ENT 2019 Flashcards
Epiglottitis What is the clincial picture?
- high fever/toxic
- sudden onset sore throat/drooling + hyperextended neck + tripod position
- dyspnea + rapidly progressing respiratory obstruction in an attempt to maintain the airway.
- The barking cough typical of croup is rare. Usually, no other family members are ill with acute respiratory symptoms.
1 mo boy presenting with progressively worsening biphasic stridor. No history of intubation. What is the most likely diagnosis? Next step in management? -what if it was inspiratory stridor? Expiratory?
Vocal cord paralysis!! Next step: immediate ENT referral and get MRI of the brain!! Need to look at course of recurrent laryngeal nerves from Cranial nerve CN 9/10 to the nerve itself -Insp stridor = think laryngomalacia -exp stridor = think trachomalaceia -biphasic stridor = think vocal cord paralysis
16 month boy has weekly episodes of falling down suddenly and refusing to get up. Remains conscious and eyes are noted to move during the episode. Recovers within a few minutes. Sometimes vomits. What is his diagnosis? -what is one associated condition?
Benign paroxysmal vertigo = sudden attack of feeling like the room is moving around you; dizziness, little children will tend to drop to the floor, horizontal nystagmus can occur. Can have nausea and vomiting. Usually seen in children
2 most common causes of swollen midline neck mass? -first step in work-up?
- Thyroglossal duct cyst = cystic mass 2. Dermoid cyst = solid mass 3. Lymphadenopathy First step in work-up: ultrasound
A patient has recurrent sore throats. What are indications for tonsillectomy?
- Watchful waiting should be done for recurrent sore throats UNLESS Paradise criteria are met: -7 episodes in preceding year or 5/yr x 2 yrs or 3/yr x 3 yrs PLUS s/s with each episode: fever, lymphadenopathy, recurrent strep infections, etc.
Answer the following questions: 1. Most common cause of nasal obstruction is? 2. Unilateral purulent discharge in a child is most commonly due to? 3. 90% of epistaxis is from the?
- Nasal obstruction = viral rhinitis 2. Unilateral purulent discharge = foreign body 3. 90% of epistaxis = anterior septum
Branchial cleft cysts How do they present?
- may be unilateral or bilateral (2-3%)
- may open onto the cutaneous surface or drain into the pharynx.
- Secondary infection is an indication for systemic antibiotic therapy.
- These anomalies may be inherited as autosomal dominant traits.
Branchial cleft cysts and sinuses What are they?
- 2nd branchial cleft cysts are the most common.
- Cysts and sinuses in the neck may be formed along the course of the 2nd branchial clefts as a result of improper closure during embryonic life.
Hearing How common is hearing loss in newborns?
What is the average age of detection if no screening?
- 2-4 /1000 births
- without screening –mean age of detection is 18-30 months
Hearing loss etiology What are the types and which one is most common?
- Central in origin
-
Peripheral
a. conductive…most common
b. sensorineural, or mixed.
Hearing loss What are causes of SNHL?
-
Damage:
a. hair cell destruction from noise, disease, or ototoxic agents
b. lesions of the acoustic division of the 8th nerve. -
Malformations:
a. cochlear malformation;
b. perilymphatic fistula of the round or oval window membrane;
Hearing loss What are the causes of CHL?
- Congenital causes:
Anomalies of the pinna, external ear canal, TM, and ossicles.
- Genetic -Pierre-Robin, Treacher Collins, Klippel-Feil, OI
-
Acquired (Most CHL is acquired, OME the most common)
a. TM perforation (e.g., trauma, OM), OME , tympanosclerosis
b. ossicular discontinuity (e.g., infection, cholesteatoma, trauma), ,
c. acquired cholesteatoma, or masses in the ear canal or middle ear (e.g., Langerhans’ cell histiocytosis, salivary gland tumors, glomus tumors, rhabdomyosarcoma)
Hearing loss What are the causes of conductive hearing loss?
1. Ear canal: atresia or stenosis, impacted cerumen, or foreign bodies.
-
middle ear,
a. perforation of the tympanic membrane (TM),
b. discontinuity or fixation of the ossicular chain,
c. otitis media (OM) with effusion,
d. otosclerosis, and cholesteatoma
Hearing loss What are the causes of Central (or retrocochlear) hearing loss?
An auditory deficit originating along the central auditory nervous system pathways from the proximal 8th nerve to the cerebral cortex
a. Tumors or demyelinating disease of the 8th nerve and cerebellopontine angle.
b. Central auditory processing disorders
Hearing loss What are the risk factors?
Risk factors (ABCD’s)
A- affected family member
B - bilirubin and hypoxia, Low APGARS, meningitis
C congenital intrauterine infection (TORCH)
D defects of the ear, nose and throat
S small at birth (<1500 g), low apgar, NICU
Hearing loss What are the syndromic to non syndromic causes of Hearing loss?
Branchio-otorenal syndromes = (BOR)

Hearing loss What is Connexin 26?
What are the genetics of Connexin 26?
How does it cause hearing loss?
- most common genetic cause of hearing loss
• AR & mutations in GJB2 gene
- How does it cause hearing loss?
- codes trans-membrane gap junction protein
- allows fluids and small molecules. to pass
- recycles K+ between endolymph
and stria vascularis
Hearing screening What are the 3 screening tests?
- Otoacoustic emissions (OAE) – initial screen
- Auditory brainstem evoked responses (ABR) – correlates highly with hearing, more detailed
- Audiometry – provides fundamental description of hearing sensitivity
Hearing What % of hearing losss is perinatal vs genetic/hereditary?
- 50 % genetic/hereditary & 50% perinatal/acquired
In a child with recurrent severe chronic epistaxis with normal coags, what is your next step in management?
Refer to ENT for endoscopy/possible imaging = need to rule out angiofibroma!
Retropharyngeal abscess What are the complications?
- upper airway obstruction,
- rupture leading to aspiration pneumonia
- extension to the mediastinum.
- Thrombophlebitis of the internal jugular vein and erosion of the carotid artery sheath can also occur
Sinuses Pneumatized by age?
- ethmoidal and maxillary sinuses are present at birth, but only the ethmoidal sinuses are pneumatized.
- maxillary sinuses pneumatized by 4 yr of age.
- sphenoidal sinuses are present by 5 yr of age, whereas the 4. Frontal sinuses begin development at 7-8 yr and completely developed adolescence
Sinusitis What % of viral sinusitis becomes bacterial?
2%