Environmental Trauma Flashcards

1
Q

Hyperthermia is

A

warming of body temp greater than 38 degrees

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2
Q

The body’s mechanisms for heat loss are:

A
radiation
conduction
convection
evaporation
vasodilation
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3
Q

Causes of hyperthermia

A

external heat source
exercise
infection
seizures

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4
Q

Risk factors of hyperthermia

A
long period of warm weather
high humidity 
athletes
military
labourers 
elderly 
infants and children
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5
Q

Patho of hyperthermia

A

excessive heat denatures proteins and stabilises lipids leading to failure of vital energy producing processes and loss of cell membrane function.
redistribution of blood flow and loss of fluids and electrolytes

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6
Q

Heat stress or cramps

A

muscular cramps as a result of water and sodium loss

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7
Q

presentation of heat stress

A
alert
pale, sweaty
tachycardia
weakness
nausea
painful cramps
history of vigorous activity
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8
Q

treatment of heat stress

A
gently stretch muscles, dont massage 
move to cooler place
oral fluids (500ml)
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9
Q

Heat exhaustion

A

water and electrolyte loss and peripheral blood pooling

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10
Q

presentation of heat exhaustion

A
anxious, restless, disorientated 
headache
thirst/dehydration 
nausea and vomiting 
pale and sweaty
tachycardia
orthostatic hypotension
tachypnoea
dizziness
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11
Q

treatment of heat exhaustion

A

move to cool place
auto infuse
fluid replacement
passive cooling until temp less than 38

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12
Q

Heat stroke

A

body unable to regulate temp through hypothalamic thermostat

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13
Q

presentation of heat stroke

A
elevated temp greater than 40 
hot, flushed, dry
neuro dysfunction - anxious, restless, disorientated, seizures, coma 
vomiting and dirrhoea 
rapid bounding pulse 
tachypnoea/hyperventilation 
hypotension
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14
Q

Hypothermia

A

core body temp less than 35 degrees

mild: 32-35
mod: 28-32
severe: less than 28

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15
Q

Risk factors for hypothermia

A
elderly
dementia
neonates
children
sedatives and TCA
trauma
alcohol
immersion
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16
Q

Patho of hypothermia

A

exposure to cold stimulus leads to peripheral vasoconstriction and increase catecholamine release
Overall increase in BMR
Shivering mechanisms designed to increased CBT
Increased HR, BP, RR
Initial increase in temp then continued decrease
Reduced HR, BP, RR and tidal volume
FLuid shift from intravascular space to intersitial space

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17
Q

Signs and symptoms of hypothermia

A
mild: ACS
increased HR and BP 
shivering 
lethargy
hungry 
Mod: ACS/confusion
no shivering
uncoordinated movement 
myocardial irritability and potential for arrhythmias 
severe: usually unconscious
absent vital signs
hypercapnic 
may be cyanosed
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18
Q

Paradoxical undressing

A

pts remove their clothing due to disruption of hypothalamus temp control ability
Due to loss of peripheral vasomotor tone and rush of warm blood back to extremities, creates sense of overheating

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19
Q

Terminal Burrowing

A

Hide and die, pts crawl into a small enclosed space

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20
Q

Osborne/J Wave

A

positive deflection occurring at the junction between QRS segment and T wave

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21
Q

Passive External rewarming techniques

A

pts own body heat
dry clothing
warm, dry, wind free environment

22
Q

Active external rewarming techniques

A

apply warm devices such as warm forced air blankets
hot bath immersion
heat packs/water bottles

23
Q

Active core rewarming techniques

A

warmed IV fluids
irrigation of body cavities with warmed fluids
warmed humidified inhaled air
extracorporeal rewarming

24
Q

Frostbite

A

when an isolated part of the body is exposed to intense cold for prolonged period of time

25
presentation of frostbite
``` cold grey mottled inflammed waxlike hardening ```
26
Frost nip
usually only ear, nose etc | unaware until skin appears white
27
Superficial frostbite
cold and numbness, followed by extreme pain during warming | Oedema after rewarming which them becomes blistered and hard scar
28
Depp frostbite
medical emergency solid to touch, but oedema slow to develop do NOT rewarm
29
Altitude
``` the body can adapt to hypoxic environment: hyperventilate increased production of RBC increased capacity of lungs to diffuse increase vascularity of tissues ```
30
Acute mountain syndrome
6-24 hours after arrival
31
Early/mid signs of AMS
``` similar to hangover headache nausea and vomiting weakness normally self limiting ```
32
late/severe signs of AMS
``` dyspnoea vomiting ACS headache ataxia retinal haemorrhage may progress to HACE or HAPE ```
33
high altitude cerebral oedema patho
increased ICP causes decreased CPP and death vasogenic oedema hypoxia = increased ECF and microvascular permeability
34
signs and symptoms of high altitude cerebral oedema
progressive decline in mental status truncal ataxia cranial nerve palsies
35
Treatment of HACE
stop climbing, rapid descent analgesic and antiemetics dexamethasone to reduce capillary permeability and reduce ICP oxygen 2-4l/m
36
High altitude pulmonary oedema
non cardiac pulmonary oedema due to increased pulmonary artery pressure Increased pressure due to hydrostatic overcomes oncotic pressure therefore fluid shift into intersitial space
37
early signs of HAPE
fluid is interstitial | dry cough and reduced exercise tolerance
38
late signs of HAPE
``` tachycardia increased dyspnoea marked weakness productive cough and frothy sputum cyanosis ```
39
Barotrauma
develops secondary to breathing gases at higher than normal atmospheric pressure
40
Boyle's law
volume of gas inversely proportional to pressue
41
Dalton's law
total sum of the mixture of gases is equal to the sum of the partial pressures of each gas
42
Henry's law
at a constant temp the amount of gas that will dissolve into a liquid is proportional to the partial pressure of the gas
43
Decompression illness (DCI)
ambient pressure increase as diver ascends, increases the partial pressure of nitrogen which is dissolved into body fluids
44
signs and symptoms of DCI
musculoskeletal pain itching resp complaint neurological changes
45
Specific diving history
``` number of dives performed surface interval between dives type of ascent depth of dive breathing gas mixes used levels of exertion before and after dive ```
46
Submersion
process of experiencing resp impairment from submersion/immersion in liquid
47
Indicators of good outcomes after submersion
less than 5min submersion higher GCS post drowning initiation of good quality CPR within 10m first spontaneous breath within 30 m
48
What happens during submersion:
voluntary apnoea, increased BP and HR effects of hypercapnia and hypoxia lead to involuntary breath and sudden increase in airway pressure Secondary apnoea, LOC, involuntary gasping Hypoxia leads to bradycardia, hypotension and irreversible brain injury
49
Factors suggesting aspiration
``` prolonged immersion time any LOC mouth to mouth cough CPR required cyanosis tachycardia wheeze/crackles pink, frothy sputum altered conscious state reduced Spo2 readings ```
50
Diving reflex
caused by cooling of the forehead
51
Diving reflex results in:
bradycardia peripheral vasoconstriction bradypnoea movement of blood to the core helps maintain higher core temps longer and initiates a bolus of oxygenated blood to vital organs prior to cardiac arrest
52
History of drowning
``` underlying medical conditons how long before pt was discovered submersion time why was pt in water how was pt found type of water and temp did the pt ingest water any LOC before during or after any witnessed headstrike CPR/mechanical ventilations seizure activity ```