Final Flashcards
virchow triad
venous tasis
hypercoag
vascular endo disruption
thoracic aneurysm cause
genetic
abdominal anesurysms cause
lifestyle
peripheral vascualar disease casuse
smoking
lifestyl
coronary subclavian steal
sends blood from the heart to arm
subclavian steal
sends blood from brain to arm
BP in affected arm of subclavian steal is
lower
raynaud’s treatment
keep warm
avoid art line
avoid epi additive
3 protection mechanisms for neuro
SSEP/EEG
ice epidural
CSF drain
AAA ruptures into
L retroperitoneum
AAA rupture triad
pulsatile abdominal mass
severe back pain
hypotension
temporal arteritis risk
high fire risk
brain blood supply is ______ of CO
20% of CO
brain blood supplied by
internal carotids
vertebral arteries
vertebral arteries combine into the
basillar arteries
what combines in the circle of willis
basilar and internal carotids
innermost layer of artery
endothelium
atherosclerosis
response to vessel wall injury
results in endothelium dysfuntion
indication of vascular disease
claudication
aneurysm
abnormal weakening of vessel
dissection
tear in intima
false lumen
primary cause of thoracic aortic disease
genetic bicuspid aortic valve
stanford A
ascending
stanford B
no ascending
goal vascular ACT
200-300s
heparin dose for vascular
100U/kg
spinal cord ischemis risk increases with
corss clamp > 30s
radial art line for thoracic repair
right
BP above/below clamp
above: 100 mmHg
below: 50 mmHg
clevidipine dosing
2mg/hr
max: 32 mg/hr
highest risk of endovascular AAA repair
stent migration/thrombosis
when can you extubate carotid endarctectmy
when awake enough to follow commands
CAE damage
laryngeal nerve injury
carotid body injury
most important DVT complication
PE
best DVT prevention
lovenox
recurrent PE treatment
IVC filter
DVT prevention in pts who are CI to anticoags
IVC filter
CoW
protects agains ischemia/stroke
collateral circulation
in order for collateral flow through CoW
pressure must be high enough for retrograde flow
- contralateral anterior
- middle cerebral
SV factors
preload
afterload
contractility
contractility is independend of
preload
afterload
estimate preload
LV EDP ~ PAWP
estimate SVR
SVR ~ MAP or SBP
incr SVR ____ CO
incr SVR = decr CO
all heart disease has
incr VEDP
LVEDP ~
LVEDP ~ PAWP
LVEDV ~
LVEDV ~ CO = HRxSV
normal CI
2.5-4.2
systolic HF
HFrEF
LV systolic dysfunction
diastolic HF
HFpEF
poor relaxation = poor filling
systolic HF tachycardia
incr CO
diastolic HF tachycardia
decr CO
HF general compensation
decr Vmax
incr SVR
incr sympa
incr vascular tone
Frank starline
incr filling pressure needed to mx SV/CO
what compensates for increased pressure
hypertrophy
hypertrophy state
low inotropic state
what compensates for fluid overload
dilation
most common myocardial remodeling
ischemic injury
treat myocardial remodeling
ACE
aldosterone inhibitors
what is associated w/HF
elevated BNP
balloon pump diastole
inflates
balloon pump systole
deflates
causes of RHF
LV failure
PHTN
RH MI
most common cause of RHF
LV failure
causes of LHF
LH MI
chronic HTN
acute HTN
aortic/mitral dz
high output HF
good CO
poor perfusion
high output CO diagnosis
CO > 8L/min
CI> 4 L/min/m2
normal SVR
700-1200
normal PA
< 20 mmHg
low contractility (LOW CI), give
milrinone
low afterload, give
vasopressor (NE)
low contractility, give
inotrope (dobutamine)
low contractility, low HR, low SVR, give
EPI
transplanted heart is
preload dependent
vasopressor for transplanted heart
phenylephrine
worsen LVOT
incr contractility
decr preload
decr SVR
PPV
labor
drugs worsen LVOT
ephedrine
dobutamine
epi
improve LVOT
decr contractility
incr preload
incr SVR
drugs improve LVOT
BB
CCB
HOCM goals
decr LVOT
decr ischemia
avoid arrythmia
HOCM pregnancy labor analgesia
epidural
HOCM pregnancy vasopressor
phenylephrine
fluids
dilated CM
LV dilation
preipartum CM
type of dilated CM
peripartum CM treatment
give diuretics
give vasodilators
treat restrictive CM
ketamine
no PPV
avoid brady
cor pulmonale
RV enlargement
can turn into RHF
cor pulmonale management
avoid hypoxia
avoid hypercarbia
avoid overnarcsq
open diastole
T
M
open systole
P
A
starling
incr preload = incr SV
starling overstrethcing
decr contractility
decr SV
concentric hypertrophy
incr P
eccentric hypertrophy
incr V
LV failure
pulm edema
RV failure
lower edema
stenosis
normal HR
mx preload
mx afterload
avoid myocardial depression