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EORs > FM 1 > Flashcards

FM 1 Flashcards

1
Q

anticoagulation determination for AFIB

A

CHA2DS2VAS

CHF---1 
HTN----1 
age >75---1 
DM--1
Stroke, TIA, TE-----2 
Vascular disease (prior MI, PAD, CABG)---1 
Age 65-74----1 
Sex---female---1

Score 0= no anticoag
score 1— no coag or just ASA (81-324) or OAC
score 2—- OAC
score >3—- OAC

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2
Q

rhythm control for AFIB

  • under 48 hours
  • over 48 hours
  • unstable
A

CCB (diltiazem or verapamil)
or
BB like metoprolol

under 48 hrs: cardioversion, amiodarone (obtain transesophageal echo (TEE) to determine if a clot is present prior to cardioversion)

over 48: anticoagulate for 21 days prior to cardioversion

unstable= synch cardiov

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3
Q

list DOACs

A

direct oral anticoags

dabigatran, rivaroxaban, apixaban, or edoxaban

-ban or -an

**better than warfarin

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4
Q

when to give warfarin for afib

A

mechanical heart valves, mitral stenosis, unacceptable increase in cost, EGFR < 30 ml/min, on certain medications (phenytoin or certain antiretroviral therapy

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5
Q

Adjusted-dose warfarin target INR is

A

2.5

range=2-3

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6
Q

what is top picked DOACs

A

apixaban or Eliquis best balance of safety and efficacy,

Xarelto (rivaroxaban) for once-daily dosing

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7
Q

dont jump to what meds for AFIB anticoag control

A

ANTI PLATS

  • asa
  • clopidogrel

These antiplatelets aren’t as effective as an anticoagulant in reducing stroke risk in atrial fib patients…and bleeding risk may not be lower

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8
Q

consider what drug if CrCl is below 30 mL/min

A

warfarin

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9
Q

what is clopidogrel

A

antiplatelet

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10
Q

when would you have to lower the dose of ELiquis

A

if taken with Clarithromycin

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11
Q

Virchow triad

A

Stasis, hypercoaguability and trauma (like surgery)

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12
Q

Describe homan’s sign

A

Extend the leg…and then push the foot towards the head (Doris flex) and will cause pain

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13
Q

DVT

  • diagnosis —-GS, 1st line, others
  • tx
A

DX= 1st line is venous duplex US, D-diner (will r/o DVT in low risk PT), GOLD STANDARD= venography,

TX

  1. Immediate anti coagulation:
    * LMW heparin + warfarin, LMWH + either Dabigatran or Eboxaban as mono therapy with Rivaroxaban or Apixaban (all DOACs) or fondaparinux, or the oral factor Xa inhibitors
  2. IVC filter—- 3 indications
    * recurrent despite adequate anticoagulation
    * stable PT in whom anticoagulation is contradinicated
    * right ventricular dysfunction with an enlarged RV on echo
  3. Thrombosis is or thrombectomy: generally not done—reserved for massive DVT or severe cases
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14
Q

Tx for a pregnant woman with DVT

A

LMWH as initial and long term tx

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15
Q

Leg pain that worsens with walking, elevation of leg

A

Peripheral ARTERY disease

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16
Q

Leg pain that is improved with walking or elevation

A

PVD

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17
Q

Cyanotic leg with dependency

A

PVD

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18
Q

Red leg with dependency

A

PAD

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19
Q

Leg ulcers at the medial malleolus with uneven ulcer margins

A

PVD

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20
Q

Leg ulcers at lateral malleous with clean margins

A

PAD

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21
Q

Leg/foot has brownish pigmentation—— eczema toys rash, thickening of skin

A

PVD

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22
Q

Atrophic changes to leg/foot…. thin shiny skin, loss of hair, muscle atrophy, pallor, thick nails, Mottled appearance

A

PAD

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23
Q

Pericarditis

  • two MCC
  • CM-what makes pain better/worse
  • DX
  • TX *****
A

MCC=viral (coxsachkike virus , echovirus) and idiopathic
OTHER CAUSES= dressier syndrome (post MI + fever +pleural eff), autoimmune, uremia, bacterial, radiation, medications

CM
*sudden onset CP==sharp, worse with inspiration, persistent, postural—-worse when supine and improved with sitting forward

DX

  1. EKG: diffuse ST elevations in precordial leads (v1-v6) with PR depressions
  2. +/- cardiac enzymes
  3. ECHO:

TX

  1. NSAIDs or ASA first line x7-14 days
  2. Colchicine 2nd line

If dressier syndrome—> ASA or colchicine (AVOID NSAIDs bc they can interfere with Myocardial scar formation)

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24
Q

Sinus rate that varies with inspiration

A

Sinus arrhythmia

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25
Q

systolic-ejection murmur (SEM) heard in the second intercostal space (ICS) at the right sternal border with radiation to the carotids and the apex.

