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Physiology I > Exam #1: NMJ Diseases > Flashcards

Exam #1: NMJ Diseases Flashcards

1
Q

Is Lambert-Eaton Syndrome a depolarizing blocker or a non-depolarizing blocker?

A

Non-depolarizing blocker

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2
Q

Is Botulism a depolarizing blocker or a non-depolarizing blocker?

A

Non-depolarizing blocker

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3
Q

Is Curate a depolarizing blocker or a non-depolarizing blocker?

A

Non-depolarizing blocker

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4
Q

Is Myasthenia Gravis a depolarizing blocker or a non-depolarizing blocker?

A

Non-depolarizing blocker

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5
Q

Is Hypermagnesemia a depolarizing blocker or a non-depolarizing blocker?

A

Non-depolarizing blocker

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6
Q

Is a Black Widow bite a depolarizing blocker or a non-depolarizing blocker?

A

Depolarizing blocker

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7
Q

Are Acetylcholinesterase Inhibitors a depolarizing blocker or a non-depolarizing blocker?

A

Depolarizing blocker

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8
Q

Is Hypomagnesemia a depolarizing blocker or a non-depolarizing blocker?

A

Depolarizing blocker

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9
Q

What is the mechanism behind Lambert-Eaton Syndrome? What does this result in (non-depolarization or depolarization)?

A

Blocks Ca2+ channels

- Non-depolarizing

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10
Q

What is the mechanism behind Botulism? What does this result in (non-depolarization or depolarization)?

A

Blocks/degrades SNARE proteins

- Non-depolarizing

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11
Q

What is the mechanism behind Curate? What does this result in (non-depolarization or depolarization)?

A

Competitively and reversibly inhibits nicotinic ACh receptors
- Non-depolarizing

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12
Q

What is the mechanism behind Myasthenia Gravis? What does this result in (non-depolarization or depolarization)? What is the treatment?

A

Blocks ACh receptors
- Non-depolarizing

Treat with Neostigmine or Pyridostigmine

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13
Q

What is the mechanism behind Hypermagnesemia? What does this result in (non-depolarization or depolarization)?

A

Too much Mg blocks Ca2+ influx, decreased ACh exocytosis

- Non-depolarizing

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14
Q

What is the mechanism behind a Black Widow bite? What does this result in (non-depolarization or depolarization)?

A

Increased ACh release

- Depolarization

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15
Q

What is the mechanism behind Acetylcholinesterase Inhibitors? What does this result in (non-depolarization or depolarization)? What are three examples?

A

Increased ACh in the synaptic cleft due to decreased degradation of ACh by Acetylcholinesterase

  • Depolarization
  • Ex. Succinylcholine, Neostigmine and Pyridostigmine
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16
Q

What is the mechanism behind Hypomagnesemia? What does this result in (non-depolarization or depolarization)?

A

Not enough Mg competing at Ca2+ channels, increasing ACh exocytosis
- Depolarization

Physiology I (9 decks)

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