EDs Flashcards

1
Q

What are the spectra, subfactors and syndromes/disorders associated with eating disorders

A

Spectra: internalizing
Subfactors: eating pathology
Syndromes/disorders: bulimia nervosa, anorexia nervosa, binge eating disorder

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2
Q

What are the key symptoms of eating disorders

A

Cognitive restraint
Dietary restriction
Binge eating
Compensatory behaviours
Weight/shape concern

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3
Q

What is cognitive restraint

A

The intent to reduce food consumption

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4
Q

What is dietary restriction

A

An actual decrease in energy intake - the amount and the kinds of foods that are eaten

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5
Q

What is binge eating

A

Consuming an objectively large amount of food, in a discrete period of time with a feeling of loss of control

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6
Q

What are purging compensatory behaviours

A

They physically remove the food like vomiting, laxatives and diuretics

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7
Q

What are non-purging compensatory behaviours

A

Counteracts the ingestion of food indirectly - restriction and compensatory exercise
“Making up” for eating too much

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8
Q

How would we explain weight and shape concern

A

Characterized by body dissatisfaction, preoccupation with weight and shape and the over-valuation of weight and shape
Weight and/or shape play an excessively important role in determining self-worth

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9
Q

Who termed Anorexia Nervosa (AN) and who termed Bulimia Nervosa (BN). Which one is older

A

AN: Sir William Gull
BN: Gerald Russell
AN is older

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10
Q

What are the core features of AN

A

Underweight BMI (usually < 17.5)
Dietary restrictions

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11
Q

How does AN typically present vs how can it sometimes present

A

Typical presentation: body dissatisfaction, weight concerns, undue influence of weight/shape on self-esteem
Sometimes: binge eating, purging, excessive exercise

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12
Q

When someone is in a state of AN, are their action ego-dystonic or ego-syntonic

A

Ego-syntonic

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13
Q

What are the core features of BN

A

Binge eating
Compensatory behaviours
Undue influence of weight/shape on self-esteem

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14
Q

How does BN typically present

A

Body dissatisfaction
Weight concerns
Dietary restrictions

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15
Q

Do individuals with BN experience significant distress or impairment

A

There is some impairment

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16
Q

What are the core features of BED

A

Binge eating
Absence of compensatory behaviours

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17
Q

How can BED sometimes present itself

A

Body dissatisfaction
Weight concerns
Dietary restrictions

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18
Q

What is one symptom that individuals with BED must present with

A

Significant distress

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19
Q

What are the specifiers of AN

A

Restrictive and Binge-purge

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20
Q

What are the residual diagnostic categories of eating disorders

A

ARFID: Avoidant/Restrictive Food Intake Disorder
OSFED: Other Specified Feed and Eating Disorder
USFED: Unspecified Feeding and Eating Disorder

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21
Q

What disorders fit into the OSFED category

A

Sub-threshold AN, BN, BED
Atypical AN (all criteria except low BMI)
Purging Disorder
Night Eating Syndrome

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22
Q

True or false: more people fall into the strict diagnostic categories of AN, BN and BED than into the residual diagnostic categories

A

False: more people are fall into OSFED and USFED

23
Q

True or false: it is strongly and equally likely for someone with AN-R to develop AN-BP and the reverse as well

24
Q

True or false: It is more likely for someone with AN-BP to develop BN than it is for someone with BN to develop AN-BP

