Equine Lameness Flashcards

1
Q

What background history should we initially collect for a potentially lame patient?

A
Signalment
Use
Duration of ownership
Recent management - work/exercise, feeding, shoeing, housing
Previous medical problems
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2
Q

What specific history should we collect about a potentially lame horse?

A
Limb(s) affected
Timing and nature of onset of signs
Associated events or incidents
Details of any areas of swelling/heat/pain
Progression of signs since onset
Treatments/management employed to date
Current state of problem
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3
Q

What are the aims of the initial lameness workup?

A
Is horse actually lame? If so:
Identify the limb(s) affected
Score the severity of lameness
Try to identify source/cause
Implement treatment plan
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4
Q

What does the initial lameness workup generally include?

A
Physical examination
Focused exam of MSK system
Gait evaluation - walk/trot/lunge
Flexion tests
Further exam of affected limb
Further workup - nerve/joint blocks, diagnostic imaging
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5
Q

How do we carry out a lameness physical examination?

A
General clinical exam
General body condition
Conformation of body, limbs and feet
Posture and weightbearing on limbs
Skeletal and soft tissue symmetry
Localised swellings/thickenings
Detailed evaluation - inspection/palpation/manipulation
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6
Q

What do we want to establish when evaluating gait?

A
Is there a gait abnormality?
Is this due to lameness or something else (e.g. neurological)?
What degree of lameness?
Which limbs are affected?
What exacerbates the lameness?
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7
Q

How do we carry out a gait evaluation?

A

Different surfaces - soft or hard
Start with walk - if obviously lame at walk then will not trot
Trot up in a straight line
Move on to lunging (soft and hard surface)

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8
Q

What considerations should we have when trotting up a horse?

A
PPE - hard hat, steel toe-capped boots, overalls, gloves
Location - safe, flat surface, weather
Contained?
How lame?
Temperament of horse
Restraint - headcollar +/- bridle
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9
Q

How do we recognise forelimb lameness?

A

Assess as the horse is walking/trotting towards you
Head nod
Head lifts UP as the LAME leg hits the ground or vice versa

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10
Q

How do we recognise hindlimb lameness?

A

Assess as horse is walking/trotting away from you
Harder to recognise and identify than forelimb
Hindquarters as a whole pushed up by sound limb and sink during stance phase of lame limb
‘Hip’ of the lame limb will rise and fall through a greater range of motion than the sound side

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11
Q

What other aspects are we assessing in gait evaluation?

A

Relative lengths of phases of stride
Arc of foot flight
Path of foot flight (medial/lateral)
Foot placement

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12
Q

How do we grade lameness?

A

Two different grading systems used commonly - out of 10 or out of 5
Most useful for individual clinician to assess improvement

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13
Q

Why do we use provocative (flexion) tests?

A

To demonstrate occult lameness in ‘sound’ horse
To exacerbate a mild lameness
To aid localisation of source of lameness

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14
Q

How do we carry out a flexion test?

A

Limb held in flexion for about 1 minute
Horse trotted away as soon as limb released
Allowed a few lame strides
Horse should be standing ready to trot away
Attempt to flex only the joints you are testing

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15
Q

What are the limitations of the flexion test?

A

Lack of specificity to site
Inconsistency
Lack of hard criteria for ‘positive’
False positives and negatives

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16
Q

How do we lunge a horse?

A

Lameness often exacerbated on a circle
Usually exacerbates lameness with lame leg on the inside
Can be performed on hard or soft ground - hard usually exacerbates more

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17
Q

What considerations should we have for lunging a horse?

A

PPE - hard hat, steel toe-capped boots, gloves, overalls
Correct equipment - lunge line, lunge whip, bridle/lunging cavesoon, boots for horse
Does the horse lunge well?
Location - soft e.g. school, arena, lunging pen / hard e.g. hospital area
Is location flat, large, secure, safe, appropriate surface?

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18
Q

How do we carry out nerve blocks?

A

Start distally and work up
Clean area with chlorhex and spirit +/- clipping
Usually unsedated - consider safety
Left 10 mins then trot up to check for improvement

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19
Q

What are common sites and equipment for nerve blocks?

A

Palmar/plantar digital, abaxial sesamoid, low 4 point
23-25G, 5-8” needles, 2ml syringes
Other nerve blocks if need to go higher up limb

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20
Q

How do we carry out joint blocks?

A

Intrasynovial admin - sterility is key!
Sterile skin prep - chlorhexidine and spirit
Evaluated at 10 mins and then later

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21
Q

How do we radiograph horses?

A

Remember PPE - lead gowns, thyroid protectors
Usually sedate horses - keep them still
Useful for identifying bony change

22
Q

Why do we use ultrasound on lame horses?

A

Distinguishes tendon/ligament injuries from peritendinous swelling
Defines which tendon/ligament is injured
Evaluates the type and degree of damage
Monitors healing

23
Q

How do we carry out ultrasound on horses?

A

Clip area if required
Clean to remove dirt
Apply ultrasound gel to area
May require sedation

24
Q

What are we looking for on lameness ultrasounds?

A

Increase in tendon/ligament size
Change in internal architecture - hypoechoic core lesions, heterogenous pattern, loss of longitudinal fibre alignment
Indistinct margination
Peritendinous fluid in tendon sheaths

25
Q

Why do we use diagnostic arthroscopy?

A

Direct visualisation of joint cavities, including - articular cartilage, synovial membrane, intra-articular ligaments, menisci

26
Q

Why do we use MRI on lame horses?

A

Simultaneous demonstration of bone and soft tissue structures
Potential for evaluation of tendons/ligaments and other soft tissues

27
Q

What lesions can be detected using nuclear scintigraphy?