A

AS

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26
Q

Murmur is decreased with Valsalva

A

AS

27
Q

Harsh systolic ejection crescendo-decrescendo murmur at the right upper sternal border with radiation to the neck and apex heard best by leaning forward with expiration and squatting associated with a split S2.

A

AS

28
Q

What happens to LV with aortic stenosis

A

Increases after load in LV—-> LV hypertrophy

29
Q

Causes of AS

A

Rheumatic, congenital, degenerative

30
Q

A common mnemonic for aortic stenosis symptoms is

A

SAD

Syncope
Angina
Dyspnea

31
Q

AS

-decreases and increase with>

A

Increases when squatting

Decreases with hand grip and Valsalva or straining of any kind

32
Q

Murmur may rad to the neck or apex

A

AS

32
Q

Murmur may rad to the neck or apex

A

AS

32
Q

Murmur may rad to the neck or apex

A

AS

33
Q

Helmet cells

A

fragmented red blood cells that can be caused by aortic stenosis due to mechanical damage of red blood cells as they pass through the calcified valves. Helmet cells are also known as schistocytes

34
Q

Etiologies of AS

A
  1. Degenerative: calcification, wear and tear esp >70
  2. Congenital and bicuspid valve common in <70
  3. Rheumatic HD: isolated or also pained with AR
35
Q

What can happen in peripartum period to the heart

A

Dilated cardiomyopathy

36
Q

S/s of dilated cardiomyopathy

A

S3 gallop ********
Edema of LE
Lungs—- rales
JVD

Exertional dyspnea, edema, fatigue, loss of appetite, and cough

37
Q

To dilated cardiomyopathy

A

Tx is similar to CHF

  • ACEI + BB + Loop diuretic
  • dig to increase contractility
  • exrtreme cases= transplant or LVAD
38
Q

Graves Dz

-dx

A

HYPERthyroid

  1. Primary HYperthyroid profile: Low TSH and high T3** or T4
    • thyroid stimulating immunoglobulins is HALLMARK
  3. Radioactive uptake scan: diffuse, increased iodine uptake

Test to measure Anti-thyrotropin antibodies (TSHR-Ab).
***TSI—- thyroid stimulating immunoglobulin
Or
** TSH-binding inhibitor immunoglobulin (TBII)

39
Q

Sudden ocular pain. She reports she was visiting the planetarium when the pain started and when she walked outside she saw halos around the street lights. The pain was so bad that she began to vomit. She reports her vision is decreased. Physical examination reveals conjunctival injection, a cloudy cornea, and pupils

A

Acute angle closure glaucoma

40
Q

Tx for acute angle closure glaucoma

  • 1st line
  • definitive
  • others
A

COMBO OF SYSTEMIC (IV) AND TOPICAL AGENTS

IV or PO  Acetazolamide or Mannitol: the first-line agent - decrease IOP by decreasing aqueous humor production 
\+
timolol
OR 
Pilocarpine, Carbachol 
OR
Apraclonidine

Peripheral iridotomy (punches a hole in the iris) is the definitive treatment

41
Q

Injected conjunctiva, cloudy cornea and fixed dilated pupil

A

Triad for acute narrow angle closure glaucoma

42
Q

Main difference b/w s/s of iritis and acute narrow angle closure glaucoma

A

IRITIS—- small constricted pupil
GLAUCOMA—— fixed dilated pupil

**BOTH HAVE SIM S/S—— sudden pain, red conjunctiva, cloudy cornea etc

43
Q

patient cannot wrinkle forehead

A

bells palsy

44
Q

patient can wrinkle forehead

A

TIA NOT bells palsy

45
Q

Two types of TIA exist:

A

Large artery low flow TIA (stenosis) likely carotid stenosis causing short live (minutes) decrease in flow to the brain. If the stenosis is > 70% treat surgically

Embolic TIA: emboli often form in the heart (afib)

46
Q

s/s of TIA and what do the s/s correlate to which vessel

A

Interal Carotid: amaurosis Fugaux—monocular vision loss–temporary “lampshape down on the eye”, weakness in CONTRAlateral hand

ICA/MCA/ACA: Cerebral hemisphere dysfunction. Sudden headache, speech changes, confusion

PCA: somatosensory deficit

Vertebrobasilar: brainstem/cerebral symptoms (gait and proprioception)

47
Q

Diagnostic studies for TIA

TOC for pt with suspected cardiac etiology?

A

Most sensitive = MRI
CT, carotid doppler to look for stenosis, CT angiography, MR angiography

-EKG to r/o AFIB

Transthoracic echocardiogram (TTE) is the preferred initial test for the majority of patients with a suspected cardiac or aortic source of emboli

48
Q

Essential tremor– genetics?

