ECG 2_arrhythmias Flashcards
Changes on ECG seen in hypokalaemia
Changes on ECG seen in Hyperkalaemia
What’s Torsades de Pointes?
Polymorphic VT = VT with varying amplitude
What’s characteristic of asystole?
No pulse, flat line
Pathological changes in infraction
- ST elevation (fisrt change)
- T wave inversion
- pathological Q wave (full thickness MI, develop 8-12 hrs after full-thickness MI not re-perfused)
What’s the significance of pathological Q wave?
pathological Q wave (full thickness MI, develop 8-12 hrs after full-thickness MI not re-perfused)
(3) STEMI criteria
- ST elevation in >2 small squares in 2 adjacent chest leads
OR
- ST elevation in >1 square in 2 adjacent limb leads
OR
- new LBBB
(2) ischaemic changes on ECG
- ST depression
- new T wave inversion
(2) signs on ECG of the previous infarct
- T wave inversion (persists weeks to months)
- pathological Q wave (permanent)
Signs on ECG of posterior wall (LV) infraction
- dominant R wave in V1/V2
- horizontal ST depression V1-V3
*Q waves only seen if chest leads are placed on pt’s back
Signs of L ventricular hypertrophy on ECG (4)
- L axis deviation
- dominant S wave in V1
- tall R wave in V5/V6 (>big squares)
- T wave inversion in lateral leads
Signs of R ventricular hypertrophy on ECG
- R axis deviation
- dominant R wave in V1
- dominant S wave in V5/V6
- T wave inversion in V1-3, II, III, aVF (right inferior)
What can be seen in Right bundle branch block?
RBBB pattern
What can be seen on ECG in ‘left anterior hemiblock’?
L anterior hemiblock = anterior fascicle of L bundle brunch
ECG: marked L axis deviation