25
True or false: It is more likely for someone with BED to develop BN than it is for someone with BN to develop BED
True
26
True or false: someone with BED can develop AN-R and someone with AN-R is also just as likely to develop BED
False; there is no relationship between AN-R and BED
27
Rank the EDs from most to least common prevalence
BED, BN, AN
28
Gauvin, Steiger & Brodeur (2009) did a study of eating pathology among women in Montreal. What did they find
A higher percentage of women engaged in frequent compensatory behaviours, than frequent binge eating and regular purging behaviour BED had the highest prevalence among AN and BN but the highest prevalence was OSFED
29
Rank the EDs from the earliest to the latest onset
AN, BN, BED
30
Which ED has the highest mortality rate
AN
31
Looking beyond the SWAG stereotype, who has high rates of EDs
Sexual and gender minorities Ethnic minorities - multiracial and indigenous individuals
32
True or false: men make up at least 1/4 of the ED population
True
33
True or false: a significant relationship has been found between high SES, EDs and food insecurity
False; there is no relationship between high SES and EDs but there is an association with food insecurity
34
Men with EDs are more likely to:
Over-exercise Want to gain muscle/lose fat
35
The drive for muscularity can lead to rigid behaviours, what are those behaviours
Restricting low-protein foods Prioritizing eating more meals over other tasks Eating beyond being full
36
Why do men usually delay help-seeking with EDs
They are often more secretive due to stigma
37
What are the differential diagnoses of EDs
Body Dysmorphic Disorder (no eating pathology) OCD Anxiety disorders - SAD and GAD Depression Psychosis-spectrum disorders
38
What are the effects of malnutrition
Low mood Anhedonia Insomnia Preoccupation and rituals related to food
39
What are common comorbidities with EDs in general
Mood disorders Substance use and abuse PDs Anxiety disorders
40
Rank the EDs that are most to least comorbid with depression
BN, AN(lifetime), AN(concurrent)
41
AN-R is most likely to be associated with which cluster of PDs
Cluster C
42
AN-BP is most likely to be associated with with PD cluster
cluster B and C
43
BN is most likely to be associated with which PD cluster
Cluster B
44
Heritability of EDs is not static over development and emerges after ______ suggesting that _____ hormones may be playing a role
Puberty; ovarian
45
True or false: there is an additive genetic component and increased familial risk for all EDs
True
46
What is the GxE that is seen in AN & BN symptoms and the drive for thinness. What does this further suggest
The short allele of 5-HTTLPR interacts with parenting style and abuse history Suggests that adverse effects of genes on mental health only manifest under certain environmental conditions
47
How are appearances ideals contributing to EDs as sociocultural factors
Standards of beauty are not universal Media messaging playing crucial roles Minorities fat-shamed more Thin-deal internalization for girls Focus on getting muscles for boys
48
How can the family affect the development of EDs
The thin ideal is transmitted and internalized Attitudes and behaviours of family members are powerful influences on the development of disordered eating Greater influence than media exposure Fail to teach adaptive eating behaviour or coping skills to build self-esteem
49
A study by Lydecker and colleagues looked at the associations of parents’ self, child and other “fat talk” with child eating behaviour and weight. What did they find
Parental fat talk that was directed toward the self was associated with parental pathology (eating, anxiety, depression) Parental fat talk that was directed toward the child was associated with child pathology (strongest predictor)
50
How are EDs influenced by peers and among who was the effect most seen
Teasing someone about their weight regardless of their body type and implying that their weight is tied to character. Most seen with the overweight children Teased at home and by peers almost doubled rates of suicidal ideation compared to those who are not teased
51
What did the Fiji Study by Becker et al. 2004 find
When there was no TV (no interaction with western media) there were no EDs and then purging behaviours began to rise as well as feeling “too fat”
52
True or false: EDs can be conceptualized as a multi-hit/dual pathway model
True
53
What is the ongoing/vicious cycle that is known as the Abstinence violation effect (the fuck it effect)
Starts diet and restrict food Feelings of hunger Eat “forbidden” foods Overeat because you already “broke” the diet Feeling guilty and fat The cycle starts again
54
What are maintenance factors of EDs
Perfectionism: high standards, fear of failure, self-criticism Low self-esteem: omnipresent and unconditional, treatment obstacle Emotion regulation: negative mood intolerable, binge trigger Interpersonal difficulties: Isolation, negative interactions precede binges