A

autosomal dominant

so pt will have a + fam hx

49
Q

when does shaking occur with essential tremor

  • tremor aggrivated by?
  • tremor better with?
A

simple tasks such as tying shoelaces, handwriting, shaving or simply holding hands against gravity

aggrivated by stress, caffiene, fatigue

better with ETOH

50
Q

first line tx for essential tremor

0ther drugs

A

propranolol 1st line

Primidone, alprazolam, small amounts of alcohol, gabapentin, topiramate, or nimodipine
Drug-resistant cases - Deep brain stimulation

51
Q

essential tremor is also called

A

intentional tremor

***PKD tremor is at rest

52
Q

tx for animal bite

A
  1. stop any bleeding
  2. high pressure irrigation
  3. The wound should be explored under local anesthesia to assess its depth and look for foreign bodies
  4. Primary wound closure is indicated for superficial dog bites
  5. Cat and human bites aren’t closed and instead should close on their own by secondary intention due to the high risk of infection
  6. . Regardless of the biting mammal, a wound to the face should be closed because cosmesis is important
  7. tetanus prophylaxis tetanus toxoid and tetanus immunoglobulin should be considered
  8. wild animal bite = rabies vaccine and/or immunoglobulin
  9. Augmentin 875 one tab PO BID x 7-10 days***** or Clindamycin 450 mg TID + Bactrim DS 1-2 tab BID or PCN Allergy/Pregnant - Azithromycin 500 PO x 1 then 250 mg PO x 5 days
53
Q

When a decision is made to close a mammal bite wound, four criteria must be fulfilled

A
  1. uninfected
  2. <24 hrs
  3. not located on hand or foot
  4. pt should have healthy immune system
54
Q

Osteoporosis

-diagnosis tests–gs etc

A

TESTS:
GS: DEXA
***POST MENOPAUSAL WOMEN EVERY 2 YRS >65 yo
**POSTMEN WONEN <65 YO WTH RF OR FXS

55
Q

pt is taking bisphosphonates– what do you tel htem to do right away after taking it

A

sit uprgith for at leat 30 min

Oral bisphosphonates must be taken on an empty stomach with a full (8-oz, 250 mL) glass of water, and the patient must remain upright for at least 30 min

56
Q

list t score for osteporosis and osteopneia

A 
porosis= < or equal to -2.5 
penia= -1 to -2.5
57
Q

DeXA scannig frequency based on T scores
-1.0–>-1.5

-1.5 to -2.0

greater than -2.0

A

T score of -1.0 to -1.5 every 5 years

T score -1.5 to -2.0 every 3-5 years

T score of greater than - 2.0 every 1 to 2 years”

58
Q

PE findings for osteoarthritis

  • common joints
  • xray findings
A

bony enlargement of fingers—–DIP (herbenderbs nodes), and or bouchards nodes (PIP)

PAIN WILL IMPROVE WITH REST AND WORSEN WITH ACTIVITY********(comp to RA….. you have the morning stiffness and then improves with activity)

***morning stiffness UNDER 30 mins

***evening joint stiffness

main thing is that the pain/stiffness imrpoves with rest and worsens with use

MC hands, hips and knees
As OA progresses, joint motion becomes restricted, and tenderness and crepitus or grating sensations develop

Osteoarthritis does not involve the metacarpophalangeal joints

XRAY findings

  • osteophytes: bony projections that form along joints
  • joint space narrowig
  • joint sclerosis: area just below the cartilage layer fills with collagen and becomes denser than healthy bone
59
Q

Hypertensive crisis

  • define
  • crisis vs urgency
A

57 y/o with BP of 170/110, intermittent loss of vision right eye, and headaches—– CRISIS

crisis/emergency= evere hypertension (SBP ≥ 180 and/or DBP ≥ 120) WITH signs of damage to target organs (except papilledema which = hypertensive retinopathy) - encephalopathy, nephropathy, intracranial hemorrhage, aortic dissection, pulmonary edema, unstable angina or MI

urgency= Very high blood pressure (systolic ≥ 180 or diastolic ≥ 120) WITHOUT** target-organ damage

60
Q

tx for HTN urgency and crisis/emergency

A

Hypertensive urgency = clonidine (drug of choice)

Hypertensive emergency = IV sodium nitroprusside (nicardipine) (drug of choice)— CCB

  • ** should be used with a BB like labetalol
  • **BP must be reduced in 1 hour to avoid morbidity or death
61
Q

tx for BPH

A
  1. Relax the bladder/urethra: α-1 blockers - tamsulosin (Flomax) most uroselective provides rapid symptom relief - smooth muscle relaxation of prostate and bladder neck decreases urethral resistance and obstruction which increases urinary flow can cause dizziness and orthostatic hypotension as well as retrograde ejaculation
  2. Decrease prostate size (shrink prostate): 5 alpha-reductase inhibitors (finasteride) and (dutasteride) (androgen inhibitor - inhibits the conversion of testosterone to dihydrotestosterone suppressing prostate growth, and reducing bladder outlet obstruction) has a positive effect on the clinical course of BPH
  3. TURP (transurethral resection of the prostate) if refractory to meds - removes excess prostate tissue to relieve obstruction - sexual dysfunction and urinary incontinence

EORs (15 decks)

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