Exam 2 neuro blueprint Flashcards
what are the early signs of ICP monitoring?
Early signs:
Agitation
restless/ irritability
Change in LOC
Decreased mental status
Sudden vomiting w/o nausea
what are the late signs of ICP monitoring?
Late signs:
Seizures
Posturing (decorticate and decerebrate)
Cushing’s triad
what are the late deadly signs of ICP monitoring?
Late deadly signs:
Lungs: irregular respirations and Cheyne strokes respirations
Neck: nuchal rigidity (stiff neck)
Brain stem affected:
Eyes: pupils “fixed and dilated” unequal and 8mm (normal = 2-6 mm)
Foot: babinski reflex and toes fan out when stimulated = BAD
Seizures and coma
Abnormal posturing: decorticate and decerebrate
what is the glascow coma scale?
Behavior = eyes opening, verbal response, and motor response
Response = eyes (1-4 grade), verbal (1-5 grade), and motor (1-6 grade)
Eyes opening = 4 (open spontaneously), 3 (open to speech), 2 (open to pain), 1 (no eye opening)
Verbal response = 5 (oriented to time, person, place), 4 (confused), 3 (inappropriate words), 2 (incomprehensible sounds), 1 (no verbal response)
Motor response = 6 (obey commands), 5 (moves to localized pain), 4 (withdrawal from pain), 3 (abnormal flexion), 2 (abnormal extension), 1 (no motor response)
what are the ratings for the glascow coma scale?
13-15 mild
9-12 moderate
3-8 severe
Less than 8 = intubate
what is mannitol?
Is an osmotic diuretic. Elevated blood plasma osmolality, resulting in enhanced flow of H2O from tissues, including the brain and CSF, into interstitial fluid and plasma
Used to decrease ICP
Must be given IV
Systemic EDEMA may occur due to enhanced flow of fluid AWAY from the brain and INTO interstitial space.
Watch for signs of heart failure as a result.
what does ICPS for TBI stand for?
I - immobilize C spine, log roll pt
C - CO2 low; lower CO2 means lower ICP. CO2 dilates vessels and will increase ICP
P - positioning: 30-35 degrees (semi-fowlers), no flexing/bending, not cough, sneezing, etc.
S - suctioning: 10 secs or less, infrequent, hyperventilate pt
what is the management for TBIs?
A,B,C’s (VS w/ O2 stat), stabilize cervical spine (position pt w/ HOB 30-45 degrees), intubate (GCS <8) or if gag impaired or absent, control external bleed, and maintain normothermia
Prevent secondary injury: by treating cerebral edema and increased ICP
Concussion and contusion: monitor and manage ICP
Skull fx: if CSF leak, prophylactic ABX, and surgery (elevate depressed bones, remove fragments; craniotomy and cranioplasty.
Manage ICP monitoring system and ICP: manage N/V and minimize environmental stimulation
Manage eye problems: prevent corneal damage
Manage hyperthermia (goal 36-37 degrees C): cooling blanket, etc.
Manage CSF leak: rhinorrhea or otorrhea (notify HCP), teach client not to sneeze or blow nose and no NGT or NT suction (if basilar skull fx r/f meningitis)
Implement seizure precautions and prophylaxis: manage headache and prevent
VTE, skin breakdown
what is an open TBI?
Basilar skull fracture (visible of the external skull)
Key finding: clear of blood-tinged drainage from the eyes, ears, nose that is positive for glucose = CSF
what is a closed TBI?
Concussion (CM: brief LOC, headache, retrograde amnesia (memory loss of events immediately prior to injury)
Contusion (more emergent; described as: coup, countercoup, and together)
Coup
Countercoup
Together: coup-countercoup
affecting 1 area of the brain due to impact
coup
affecting the area opposite of coup
countercoup
typically affecting the frontal and occipital lobes
together; coup-countercoup
what is a CVA?
1 risk factor = HTN (over 140 SBP)
Types: ischemic & hemorrhagic strokes
what is “BE FAST”?
Be Fast = Balance (headache & dizzy), eyes (sudden loss of vision in one of both eyes), face (does the pts face look uneven?, arm (weak), speech (difficulty), and time (call 911).