphysis
ADDuctor group
315
Pincer v Cam Impingement : For each: -age, gender
Pincer: middle-age women Cam: young men
316
Pincer v Cam Impingement : Which is: Over coverage of femoral head by acetabulum
Pincer
317
Pincer v Cam Impingement : Which is: “Cross over sign”
Pincer
318
Pincer v Cam Impingement : Which is: Bony protrusion on antero-superior femoral head-neck junction
Cam
319
Pincer v Cam Impingement : Which is: Pistol grip deformity (appearance of the femur)
Cam
320
Osteochondroses: Kohlers: - location - age, gender - treatment
- tarsal navicular - male 4-6 - treatment NOT surgical
321
Osteochondroses: Freiberg Infarction: - location - age, gender - can lead to _____
- Second metatarsal head - adolescent girls - can lead to secondary OA
322
Osteochondroses: Severs: - location - some say this is ______
- calcaneal apophysis | - some say it’s a normal growing pain
323
Osteochondroses: Panners : - location - age - does not have ______
- capitellum - kid 5-10 “thrower “ - does not have loose bodies
324
Osteochondroses: Perthes: - location - age, race
- femoral Head | - white kid, 4-8
325
Osteochondroses: Kienbock: - location - age - associated with....
- carpal lunate - person 20-40 years - associated with negative ulnar variance
326
Finger tip tumors: T1 dark, * T2 BRIGHT*, * ENHANCES AVIDLY* Diagnosis?
Glomus
327
Finger tip tumors: T1 dark, T2 Dark, Variable enhancement, *BLOOM ON GRADIENT* Diagnosis?
Giant cell tumor tendon
328
Finger tip tumors: T1 dark, T2 dark, *NO BLOOMING* Diagnosis?
Fibroma