A

Stress fractures
Arthropathies
Enthesiopathies

28
Q

How do we carry out nuclear scintigraphy?

A

Lunged/exercised prior to injection to increase uptake if safe to do so
IV catheter placed
Horses will be radioactive for period of time after injection!
PPE - lead gowns etc.
Urine will need to be collected during image acquisition
Must be kept in isolation until no longer radioactive - no handling/mucking out etc.
Be aware of specific protocol

29
Q

What is synovial sepsis?

A

Bacterial contamination of synovial structure
Usually wounds in adults
Causes septic arthritis and chronic lameness if not treated

30
Q

How can we investigate synovial sepsis?

A

Synoviocentesis and analysis of synovial fluid
May inject sterile saline into the joint and check for egress
Contrast radiography?

31
Q

How do we carry out arthrocentesis?

A

Sterile prep of site - chlorhex 5min minimum scrub, wipe with surgical spirit
Needle requirement depends on joint
Sedation usually

32
Q

What is the role of the nurse during arthrocentesis?

A

Prep the site (vets may choose to do this themselves)
Non-sterile assistant during procedure
Have equipment/spares ready (needles, syringes, sterile gloves, tubes/pots for collection)
Monitoring for lameness/deterioration in lameness in post 48hrs

33
Q

How can we analyse arthrocentesis samples?

A

Cytology - TNCC and neutrophil %
Protein concentration
Lactate (slow to rise in first 24hrs)
May take blood sample for SAA

34
Q

What is laminitis?

A

Inflammation of laminae/lamella in the hoof
Dermal/epidermal separation
Very painful!
Can lead to structural changes/failure in foot - rotation/sinking of P3

35
Q

What are the phases of laminitis?

A

Developmental - between trigger and clinical signs
Acute - onset of clinical signs, 72hrs, may become chronic or subacute after this
Subacute - from 72hrs, 2-3 months repair
Chronic - structural failure

36
Q

What are the clinical signs of laminitis?

A
Stilted, pottery gait
Increased, bounding digital pulses
Leaning back on heels
Recumbency
Worse on hard ground
Struggle to turn
Reluctance to pick up feet
37
Q

What are the possible causes of laminitis?

A

Endocrinopathies e.g. PPID, EMS
Excessive carbohydrates e.g. grain overload, pasture
Excessive weightbearing
Endotoxaemia/SIRS
Corticosteroids?
Pathogenesis not precisely understood, potentially multiple mechanisms

38
Q

What is endocrinopathic laminitis?

A

Underlying endocrinopathy - Equine Metabolic Syndrome (EMS) or Pituitary Pars Intermedia Dysfunction (PPID)
Pathophysiology not known but likely hyperinsulinaemia and insulin toxicity

39
Q

Describe the other causes for laminitis.

A

Supporting limb laminitis - excessive weightbearing, particularly when non-weightbearing on one limb e.g. fractures, cellulitis
Endotoxinaemia - secondary colic, colitis, retained foetal membranes

40
Q

What are some risk factors for laminitis?

A
History of laminitis
Obesity
Endocrinopathies - PPID,  EMS, insulin resistance
Season
Heavy horses
Native ponies
Excessive weightbearing
Excessive carbohydrates
At risk of endotoxaemia - colitis, post-op colics, RFM
41
Q

How can we intervene and try to prevent development of clinical signs of laminitis?

A

At-risk patients e.g. endotoxic, RFMs, supporting limb
Cold therapy - ice boots to reduce perfusion
NSAIDs to reduce inflammation
Support for feet - frog supports, deep shavings bed
Treat underlying endocrinopathy and discuss long-term management

42
Q

How can we manage acute laminitis patients?

A
Strict and complete box rest
Deep (shavings) bed
Frog supports/Styrofoam pads
NSAIDs - analgesia and anti-inflammatory
Increase perfusion - ACP?
Treat underlying cause
Address diet if endocrine-related (reduce carbohydrates gradually)
Once more comfortable - farriery
43
Q

How do we manage subacute and chronic laminitis patients?

A

Subacute - gradually withdraw treatment as long as improving, keep on strict box rest
Farriery - shorten toe over time, remedial shoeing e.g. heart bars, silicone, glue-on shoes

44
Q

What specific considerations should we have for radiographing laminitis patients?

A

Lateromedial and dorsopalmar/plantar projections
Measuring rotation and sinking, prognostic indicators
Comfort - pick up feet and stand on blocks
Metal marker on dorsal hoof wall - coronary band downwards

45
Q

How can we try to prevent laminitis?

A

Control risk factors
Particularly for endocrinopathies - treat underlying, exercise if possible to increase insulin sensitivity, weight loss/promote ideal BCS
Diet - restrict carbohydrates, soaking hay + balancer
Grazing restricted - not just by time!

46
Q

What is box rest?

A

Stabling the horse
Confined rest
No exercise, no turnout

47
Q

What GI considerations should we have when box resting a horse with laminitis?

A

Management changes - colic
Reduced exercise - gut motility, impactions
Diet - sudden change = colic
Ulcers - reduction in eating time, omeprazole?, smaller holed haynets

48
Q

What behaviour considerations should we have for box resting horses with laminitis?

A

Behaviour - stereotypies, stable mates, used to stabling?

Increased energy? - misbehaviour, try to escape/climb up/excitable in stable

49
Q

What nursing can we provide for box resting laminitis patients?

A

Monitor faecal output / colic signs / appetite / signs of stress
Mirrors, company
Watch out for behaviour - more keen to escape, more difficult to handle

50
Q

How do we take a laminitis horse off box rest?

A

Gradual changes in management
Gradual changes in diet
Behaviour when turned out - gradual return to turnout
Slow reintroduction to work